Hybrid protein endostatin-bax presents higher degree of apoptosis than endostatin after its internalization by endothelial cell line. (C-PAE) (2007)
- Authors:
- Autor USP: MENDES, JOAO GUSTAVO PESSINI AMARANTE - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: Brazilian Society for Biochemistry and Molecular Biology
- Publisher place: Salvador
- Date published: 2007
- Source:
- Título do periódico: Abstracts
- Conference titles: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology - SBBq
-
ABNT
CHURA-CHAMBI, R. M. et al. Hybrid protein endostatin-bax presents higher degree of apoptosis than endostatin after its internalization by endothelial cell line. (C-PAE). 2007, Anais.. Salvador: Brazilian Society for Biochemistry and Molecular Biology, 2007. . Acesso em: 14 maio 2024. -
APA
Chura-Chambi, R. M., Amarante-Mendes, J. G. P., Jacysyn, J. F., Prieto-da-Silva, A., Malavasi, N. V., Rodrigues, D. B., & Morganti, L. (2007). Hybrid protein endostatin-bax presents higher degree of apoptosis than endostatin after its internalization by endothelial cell line. (C-PAE). In Abstracts. Salvador: Brazilian Society for Biochemistry and Molecular Biology. -
NLM
Chura-Chambi RM, Amarante-Mendes JGP, Jacysyn JF, Prieto-da-Silva A, Malavasi NV, Rodrigues DB, Morganti L. Hybrid protein endostatin-bax presents higher degree of apoptosis than endostatin after its internalization by endothelial cell line. (C-PAE). Abstracts. 2007 ;[citado 2024 maio 14 ] -
Vancouver
Chura-Chambi RM, Amarante-Mendes JGP, Jacysyn JF, Prieto-da-Silva A, Malavasi NV, Rodrigues DB, Morganti L. Hybrid protein endostatin-bax presents higher degree of apoptosis than endostatin after its internalization by endothelial cell line. (C-PAE). Abstracts. 2007 ;[citado 2024 maio 14 ] - Modulation of fasl and activation-induced cell death (aicd) of do11.10 T-cells hybridoma by imiquimod
- Aumento de galectina-I mediado por BCR-ABL contribuiu para a transformação maligna na leucemia mielóide crônica
- Apoptosis: a programme of cell death or cell disposal?
- Therapeutic applications of TRAIL receptor agonists in cancer and beyond
- Calpain functions in a caspase-independent manner to promote apoptosis-like events during platelet activation
- Transcriptional changes induced by the BCR-ABL inhibitor STI571 in an in vitro experimental model of chronic myelogenous leukemia
- Apoptose: programa molecular de morte celular
- Butyrate increases apoptosis induced by different antineoplastic drugs in monocytic leukemia cells
- The point of no return: mitochondria, caspases, and the commitment to cell death
- Bcl-2-independent Bcr-Abl-mediated resistance to apoptosis: protection is correlated with up regulation of Bcl-'X IND. L'
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas