Prostaglandin pathway mediates endotoxemia-induced hypophagia and it is associated with an activation of proopiomelanocortin (POMC) and corticotrophin-release factor(CRF) neurons in the hypothalamus (2008)
- Authors:
- USP affiliated authors: CASTRO, MARGARET DE - FMRP ; RODRIGUES, JOSE ANTUNES - FMRP ; ELIAS, LUCILA LEICO KAGOHARA - FMRP
- Unidade: FMRP
- Subjects: HIPOTÁLAMO; ANOREXIA
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2008
- Source:
- Título do periódico: Program and Abstracts Book
- Conference titles: Annual Meeting of the Endocrine Society
-
ABNT
RORATO, R. C. et al. Prostaglandin pathway mediates endotoxemia-induced hypophagia and it is associated with an activation of proopiomelanocortin (POMC) and corticotrophin-release factor(CRF) neurons in the hypothalamus. 2008, Anais.. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2008. . Acesso em: 21 maio 2024. -
APA
Rorato, R. C., Menezes, A. M., Castro, M. de, Antunes-Rodrigues, J., & Elias, L. L. K. (2008). Prostaglandin pathway mediates endotoxemia-induced hypophagia and it is associated with an activation of proopiomelanocortin (POMC) and corticotrophin-release factor(CRF) neurons in the hypothalamus. In Program and Abstracts Book. San Francisco: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Rorato RC, Menezes AM, Castro M de, Antunes-Rodrigues J, Elias LLK. Prostaglandin pathway mediates endotoxemia-induced hypophagia and it is associated with an activation of proopiomelanocortin (POMC) and corticotrophin-release factor(CRF) neurons in the hypothalamus. Program and Abstracts Book. 2008 ;[citado 2024 maio 21 ] -
Vancouver
Rorato RC, Menezes AM, Castro M de, Antunes-Rodrigues J, Elias LLK. Prostaglandin pathway mediates endotoxemia-induced hypophagia and it is associated with an activation of proopiomelanocortin (POMC) and corticotrophin-release factor(CRF) neurons in the hypothalamus. Program and Abstracts Book. 2008 ;[citado 2024 maio 21 ] - Concentração plasmática de peptídeo atrial natriurético (ANP), ocitocina (OT), vasopressina (AVP) e corticosterona após estimulação osmótica angiotensinérgica e colinérgica da região AV3V
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