Serum from calorie-restricted rats activates vascular cell eNOS through enhanced insulin signaling mediated by adiponectin (2012)
- Authors:
- USP affiliated authors: CUNHA, FERNANDA MARQUES DA - EACH ; KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidades: EACH; IQ
- DOI: 10.1371/journal.pone.0031155
- Subjects: ADIPONECTINA; INSULINA
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2012
- Source:
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
CERQUEIRA, Fernanda Menezes et al. Serum from calorie-restricted rats activates vascular cell eNOS through enhanced insulin signaling mediated by adiponectin. PLOS ONE, v. 7, n. 2, p. 1-6, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0031155. Acesso em: 28 abr. 2024. -
APA
Cerqueira, F. M., Brandizzi, L. I., Cunha, F. M. da, Laurindo, F. R. M., & Kowaltowski, A. J. (2012). Serum from calorie-restricted rats activates vascular cell eNOS through enhanced insulin signaling mediated by adiponectin. PLOS ONE, 7( 2), 1-6. doi:10.1371/journal.pone.0031155 -
NLM
Cerqueira FM, Brandizzi LI, Cunha FM da, Laurindo FRM, Kowaltowski AJ. Serum from calorie-restricted rats activates vascular cell eNOS through enhanced insulin signaling mediated by adiponectin [Internet]. PLOS ONE. 2012 ; 7( 2): 1-6.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1371/journal.pone.0031155 -
Vancouver
Cerqueira FM, Brandizzi LI, Cunha FM da, Laurindo FRM, Kowaltowski AJ. Serum from calorie-restricted rats activates vascular cell eNOS through enhanced insulin signaling mediated by adiponectin [Internet]. PLOS ONE. 2012 ; 7( 2): 1-6.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1371/journal.pone.0031155 - Aging and calorie restriction modulate yeast redox state, oxidized protein removal, and the ubiquitin-proteasome system
- Calorie restriction increases cerebral mitochondrial respiratory capacity in a N'O POT. PONTO'-mediated mechanism: Impact on neuronal survival
- Restrição calórica em Saccharomyces cerevisiae: efeito do pH
- Study of the role of RTG-dependente retrograde signaling in mitochondrial activity maintenance of S. cerevisiae
- Mitochondrial compartmentalization of redox processes
- Mild mitochondrial uncoupling as a therapeutic strategy
- Long-term intermittent feeding, but not caloric restriction, leads to redox imbalance, insulin receptor nitration, and glucose intolerance
- Redox state, insulin sensitivity and aging
- Calorie restriction hysteretically primes aging Saccharomyces cerevisiae toward more effective oxidative metabolism
- Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
Informações sobre o DOI: 10.1371/journal.pone.0031155 (Fonte: oaDOI API)
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