Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells (2008)
- Authors:
- USP affiliated authors: MALPARTIDA, HUMBERTO MIGUEL GARAY - EACH ; SCHUMACHER, ROBERT - IQ ; SOGAYAR, MARI CLEIDE - IQ ; BECHARA, ETELVINO JOSE HENRIQUES - IQ
- Unidades: EACH; IQ
- DOI: 10.1021/tx8001753
- Subjects: ESTRESSE OXIDATIVO; DIABETES MELLITUS; BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Publisher place: Washington
- Date published: 2008
- Source:
- Título do periódico: Chemical Research in Toxicology
- ISSN: 0893-228X
- Volume/Número/Paginação/Ano: v. 21, n. 9, p. 1841-1850, Sept. 2008
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
SARTORI, Adriano et al. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Chemical Research in Toxicology, v. 21, n. 9, p. 1841-1850, 2008Tradução . . Disponível em: https://doi.org/10.1021/tx8001753. Acesso em: 30 abr. 2024. -
APA
Sartori, A., Garay-Malpartida, H. M., Forni, M. F., Schumacher, R. I., Dutra, F., Sogayar, M. C., & Bechara, E. J. H. (2008). Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Chemical Research in Toxicology, 21( 9), 1841-1850. doi:10.1021/tx8001753 -
NLM
Sartori A, Garay-Malpartida HM, Forni MF, Schumacher RI, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells [Internet]. Chemical Research in Toxicology. 2008 ; 21( 9): 1841-1850.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1021/tx8001753 -
Vancouver
Sartori A, Garay-Malpartida HM, Forni MF, Schumacher RI, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells [Internet]. Chemical Research in Toxicology. 2008 ; 21( 9): 1841-1850.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1021/tx8001753 - Aminoacetone impairs prodution of insulin by RINm5f cells
- Recombinant human prolactin (rhPRL) promotes human beta-cell survival through apoptosis inhibition
- Recombinant human prolactin (rhPRL) promotes human beta cell survival by inhibiting apoptosis
- Cell cycle analysis in glucocorticoid - induced phenotypic reversion of c6 / st1 glioma cell line
- The novel cytoline PANDER/FAM3B inhibits cell death in prostate tumor cells
- Glypican-3 reexpression regulates apoptosis in murine adenocarcinoma mammary cells modulating PI3K/Akt and p38MAPK signaling pathways
- The novel cytokine PANDER/FAM3B inhibits cell death in prostate tumor cells
- Recombinant human prolactin (rhPRL) promotes human beta-cell surviral by inhibiting apoptosis
- Aminoacetone induces copper-mediated oxidative damage to insulin-producing beta cells
- Effects of aminoacetone, a threonine metabolite, on insulin-producing cells
Informações sobre o DOI: 10.1021/tx8001753 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
