Quercetin treatment restores antioxidant activity and blocks 'H IND. 2''O IND. 2' production in isoproterenol-induced cardiac hypertrophy (2019)
- Authors:
- Autor USP: KOWALTOWSKI, ALÍCIA JULIANA - IQ
- Unidade: IQ
- Subjects: ESTRESSE OXIDATIVO; HIPERTROFIA VENTRICULAR ESQUERDA
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq
- Publisher place: São Paulo
- Date published: 2019
- Source:
- Título do periódico: Abstracts
- Conference titles: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology/SBBq
-
ABNT
ALEXANDRE, Joana Varlla de Lacerda et al. Quercetin treatment restores antioxidant activity and blocks 'H IND. 2''O IND. 2' production in isoproterenol-induced cardiac hypertrophy. 2019, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq, 2019. Disponível em: http://www.sbbq.org.br/reuniao/2019/images/Livro_Resumos_2019.pdf. Acesso em: 01 maio 2024. -
APA
Alexandre, J. V. de L., David, C. É. B., Viana, Y. I. P., Varela, A. L. N., Facundo, H. D. T. F., & Kowaltowski, A. J. (2019). Quercetin treatment restores antioxidant activity and blocks 'H IND. 2''O IND. 2' production in isoproterenol-induced cardiac hypertrophy. In Abstracts. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq. Recuperado de http://www.sbbq.org.br/reuniao/2019/images/Livro_Resumos_2019.pdf -
NLM
Alexandre JV de L, David CÉB, Viana YIP, Varela ALN, Facundo HDTF, Kowaltowski AJ. Quercetin treatment restores antioxidant activity and blocks 'H IND. 2''O IND. 2' production in isoproterenol-induced cardiac hypertrophy [Internet]. Abstracts. 2019 ;[citado 2024 maio 01 ] Available from: http://www.sbbq.org.br/reuniao/2019/images/Livro_Resumos_2019.pdf -
Vancouver
Alexandre JV de L, David CÉB, Viana YIP, Varela ALN, Facundo HDTF, Kowaltowski AJ. Quercetin treatment restores antioxidant activity and blocks 'H IND. 2''O IND. 2' production in isoproterenol-induced cardiac hypertrophy [Internet]. Abstracts. 2019 ;[citado 2024 maio 01 ] Available from: http://www.sbbq.org.br/reuniao/2019/images/Livro_Resumos_2019.pdf - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
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