Study of the mechanisms of action of [Ru(TERPY)(BDQ)NO+]+3 (TERPY), a new NO-donor, in aortic rings of spontaneously hypertensive rats (SHR) (2012)
- Authors:
- USP affiliated authors: BENDHACK, LUSIANE MARIA - FCFRP ; SILVA, ROBERTO SANTANA DA - FCFRP
- Unidade: FCFRP
- Subjects: HIPERTENSÃO (INDUZIDO QUIMICAMENTE); ÓXIDO NÍTRICO; TRANSPORTE DE CÁLCIO
- Language: Inglês
- Imprenta:
- Conference titles: Congresso Anual da SBFis
-
ABNT
MUNHOZ, Felipe C. et al. Study of the mechanisms of action of [Ru(TERPY)(BDQ)NO+]+3 (TERPY), a new NO-donor, in aortic rings of spontaneously hypertensive rats (SHR). 2012, Anais.. São Paulo: SBFis, 2012. . Acesso em: 24 abr. 2024. -
APA
Munhoz, F. C., Potje, S., Silva, R. S. da, Bendhack, L. M., & Antoniali, C. (2012). Study of the mechanisms of action of [Ru(TERPY)(BDQ)NO+]+3 (TERPY), a new NO-donor, in aortic rings of spontaneously hypertensive rats (SHR). In . São Paulo: SBFis. -
NLM
Munhoz FC, Potje S, Silva RS da, Bendhack LM, Antoniali C. Study of the mechanisms of action of [Ru(TERPY)(BDQ)NO+]+3 (TERPY), a new NO-donor, in aortic rings of spontaneously hypertensive rats (SHR). 2012 ;[citado 2024 abr. 24 ] -
Vancouver
Munhoz FC, Potje S, Silva RS da, Bendhack LM, Antoniali C. Study of the mechanisms of action of [Ru(TERPY)(BDQ)NO+]+3 (TERPY), a new NO-donor, in aortic rings of spontaneously hypertensive rats (SHR). 2012 ;[citado 2024 abr. 24 ] - Decreased vasodilation induced by a new nitric oxide donor in two kidney, one clip hypertensive rats is due to impaired 'K POT +' channel activation
- Relaxation induced by nitrosyl compound is decreased in 2 kidney-1 clip rat aorta
- Vasodilatation induced by a new no donor involves guanylyl cyclase, k+ channels, Cl- channels activation and inhibition of "Ca POT. 2"+ channels
- Vasodilatation induced by nitric oxide donors in potentiated in endothelium intact rat aortic rings
- Vitamina C normaliza efeito de novo doador de óxido nítrico e ativador de canais de 'K POT.+' na aorta de ratos hipertensos renais
- Óxido nítrico (NO) liberado de doadores de NO tem efeito hipotensor e induz relaxamento da aorta de rato devido à ativação de canais K+, guanilato ciclase e Ca-ATPASE do retículo sarcoplasmático
- Vasodilatation by donor is due to inhibition of 'Ca POT. +2' influx
- Vasodilatation induced by atrial natriuretic peptide (ANP) and C-type natriuretic peptide (CNP) in isolated aorta from rats
- Ruthenium-nitrite complex as pro-drug releases NO in a tissue and enzyme-dependent way
- Hipotensão e vasodilatação induzidas pelo doador de NO [RU(TERPY)(BDQ)NO+]3+ (TERPY) são modulados pelo endotélio e estresse oxidativo em ratos espontaneamente hipertensos (SHR)
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