The impaired [Ca2+]c reduction by nitric oxide (NO) donor is due to the lower concentration of intracellular no available in renal hypertensive rat aorta cells (2007)
- Authors:
- USP affiliated authors: SILVA, ROBERTO SANTANA DA - FCFRP ; BENDHACK, LUSIANE MARIA - FCFRP
- Unidade: FCFRP
- Subjects: ÓXIDO NÍTRICO; AORTA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Program and Abstracts
- Conference titles: International Congress of Pharmaceutical Sciences (CIFARP)
-
ABNT
LUNARDI, Claure N. et al. The impaired [Ca2+]c reduction by nitric oxide (NO) donor is due to the lower concentration of intracellular no available in renal hypertensive rat aorta cells. 2007, Anais.. São Paulo: ABCF, 2007. . Acesso em: 15 maio 2024. -
APA
Lunardi, C. N., Rodrigues, G. J., Silva, R. S. da, & Bendhack, L. M. (2007). The impaired [Ca2+]c reduction by nitric oxide (NO) donor is due to the lower concentration of intracellular no available in renal hypertensive rat aorta cells. In Program and Abstracts. São Paulo: ABCF. -
NLM
Lunardi CN, Rodrigues GJ, Silva RS da, Bendhack LM. The impaired [Ca2+]c reduction by nitric oxide (NO) donor is due to the lower concentration of intracellular no available in renal hypertensive rat aorta cells. Program and Abstracts. 2007 ;[citado 2024 maio 15 ] -
Vancouver
Lunardi CN, Rodrigues GJ, Silva RS da, Bendhack LM. The impaired [Ca2+]c reduction by nitric oxide (NO) donor is due to the lower concentration of intracellular no available in renal hypertensive rat aorta cells. Program and Abstracts. 2007 ;[citado 2024 maio 15 ] - Vasodilation by no donor is mainly due to inhibition of 'Ca POT.+2" influx
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- The inducing NO-vasodilation by chemical reduction of coordinated nitrite ion in cis-[Ru(NO2)L(bpy)2]+ complex
- Vitamin C improves the effect of a new nitric oxide donor on the vascular smooth muscle from renal hypertensive rats
- Vasodilation induced by a new NO donor involve KCa channels activation and cell membrane hyperpolarization
- Vasodilatation induced by nitric oxide donors is potentiated in endothelium intact rat aortic rings
- Differential effects of the new nitric oxide (NO) donor [Ru(terpy) (bdq)NO+]3+(terpy) on the activation of potassium channels in mesenteric resistance arteries of normotensive and renal hypertensive 2K-1C rats
- Nitric oxide (NO) donor is a pro-drug that releases NO in a tissue and enzyme-dependent way a promotes cell membrane hyperpolarization
- A nitrosyl ruthenium complex [Ru"(terpy)(bdqi)NO]³+ with nitric oxide release and cytotoxic effects against B16-F10 tumor cell line
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