Selection of FGF2-resistants from mouse tumor cell lines yields clonal sublines of low or non tumorigenic potential (2006)
- Authors:
- Autor USP: ARMELIN, HUGO AGUIRRE - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; CÉLULAS CULTIVADAS DE TUMOR
- Language: Inglês
- Imprenta:
- Publisher: Cold Spring Harbor Laboratory
- Publisher place: New York
- Date published: 2006
- Source:
- Título do periódico: Abstracts
- Conference titles: Cold Spring Harbor Symposium on Quantitative Biology: Mechanisms and Models of Cancer
-
ABNT
MATOS, Tatiana Guimarães de Freitas et al. Selection of FGF2-resistants from mouse tumor cell lines yields clonal sublines of low or non tumorigenic potential. 2006, Anais.. New York: Cold Spring Harbor Laboratory, 2006. . Acesso em: 21 maio 2024. -
APA
Matos, T. G. de F., Costa, É. T., Salotti, J., Yoshihara, C. K., Koga, M. M., & Armelin, H. A. (2006). Selection of FGF2-resistants from mouse tumor cell lines yields clonal sublines of low or non tumorigenic potential. In Abstracts. New York: Cold Spring Harbor Laboratory. -
NLM
Matos TG de F, Costa ÉT, Salotti J, Yoshihara CK, Koga MM, Armelin HA. Selection of FGF2-resistants from mouse tumor cell lines yields clonal sublines of low or non tumorigenic potential. Abstracts. 2006 ;[citado 2024 maio 21 ] -
Vancouver
Matos TG de F, Costa ÉT, Salotti J, Yoshihara CK, Koga MM, Armelin HA. Selection of FGF2-resistants from mouse tumor cell lines yields clonal sublines of low or non tumorigenic potential. Abstracts. 2006 ;[citado 2024 maio 21 ] - Selection of normal revertants from tumorigenic balb 3t3 mouse cell lines carrying the human ej-ras oncogene
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- Effects of ACTH balb 3T3 fibroblasts and AR-1 cells transfected with the ACTH receptor
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- Revertants of tumorigenic mouse cell lines transformed by the human ej-ras oncogene
- Pma induction of tumorigenic potential in balb 3t3 mouse cells carrying the human ej-ras oncogene
- Effects of interactions between FGF2 and PMA in cell cycle arrested BALB 3T3 fibroblasts
- Regulation of expression of fos, jun an myc protooncogenes and activity of ap.1 transcription factor in y-1 adrenocortical cell effects of acth dcamp and phorbol ester
- Role of c-Ki-Ras protein overexpression in adrenocortical Y-1 cells: a study of signal transduction
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