CD4+CD25+Foxp3+ indirect alloreactive T cells from renal transplant patients suppress both the direct and indirect pathways of allorecognition (2007)
- Authors:
- USP affiliated authors: COELHO, VERONICA PORTO CARREIRO DE VASCONCELOS - FM ; KALIL FILHO, JORGE ELIAS - FM
- Unidade: FM
- Subjects: TRANSPLANTE DE RIM (IMUNOLOGIA); HOSPEDEIRO IMUNOCOMPROMETIDO; LINFÓCITOS T; CITOCINAS; NEFROPATIAS (IMUNOLOGIA)
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Scandinavian Journal of Immunology
- ISSN: 0300-9475
- Volume/Número/Paginação/Ano: n. 66, p. 352-361, 2007
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ABNT
SPADAFORA-FERREIRA, M. et al. CD4+CD25+Foxp3+ indirect alloreactive T cells from renal transplant patients suppress both the direct and indirect pathways of allorecognition. Scandinavian Journal of Immunology, n. 66, p. 352-361, 2007Tradução . . Acesso em: 20 maio 2024. -
APA
Spadafora-Ferreira, M., Caldas, C., Faé, K. C., Marrero, I., Monteiro, S. M., Lin-Wang, H. T., et al. (2007). CD4+CD25+Foxp3+ indirect alloreactive T cells from renal transplant patients suppress both the direct and indirect pathways of allorecognition. Scandinavian Journal of Immunology, ( 66), 352-361. -
NLM
Spadafora-Ferreira M, Caldas C, Faé KC, Marrero I, Monteiro SM, Lin-Wang HT, Socorro-Silva A, Fonseca SG, Fonseca JA, Kalil Filho JE, Coelho VPC de V. CD4+CD25+Foxp3+ indirect alloreactive T cells from renal transplant patients suppress both the direct and indirect pathways of allorecognition. Scandinavian Journal of Immunology. 2007 ;( 66): 352-361.[citado 2024 maio 20 ] -
Vancouver
Spadafora-Ferreira M, Caldas C, Faé KC, Marrero I, Monteiro SM, Lin-Wang HT, Socorro-Silva A, Fonseca SG, Fonseca JA, Kalil Filho JE, Coelho VPC de V. CD4+CD25+Foxp3+ indirect alloreactive T cells from renal transplant patients suppress both the direct and indirect pathways of allorecognition. Scandinavian Journal of Immunology. 2007 ;( 66): 352-361.[citado 2024 maio 20 ] - Rejection of grafts with no H-2 disparity in TAP1 mutant mice:: CD4 T cells are important effector cells and self H-2b class I molecules are target
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