Heterozygosity for the S180L variant of Mal/TIRAP, a gene expressing an adaptor protein in the toll-like receptor pathway, is associated lower risk of developing chronic chagas cardiomyopathy (2009)
- Authors:
- USP affiliated authors: KALIL FILHO, JORGE ELIAS - FM ; CUNHA NETO, EDECIO - FM ; MADY, CHARLES - FM
- Unidade: FM
- Subjects: EXPRESSÃO GÊNICA; MIOCARDIOPATIA CHAGÁSICA; MIOCARDIOPATIA CHAGASICA (PARASITOLOGIA)
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: The Journal of Infectious Diseases
- ISSN: 0022-1899
- Volume/Número/Paginação/Ano: v. 199, n. 12, p. 1838-1845, June, 2009
-
ABNT
RAMASAWMY, Rajendranath et al. Heterozygosity for the S180L variant of Mal/TIRAP, a gene expressing an adaptor protein in the toll-like receptor pathway, is associated lower risk of developing chronic chagas cardiomyopathy. The Journal of Infectious Diseases, v. 199, n. 12, p. 1838-1845, 2009Tradução . . Acesso em: 20 maio 2024. -
APA
Ramasawmy, R., Cunha-Neto, E., Fae, K. C., Borba, S. C. P., Teixeira, P. C., Ferreira, S. C. P., et al. (2009). Heterozygosity for the S180L variant of Mal/TIRAP, a gene expressing an adaptor protein in the toll-like receptor pathway, is associated lower risk of developing chronic chagas cardiomyopathy. The Journal of Infectious Diseases, 199( 12), 1838-1845. -
NLM
Ramasawmy R, Cunha-Neto E, Fae KC, Borba SCP, Teixeira PC, Ferreira SCP, Goldberg AC, Ianni B, Mady C, Kalil J. Heterozygosity for the S180L variant of Mal/TIRAP, a gene expressing an adaptor protein in the toll-like receptor pathway, is associated lower risk of developing chronic chagas cardiomyopathy. The Journal of Infectious Diseases. 2009 ; 199( 12): 1838-1845.[citado 2024 maio 20 ] -
Vancouver
Ramasawmy R, Cunha-Neto E, Fae KC, Borba SCP, Teixeira PC, Ferreira SCP, Goldberg AC, Ianni B, Mady C, Kalil J. Heterozygosity for the S180L variant of Mal/TIRAP, a gene expressing an adaptor protein in the toll-like receptor pathway, is associated lower risk of developing chronic chagas cardiomyopathy. The Journal of Infectious Diseases. 2009 ; 199( 12): 1838-1845.[citado 2024 maio 20 ] - Plasma cytokine profile in tropical endomyocardial fibrosis: Predominance of TNF-alfa, IL-4 and IL-10
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- The effect of beta-blockade on myocardial remodelling in Chagas' cardiomyopathy
- Polymorphisms in the gene for lymphotoxin-alpha predispose to chronic Chagas cardiomyopathy
- Increased plasma levels of TNF-'alfa' in Chagas disease and Chagas disease cardiomyopathy
- Locally produced IL-15 may underlie the predominance of CD8+ IL2Ralpha- T cells at heart lesions of human Chagas`disease cardiomyopathy
- Human long-term CD8+ T cell lines express lower levels of IL-2 receptor alpha and gamma chains than long-term CD4+ T cell lines from Chagas patients
- CD8+ cells from HLA-A Trypanosoma cruzi infected recognize several distinct epitopes of T. cruzi proteins cruzipain and FL-160
- Increased plasma levels of tumor necrosis factor-'alfa' in asymptomatic / "indeterminate" and chagas disease cardiomyopathy patients
- Locally produced survival cytokines IL-15 and IL-7 may be associated to the predominance of CD8+ T cells at heart lesions of human chronic Chagas disease cardiomyopathy
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