IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous system (2016)
- Authors:
- USP affiliated authors: ALVES FILHO, JOSÉ CARLOS FARIAS - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidade: FMRP
- DOI: 10.1186/s12974-016-0628-1
- Subjects: FLAVIVIRUS; ÓXIDO NÍTRICO; INTERLEUCINAS
- Keywords: INTERLEUKIN 33; ST2 RECEPTOR; EXPERIMENTAL VIRAL ENCEPHALITIS; NITRIC OXIDE; FLAVIVIRUS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Neuroinflammation
- ISSN: 1742-2094
- Volume/Número/Paginação/Ano: v. 13, n. 1, art. 159, 2016
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
FRANÇA, Rafael Freitas de Oliveira et al. IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous system. Journal of Neuroinflammation, v. 13, n. 1, 2016Tradução . . Disponível em: https://doi.org/10.1186/s12974-016-0628-1. Acesso em: 21 maio 2024. -
APA
França, R. F. de O., Costa, R. S., Silva, J. R., Peres, R. S., Mendonça, L. R. de, Colón, D. F., et al. (2016). IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous system. Journal of Neuroinflammation, 13( 1). doi:10.1186/s12974-016-0628-1 -
NLM
França RF de O, Costa RS, Silva JR, Peres RS, Mendonça LR de, Colón DF, Alves Filho JCF, Cunha F de Q. IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous system [Internet]. Journal of Neuroinflammation. 2016 ; 13( 1):[citado 2024 maio 21 ] Available from: https://doi.org/10.1186/s12974-016-0628-1 -
Vancouver
França RF de O, Costa RS, Silva JR, Peres RS, Mendonça LR de, Colón DF, Alves Filho JCF, Cunha F de Q. IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous system [Internet]. Journal of Neuroinflammation. 2016 ; 13( 1):[citado 2024 maio 21 ] Available from: https://doi.org/10.1186/s12974-016-0628-1 - Failure of neutrophil migration to infection focus correlates with sepsis outcome: critical role of TLR, nitric-cGMP-PKG pathway and chemokines receptors
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Informações sobre o DOI: 10.1186/s12974-016-0628-1 (Fonte: oaDOI API)
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