Th17-inducing cytokines IL-6 and IL-23 are crucial for granuloma formation during experimental Paracoccidioidomycosis (2017)
- Authors:
- USP affiliated authors: MILANEZI, CRISTIANE MARIA - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- DOI: 10.3389/fimmu.2017.00949
- Subjects: PARACOCCIDIOIDOMICOSE; QUIMIOCINAS; CITOCINAS; INTERLEUCINAS
- Keywords: Paracoccidioidomycosis
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Frontiers in Immunology
- ISSN: 1664-3224
- Volume/Número/Paginação/Ano: v. 8, art. 949, 2017
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
TRISTÃO, Fabrine Sales Massafera et al. Th17-inducing cytokines IL-6 and IL-23 are crucial for granuloma formation during experimental Paracoccidioidomycosis. Frontiers in Immunology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00949. Acesso em: 18 abr. 2024. -
APA
Tristão, F. S. M., Rocha, F. A., Carlos, D., Carneiro, N. K., Souza, C. O. S., Milanezi, C. M., & Silva, J. S. da. (2017). Th17-inducing cytokines IL-6 and IL-23 are crucial for granuloma formation during experimental Paracoccidioidomycosis. Frontiers in Immunology, 8. doi:10.3389/fimmu.2017.00949 -
NLM
Tristão FSM, Rocha FA, Carlos D, Carneiro NK, Souza COS, Milanezi CM, Silva JS da. Th17-inducing cytokines IL-6 and IL-23 are crucial for granuloma formation during experimental Paracoccidioidomycosis [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 abr. 18 ] Available from: https://doi.org/10.3389/fimmu.2017.00949 -
Vancouver
Tristão FSM, Rocha FA, Carlos D, Carneiro NK, Souza COS, Milanezi CM, Silva JS da. Th17-inducing cytokines IL-6 and IL-23 are crucial for granuloma formation during experimental Paracoccidioidomycosis [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 abr. 18 ] Available from: https://doi.org/10.3389/fimmu.2017.00949 - Doxycycline and benznidazole reduce the profile of Th1, Th2, and Th17 chemokines and chemokine receptors in cardiac tissue from chronic Trypanosoma cruzi-infected dogs
- CCR2 receptor is essential to activate microbicidal mechanisms to control Toxoplasma gondii infection in the central nervous system
- Toxoplasma gondii: the severity of toxoplasmic encephalitis in C57BL/6 mice is associated with increased ALCAM and VCAM-1 expression in the central nervous system and higher blood-brain barrier permeability
- Heme oxygenase-1 activity is involved in the control of Toxoplasma gondii infection in the lung of BALB/c and C57BL/6 and in the small intestine of C57BL/6 mice
- Tr-1-like CD4+CD25-CD127-/lowFOXP3-cells are the main source of interleukin 10 in patients with cutaneous leishmaniasis due to Leishmania braziliensis
- IL-17 produced during Trypanosoma cruzi infection plays a central role in regulating parasite-induced myocarditis
- IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection
- NLRC4 inhibits NLRP3 inflammasome and abrogates effective antifungal CD8 + T cell responses
- Diet rich in lard promotes a metabolic environment favorable to Trypanosoma cruzi growth
- BALB/c mice infected with antimony treatment refractory isolate of Leishmania braziliensis present severe lesions due to IL-4 production
Informações sobre o DOI: 10.3389/fimmu.2017.00949 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
