Calorie restriction attenuates hypertrophy-induced redox imbalance and mitochondrial ATP-sensitive K+ channel repression (2018)
- Authors:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- DOI: 10.1016/j.jnutbio.2018.08.008
- Subjects: ESTRESSE OXIDATIVO; MITOCÔNDRIAS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Nutritional Biochemistry
- ISSN: 1873-4847
- Volume/Número/Paginação/Ano: v. 62, p. 87-94, 2018
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
DAVID, Cicera Edna Barbosa et al. Calorie restriction attenuates hypertrophy-induced redox imbalance and mitochondrial ATP-sensitive K+ channel repression. Journal of Nutritional Biochemistry, v. 62, p. 87-94, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jnutbio.2018.08.008. Acesso em: 23 abr. 2024. -
APA
David, C. E. B., Lucas, A. M. B., Araújo, M. T. S., Coelho, B. N., Neto, J. B. S., Portela, B. R. C., et al. (2018). Calorie restriction attenuates hypertrophy-induced redox imbalance and mitochondrial ATP-sensitive K+ channel repression. Journal of Nutritional Biochemistry, 62, 87-94. doi:10.1016/j.jnutbio.2018.08.008 -
NLM
David CEB, Lucas AMB, Araújo MTS, Coelho BN, Neto JBS, Portela BRC, Varela ALN, Kowaltowski AJ, Facundo HT. Calorie restriction attenuates hypertrophy-induced redox imbalance and mitochondrial ATP-sensitive K+ channel repression [Internet]. Journal of Nutritional Biochemistry. 2018 ; 62 87-94.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.jnutbio.2018.08.008 -
Vancouver
David CEB, Lucas AMB, Araújo MTS, Coelho BN, Neto JBS, Portela BRC, Varela ALN, Kowaltowski AJ, Facundo HT. Calorie restriction attenuates hypertrophy-induced redox imbalance and mitochondrial ATP-sensitive K+ channel repression [Internet]. Journal of Nutritional Biochemistry. 2018 ; 62 87-94.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.jnutbio.2018.08.008 - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
Informações sobre o DOI: 10.1016/j.jnutbio.2018.08.008 (Fonte: oaDOI API)
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