A novel pathway for inducible nitric oxide synthase activation by cytosolic flagellin dependent on caspase-1 (2010)
- Authors:
- USP affiliated authors: RUSSO, MOMTCHILO - ICB ; MENDES, JOÃO GUSTAVO PESSINI AMARANTE - ICB
- Unidade: ICB
- Subjects: IMUNOLOGIA; OXIDO NITRICO
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Immunology Letters
- Volume/Número/Paginação/Ano: v. 22, suppl. 1 p. PP-056-07, 2010
- Conference titles: Internacional Congress of Mucosal Immunology Abstracts
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ABNT
BORTOLUCI, K. et al. A novel pathway for inducible nitric oxide synthase activation by cytosolic flagellin dependent on caspase-1. Immunology Letters. Oxford: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 27 abr. 2024. , 2010 -
APA
Bortoluci, K., Buzzo, C., Campopiano, J., Cassado, A., Leme-Souza, R., Lage, L., et al. (2010). A novel pathway for inducible nitric oxide synthase activation by cytosolic flagellin dependent on caspase-1. Immunology Letters. Oxford: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Bortoluci K, Buzzo C, Campopiano J, Cassado A, Leme-Souza R, Lage L, Massis L, Dias da Cunha L, Russo M, Zamboni D, Amarante-Mendes JGP. A novel pathway for inducible nitric oxide synthase activation by cytosolic flagellin dependent on caspase-1. Immunology Letters. 2010 ; 22 suppl. 1 PP-056-07.[citado 2024 abr. 27 ] -
Vancouver
Bortoluci K, Buzzo C, Campopiano J, Cassado A, Leme-Souza R, Lage L, Massis L, Dias da Cunha L, Russo M, Zamboni D, Amarante-Mendes JGP. A novel pathway for inducible nitric oxide synthase activation by cytosolic flagellin dependent on caspase-1. Immunology Letters. 2010 ; 22 suppl. 1 PP-056-07.[citado 2024 abr. 27 ] - Regulação do CD95L por PGE2 e seu impacto na morte de linfócitos T.
- Activation of inos trough NAIP5/NLRC4/CASPASE-1 AXIS
- A suggestive role of NAIP5 on INOS activation
- A suggestive role of naip5 on inos activation in flagellin-stimulated macrophages
- TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3-mediated CD95L upregulation in T cells
- PGE2 derivada de macrófagos ou células dendríticas protege linfócitos T da AICD bloqueando a expressão de FasL
- A novel pathway for inducible nitric oxide synthase (iNOS) activation through inflammasomes
- Inhibition of interferon-'gama'-induced nitric oxide production in endotoxin-activated macrophages by cytolethal distending toxin
- Cross-talk between innate and adaptive immunity: TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3- mediated apoptosis of T cells
- FASL upregulation and T cell death by AICD are prevented by macrophage- and DC-derived PGE2
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