SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis (2010)
- Authors:
- USP affiliated authors: CURTI, CARLOS - FCFRP ; LEOPOLDINO, ANDRÉIA MACHADO - FCFRP
- Unidade: FCFRP
- Subjects: ESTRESSE OXIDATIVO; APOPTOSE; CARCINOMA DE CÉLULAS ESCAMOSAS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Programme and Abstracts
- Conference titles: Cancer and Metabolism: Pathways to the Future
-
ABNT
PESTANA, Cezar Rangel e CURTI, Carlos e LEOPOLDINO, Andréia Machado. SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis. Programme and Abstracts. Edinburgh: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 18 abr. 2024. , 2010 -
APA
Pestana, C. R., Curti, C., & Leopoldino, A. M. (2010). SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis. Programme and Abstracts. Edinburgh: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Pestana CR, Curti C, Leopoldino AM. SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis. Programme and Abstracts. 2010 ;[citado 2024 abr. 18 ] -
Vancouver
Pestana CR, Curti C, Leopoldino AM. SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis. Programme and Abstracts. 2010 ;[citado 2024 abr. 18 ] - SET/I2PP2A is an essential protein for cell migration in head and neck squamous cell carcinoma
- SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant
- Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation
- SET protects mitochondrial function and induces autophagy in response to redox unbalance
- SET protein regulates expression of glutathione S-transferase pi 1 (GSTP-1) in oral carcinoma
- Different responses of two HNSCC cell lineages to oxidative stress
- Knockdown of SET protein sensitizes HNSCC-HN12 cells to UCN-01
- ATP production in HnRNP K-overexpressing HEK293 cells
- Maternal protein restriction increases respiratory and sympathetic activities and sensitizes peripheral chemoreflex in male rat offspring
- Neuroprotective effects of C-Phycocyanin in neuronal cell lines and transient ischemic rat retina
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