The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction (2013)
- Authors:
- USP affiliated authors: LEOPOLDINO, ANDRÉIA MACHADO - FCFRP ; CURTI, CARLOS - FCFRP
- Unidade: FCFRP
- Subjects: NEOPLASIAS DE CABEÇA E PESCOÇO; CARCINOMA DE CÉLULAS ESCAMOSAS; ESTRESSE OXIDATIVO
- Language: Inglês
- Imprenta:
- Publisher: SBBq
- Publisher place: Foz do Iguaçu
- Date published: 2013
- Source:
- Título do periódico: Abstracts
- Conference titles: Annual Meeting of SBBq
-
ABNT
PESTANA, C. R. e LEOPOLDINO, Andréia Machado e CURTI, Carlos. The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction. 2013, Anais.. Foz do Iguaçu: SBBq, 2013. . Acesso em: 07 maio 2024. -
APA
Pestana, C. R., Leopoldino, A. M., & Curti, C. (2013). The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction. In Abstracts. Foz do Iguaçu: SBBq. -
NLM
Pestana CR, Leopoldino AM, Curti C. The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction. Abstracts. 2013 ;[citado 2024 maio 07 ] -
Vancouver
Pestana CR, Leopoldino AM, Curti C. The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction. Abstracts. 2013 ;[citado 2024 maio 07 ] - SET/I2PP2A is an essential protein for cell migration in head and neck squamous cell carcinoma
- SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant
- Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation
- SET protects mitochondrial function and induces autophagy in response to redox unbalance
- SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis
- Neuroprotective effects of C-Phycocyanin in neuronal cell lines and transient ischemic rat retina
- Potential modulation of PTEN phosphatase by PP2A and oxidative stress
- SET regulates levels of HnRNP K and its binding to nucleic acid in HNSCC
- SET protein regulates expression of glutathione S-transferase pi 1 (GSTP-1) in oral carcinoma
- Different responses of two HNSCC cell lineages to oxidative stress
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