Hydrogen peroxide activates the endothelial enzymes no-synthase (eNOS) and cyclooxigenase (COX) in renal hypertensive rat aorta (2013)
- Authors:
- USP affiliated authors: BENDHACK, LUSIANE MARIA - FCFRP ; GRANDO, MARCELLA DARUGE - FCFRP
- Unidade: FCFRP
- Subjects: DOENÇAS CARDIOVASCULARES; HIPERTENSÃO; CÉLULAS ENDOTELIAIS
- Language: Inglês
- Imprenta:
- Publisher: SBFTE
- Publisher place: Ribeirão Preto
- Date published: 2013
- Source:
- Título do periódico: Program
- Conference titles: Congresso Brasileiro de Farmacologia e Terapêutica Experimental
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ABNT
SILVA, B. R. e GRANDO, Marcella Daruge e BENDHACK, Lusiane Maria. Hydrogen peroxide activates the endothelial enzymes no-synthase (eNOS) and cyclooxigenase (COX) in renal hypertensive rat aorta. 2013, Anais.. Ribeirão Preto: SBFTE, 2013. . Acesso em: 25 abr. 2024. -
APA
Silva, B. R., Grando, M. D., & Bendhack, L. M. (2013). Hydrogen peroxide activates the endothelial enzymes no-synthase (eNOS) and cyclooxigenase (COX) in renal hypertensive rat aorta. In Program. Ribeirão Preto: SBFTE. -
NLM
Silva BR, Grando MD, Bendhack LM. Hydrogen peroxide activates the endothelial enzymes no-synthase (eNOS) and cyclooxigenase (COX) in renal hypertensive rat aorta. Program. 2013 ;[citado 2024 abr. 25 ] -
Vancouver
Silva BR, Grando MD, Bendhack LM. Hydrogen peroxide activates the endothelial enzymes no-synthase (eNOS) and cyclooxigenase (COX) in renal hypertensive rat aorta. Program. 2013 ;[citado 2024 abr. 25 ] - Endothelial cyclooxygenase differently modulates the contractile response to phenylephrine but not to thromboxane analog in aorta from normotensive and hypertensive rats
- Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats
- Increased resting tension abolishes the endothelial anti-contractile effect induced by phenylephrine in renal hypertensive (2K-1C) rat aorta
- Contractile response induced by phenylephrine in modulated by eNOS phosphorylation and by hydrogen peroxide production in renal hypertensive rat aorta
- Tolerance to nitroglycerin is due to eNOS uncoupling
- Vascular activation of K+ channels and Na+-K+ ATPase activity of estrogen-deficient female rats
- Hidrogen peroxide decreases the contractile response induced by phenylephrine in renal hypertensive rat aorta
- Augmented nitric oxide production and up-regulation of endothelial nitric oxide synthase during cecal ligation and perforation
- A tolerância induzida pela nitroglicerina pode ter início no endotélio após 5 minutos de exposição in vitro com aumento de EROs
- Relaxation induced by the nitric oxide donor and cyclooxygenase inhibitor NCX2121 in renal hypertensive rat aortas
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