NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection (2014)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; ZAMBONI, DARIO SIMÕES - FMRP
- Unidade: FMRP
- Subjects: SISTEMA IMUNE; LEISHMANIA; RECEPTORES
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Proceedings
- Conference titles: Congress of the Brazilian Society of Immunology - IMMUNO
-
ABNT
LIMA JUNIOR, Djalma de Souza e SILVA, João Santana da e ZAMBONI, Dario Simões. NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection. 2014, Anais.. Buzios: SBI, 2014. . Acesso em: 23 abr. 2024. -
APA
Lima Junior, D. de S., Silva, J. S. da, & Zamboni, D. S. (2014). NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection. In Proceedings. Buzios: SBI. -
NLM
Lima Junior D de S, Silva JS da, Zamboni DS. NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection. Proceedings. 2014 ;[citado 2024 abr. 23 ] -
Vancouver
Lima Junior D de S, Silva JS da, Zamboni DS. NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection. Proceedings. 2014 ;[citado 2024 abr. 23 ] - Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- NOD/RIP2 signaling contribute to development of a TH1 response and resistance against L. major infection
- Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection
- IFN-γ plays a unique role in protection against low virulent Trypanosoma cruzi strain
- A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo
- Restriction of Leishmania amazonensis infection by the inflammasomes
- Intrinsic expression of Nod2 in CD4+ T lymphocytes is not necessary for the development of cell-mediated immunity and host resistance to Toxoplasma gondii
- Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation
- Relevance of the myeloid differentiation factor 88 (MyD88) on RANKL, OPG, and nod expressions induced by TLR and IL-1R signaling in bone marrow stromal cells
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