Characterization of cellular process involved in vascular smooth muscle relaxation induced by a new nitric oxide deliverer complex (2004)
- Authors:
- USP affiliated authors: OLIVEIRA, FABIOLA SINGARETTI DE - FMRP ; SILVA, ROBERTO SANTANA DA - FCFRP ; BENDHACK, LUSIANE MARIA - FCFRP
- Unidades: FMRP; FCFRP
- Subjects: FARMACOLOGIA; CARDIOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Águas de Lindóia
- Date published: 2004
- Source:
- Título do periódico: Programa e Resumos
- Conference titles: Congresso Brasileiro de Farmacologia e Terapêutica Experimental
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ABNT
BONAVENTURA, D. et al. Characterization of cellular process involved in vascular smooth muscle relaxation induced by a new nitric oxide deliverer complex. 2004, Anais.. Águas de Lindóia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2004. . Acesso em: 21 maio 2024. -
APA
Bonaventura, D., Oliveira, F. S. de, Silva, R. S. da, & Bendhack, L. M. (2004). Characterization of cellular process involved in vascular smooth muscle relaxation induced by a new nitric oxide deliverer complex. In Programa e Resumos. Águas de Lindóia: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Bonaventura D, Oliveira FS de, Silva RS da, Bendhack LM. Characterization of cellular process involved in vascular smooth muscle relaxation induced by a new nitric oxide deliverer complex. Programa e Resumos. 2004 ;[citado 2024 maio 21 ] -
Vancouver
Bonaventura D, Oliveira FS de, Silva RS da, Bendhack LM. Characterization of cellular process involved in vascular smooth muscle relaxation induced by a new nitric oxide deliverer complex. Programa e Resumos. 2004 ;[citado 2024 maio 21 ] - Effect of a new nitric oxide donor in renal hypertensive 2 kidney-1 clip rats(2K-1C)
- Decreased vasodilation induced by a new nitric oxide donor in two kidney, one clip hypertensive rats is due to impaired 'K POT +' channel activation
- Relaxation induced by nitrosyl compound is decreased in 2 kidney-1 clip rat aorta
- Vasodilatation induced by a new no donor involves guanylyl cyclase, k+ channels, Cl- channels activation and inhibition of "Ca POT. 2"+ channels
- Vasodilatation induced by nitric oxide donors in potentiated in endothelium intact rat aortic rings
- Vitamina C normaliza efeito de novo doador de óxido nítrico e ativador de canais de 'K POT.+' na aorta de ratos hipertensos renais
- Óxido nítrico (NO) liberado de doadores de NO tem efeito hipotensor e induz relaxamento da aorta de rato devido à ativação de canais K+, guanilato ciclase e Ca-ATPASE do retículo sarcoplasmático
- Vasodilatation by donor is due to inhibition of 'Ca POT. +2' influx
- Vasodilatation induced by atrial natriuretic peptide (ANP) and C-type natriuretic peptide (CNP) in isolated aorta from rats
- Ruthenium-nitrite complex as pro-drug releases NO in a tissue and enzyme-dependent way
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