The dual role of p55 tumour necrosis factor-a receptor in Actinobacillus actinomycetemcomitans-induced experimental periodontitis: host protection and tissue destruction (2007)
- Authors:
- USP affiliated authors: GARLET, GUSTAVO POMPERMAIER - FOB ; CAMPOS, MARIO JULIO AVILA - ICB ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidades: FOB; ICB; FMRP
- DOI: 10.1111/j.1365-2249.2006.03260.x
- Subjects: DOENÇAS PERIODONTAIS; RATOS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Clinical and Experimental Immunology
- ISSN: 1365-2249
- Volume/Número/Paginação/Ano: v. 147, n. 1, p. 128-138, 2007
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: green
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ABNT
GARLET, Gustavo Pompermaier et al. The dual role of p55 tumour necrosis factor-a receptor in Actinobacillus actinomycetemcomitans-induced experimental periodontitis: host protection and tissue destruction. Clinical and Experimental Immunology, v. 147, n. 1, p. 128-138, 2007Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2249.2006.03260.x. Acesso em: 19 abr. 2024. -
APA
Garlet, G. P., Cardoso, C. R. B., Campanelli, A. P., Ferreira, B. R., Campos, M. J. A., Cunha, F. de Q., & Silva, J. S. (2007). The dual role of p55 tumour necrosis factor-a receptor in Actinobacillus actinomycetemcomitans-induced experimental periodontitis: host protection and tissue destruction. Clinical and Experimental Immunology, 147( 1), 128-138. doi:10.1111/j.1365-2249.2006.03260.x -
NLM
Garlet GP, Cardoso CRB, Campanelli AP, Ferreira BR, Campos MJA, Cunha F de Q, Silva JS. The dual role of p55 tumour necrosis factor-a receptor in Actinobacillus actinomycetemcomitans-induced experimental periodontitis: host protection and tissue destruction [Internet]. Clinical and Experimental Immunology. 2007 ; 147( 1): 128-138.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1111/j.1365-2249.2006.03260.x -
Vancouver
Garlet GP, Cardoso CRB, Campanelli AP, Ferreira BR, Campos MJA, Cunha F de Q, Silva JS. The dual role of p55 tumour necrosis factor-a receptor in Actinobacillus actinomycetemcomitans-induced experimental periodontitis: host protection and tissue destruction [Internet]. Clinical and Experimental Immunology. 2007 ; 147( 1): 128-138.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1111/j.1365-2249.2006.03260.x - CCR2 deficiency results in increased osteolysis in experimental periapical lesions in mice
- iNOS-derived nitric oxide modulates infection-stimulated bone loss
- The essential role of IFN-'gama' in the control of lethal Aggregatibacter actinomycetemcomitans infection in mice
- Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response
- Differential expression of osteoblast and osteoclast chemmoatractants in compression and tension sides during orthodontic movement
- Role of CCR2 in orthodontic tooth movement
- Tumor necrosis factor-alpha -308G/A single nucleotide polymorphism and red-complex periodontopathogens are independently associated with increased levels of tumor necrosis factor-alpha in diseased periodontal tissues
- Experimental periodontitis in mice selected for maximal or minimal inflammatory reactions: increased inflammatory immune responsiveness drives increased alveolar bone loss without enhancing the control of periodontal infection
- Cytokine pattern determines the progression of experimental periodontal disease induced by Actinobacillus actinomycetemcomitans through the modulation of MMPs, RANKL, and their physiological inhibitors
- The aggravation of arthritis by periodontitis is dependent of IL-17receptor A activation
Informações sobre o DOI: 10.1111/j.1365-2249.2006.03260.x (Fonte: oaDOI API)
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