Nitric oxide (NO) donor is a pro-drug that releases NO in a tissue and enzyme-dependent way a promotes cell membrane hyperpolarization (2010)
- Authors:
- USP affiliated authors: BENDHACK, LUSIANE MARIA - FCFRP ; SILVA, ROBERTO SANTANA DA - FCFRP
- Unidade: FCFRP
- Subjects: ÓXIDO NÍTRICO; AORTA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Resumos
- Conference titles: Reunião Anual da Federação de Sociedades de Biologia Experimental (FeSBE)
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ABNT
PEREIRA, A. C. et al. Nitric oxide (NO) donor is a pro-drug that releases NO in a tissue and enzyme-dependent way a promotes cell membrane hyperpolarization. 2010, Anais.. São Paulo: FeSBE, 2010. . Acesso em: 02 maio 2024. -
APA
Pereira, A. C., Lunardi, C. N., Silva, R. S. da, & Bendhack, L. M. (2010). Nitric oxide (NO) donor is a pro-drug that releases NO in a tissue and enzyme-dependent way a promotes cell membrane hyperpolarization. In Resumos. São Paulo: FeSBE. -
NLM
Pereira AC, Lunardi CN, Silva RS da, Bendhack LM. Nitric oxide (NO) donor is a pro-drug that releases NO in a tissue and enzyme-dependent way a promotes cell membrane hyperpolarization. Resumos. 2010 ;[citado 2024 maio 02 ] -
Vancouver
Pereira AC, Lunardi CN, Silva RS da, Bendhack LM. Nitric oxide (NO) donor is a pro-drug that releases NO in a tissue and enzyme-dependent way a promotes cell membrane hyperpolarization. Resumos. 2010 ;[citado 2024 maio 02 ] - Decreased vasodilation induced by a new nitric oxide donor in two kidney, one clip hypertensive rats is due to impaired 'K POT +' channel activation
- Relaxation induced by nitrosyl compound is decreased in 2 kidney-1 clip rat aorta
- Vasodilatation induced by a new no donor involves guanylyl cyclase, k+ channels, Cl- channels activation and inhibition of "Ca POT. 2"+ channels
- Vasodilatation induced by nitric oxide donors in potentiated in endothelium intact rat aortic rings
- Vitamina C normaliza efeito de novo doador de óxido nítrico e ativador de canais de 'K POT.+' na aorta de ratos hipertensos renais
- Óxido nítrico (NO) liberado de doadores de NO tem efeito hipotensor e induz relaxamento da aorta de rato devido à ativação de canais K+, guanilato ciclase e Ca-ATPASE do retículo sarcoplasmático
- Vasodilatation by donor is due to inhibition of 'Ca POT. +2' influx
- Vasodilatation induced by atrial natriuretic peptide (ANP) and C-type natriuretic peptide (CNP) in isolated aorta from rats
- Ruthenium-nitrite complex as pro-drug releases NO in a tissue and enzyme-dependent way
- Hipotensão e vasodilatação induzidas pelo doador de NO [RU(TERPY)(BDQ)NO+]3+ (TERPY) são modulados pelo endotélio e estresse oxidativo em ratos espontaneamente hipertensos (SHR)
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