The role of Rip2 in the signaling pathwy downstream of NOD1 in response to Trypanosoma cruz/infection (2010)
- Authors:
- USP affiliated authors: ZAMBONI, DARIO SIMÕES - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- Subjects: RECEPTORES; MACRÓFAGOS; TRYPANOSOMA CRUZI
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Proceedings
- Conference titles: Annual Meeting of the Brazilian Society of Protozoology
-
ABNT
HORTA, Catarina V. et al. The role of Rip2 in the signaling pathwy downstream of NOD1 in response to Trypanosoma cruz/infection. 2010, Anais.. São Paulo: SBPz, 2010. . Acesso em: 19 abr. 2024. -
APA
Horta, C. V., Silva, G. K., Silva, J. S. da, & Zamboni, D. S. (2010). The role of Rip2 in the signaling pathwy downstream of NOD1 in response to Trypanosoma cruz/infection. In Proceedings. São Paulo: SBPz. -
NLM
Horta CV, Silva GK, Silva JS da, Zamboni DS. The role of Rip2 in the signaling pathwy downstream of NOD1 in response to Trypanosoma cruz/infection. Proceedings. 2010 ;[citado 2024 abr. 19 ] -
Vancouver
Horta CV, Silva GK, Silva JS da, Zamboni DS. The role of Rip2 in the signaling pathwy downstream of NOD1 in response to Trypanosoma cruz/infection. Proceedings. 2010 ;[citado 2024 abr. 19 ] - Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- NOD/RIP2 signaling contribute to development of a TH1 response and resistance against L. major infection
- Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection
- IFN-γ plays a unique role in protection against low virulent Trypanosoma cruzi strain
- A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo
- Restriction of Leishmania amazonensis infection by the inflammasomes
- Intrinsic expression of Nod2 in CD4+ T lymphocytes is not necessary for the development of cell-mediated immunity and host resistance to Toxoplasma gondii
- Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation
- Relevance of the myeloid differentiation factor 88 (MyD88) on RANKL, OPG, and nod expressions induced by TLR and IL-1R signaling in bone marrow stromal cells
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
