Molecular genetic approach to cell proliferation control and neoplasia (1996)
- Authors:
- Autor USP: SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; BIOLOGIA MOLECULAR
- Language: Inglês
- Imprenta:
- Publisher place: Ribeirao Preto
- Date published: 1996
- Source:
- Título do periódico: Brazilian Journal of Medical and Biological Research
- Volume/Número/Paginação/Ano: v.29, p.911-9, 1996
-
ABNT
ARMELIN, M C S et al. Molecular genetic approach to cell proliferation control and neoplasia. Brazilian Journal of Medical and Biological Research, v. 29, p. 911-9, 1996Tradução . . Acesso em: 21 maio 2024. -
APA
Armelin, M. C. S., Oliveira, M. L., Mercado, J. M., Sasahara, R. M., Valentini, S. R., & Carvalho, L. H. (1996). Molecular genetic approach to cell proliferation control and neoplasia. Brazilian Journal of Medical and Biological Research, 29, 911-9. -
NLM
Armelin MCS, Oliveira ML, Mercado JM, Sasahara RM, Valentini SR, Carvalho LH. Molecular genetic approach to cell proliferation control and neoplasia. Brazilian Journal of Medical and Biological Research. 1996 ;29 911-9.[citado 2024 maio 21 ] -
Vancouver
Armelin MCS, Oliveira ML, Mercado JM, Sasahara RM, Valentini SR, Carvalho LH. Molecular genetic approach to cell proliferation control and neoplasia. Brazilian Journal of Medical and Biological Research. 1996 ;29 911-9.[citado 2024 maio 21 ] - Role of polyoma t antigens in balb-c/3t3 cells growth control deregulation
- Further characterization of balb-3t3 transfectants bearing polyoma t antigen genes
- Controle e subversao da proliferacao
- Glucocorticoid-regulated gene in transformed to normal phenotypic reversion
- T antigens role in polyomavirus transformation: c-myc but not c-fos or c-jun expression is a target for middle t
- Cdna cloning and expression of growth control genes
- Fibroblast growth factor and camp induce the expression of fos and jun proto-oncogenes in pc12 cells
- Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma
- Cloning of differentially expressed genes involved glucocorticoid-induced phenotypic reversion of c6 glioma cells
- Hydrazine derivative - induced proliferation superimposed on citotoxity in cultured moused fibroblasts
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