Isolated hyperphosphatemia (HP) does not cause cardiovascular calcification, but may contribute to bone lesion in experimental chronic renal failure (CRF) (2003)
- Authors:
- Autor USP: PASQUALUCCI, CARLOS AUGUSTO GONCALVES - FM
- Unidade: FM
- Subjects: INSUFICIÊNCIA RENAL CRÔNICA (INDUZIDO QUIMICAMENTE); OSSO E OSSOS (LESÕES)
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of the american society of nephrology
- ISSN: 1046-6673
- Volume/Número/Paginação/Ano: V. 14, p. 693A, res. SU-PO718, 2003
- Conference titles: Program and abstracts The american society of nephrology
-
ABNT
NEVES, Katia R et al. Isolated hyperphosphatemia (HP) does not cause cardiovascular calcification, but may contribute to bone lesion in experimental chronic renal failure (CRF). Journal of the american society of nephrology. Baltimore: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 31 maio 2024. , 2003 -
APA
Neves, K. R., Moyses, R. M. A., Graciolli, F. G., Reis, L. M. dos, Pasqualucci, C. A. G., & Jorgetti, V. (2003). Isolated hyperphosphatemia (HP) does not cause cardiovascular calcification, but may contribute to bone lesion in experimental chronic renal failure (CRF). Journal of the american society of nephrology. Baltimore: Faculdade de Medicina, Universidade de São Paulo. -
NLM
Neves KR, Moyses RMA, Graciolli FG, Reis LM dos, Pasqualucci CAG, Jorgetti V. Isolated hyperphosphatemia (HP) does not cause cardiovascular calcification, but may contribute to bone lesion in experimental chronic renal failure (CRF). Journal of the american society of nephrology. 2003 ; 693A.[citado 2024 maio 31 ] -
Vancouver
Neves KR, Moyses RMA, Graciolli FG, Reis LM dos, Pasqualucci CAG, Jorgetti V. Isolated hyperphosphatemia (HP) does not cause cardiovascular calcification, but may contribute to bone lesion in experimental chronic renal failure (CRF). Journal of the american society of nephrology. 2003 ; 693A.[citado 2024 maio 31 ] - Correlation between near-infrared raman spectroscopy and the histopathological ananlysis of atherosclerosis in human coronary arteries
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