Hyperlipidemic mice present enhanced catabolism and higher mitochondrial ATP-sensitive K+ channel activity (2006)
- Authors:
- Alberici, Luciane Carla - Universidade Estadual de Campinas (UNICAMP)
- Oliveira, Helena Coutinho Franco de - Universidade Estadual de Campinas (UNICAMP)
- Patrício, Patrícia Riva - Universidade Estadual de Campinas (UNICAMP)
- Kowaltowski, Alicia Juliana
- Vercesi, Anibal Eugenio - Universidade Estadual de Campinas (UNICAMP)
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- Subjects: MITOCÔNDRIAS; BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2006
- Source:
- Título do periódico: Gastroenterology
- ISSN: 0016-5085
- Volume/Número/Paginação/Ano: v. 131, n. 4, p. 1228-1234, 2006
-
ABNT
ALBERICI, Luciane Carla et al. Hyperlipidemic mice present enhanced catabolism and higher mitochondrial ATP-sensitive K+ channel activity. Gastroenterology, v. 131, n. 4, p. 1228-1234, 2006Tradução . . Acesso em: 13 maio 2024. -
APA
Alberici, L. C., Oliveira, H. C. F. de, Patrício, P. R., Kowaltowski, A. J., & Vercesi, A. E. (2006). Hyperlipidemic mice present enhanced catabolism and higher mitochondrial ATP-sensitive K+ channel activity. Gastroenterology, 131( 4), 1228-1234. -
NLM
Alberici LC, Oliveira HCF de, Patrício PR, Kowaltowski AJ, Vercesi AE. Hyperlipidemic mice present enhanced catabolism and higher mitochondrial ATP-sensitive K+ channel activity. Gastroenterology. 2006 ; 131( 4): 1228-1234.[citado 2024 maio 13 ] -
Vancouver
Alberici LC, Oliveira HCF de, Patrício PR, Kowaltowski AJ, Vercesi AE. Hyperlipidemic mice present enhanced catabolism and higher mitochondrial ATP-sensitive K+ channel activity. Gastroenterology. 2006 ; 131( 4): 1228-1234.[citado 2024 maio 13 ] - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
