Chronic exposure to hypoxia attenuates renal injury and innate immunity activation in the remnant kidney model (2019)
- Authors:
- Rempel, Lisienny Campoli Tono
- Faustino, Viviane Dias
- Foresto Neto, Orestes
- Fanelli, Camilla
- Arias, Simone Costa Alarcon
- Moreira, Gizely Cristina da Silva
- Nascimento, Thalita Fabiana do
- Ávila, Victor Ferreira de
- Malheiros, Denise Maria Avancini Costa
- Câmara, Niels Olsen Saraiva
- Fujihara, Clarice Kazue
- Zatz, Roberto
- Rempel, Lisienny Campoli Tono
- USP affiliated authors: FANELLI, CAMILLA - FM ; MALHEIROS, DENISE MARIA AVANCINI COSTA - FM ; CÂMARA, NIELS OLSEN SARAIVA - ICB ; FUJIHARA, CLARICE KAZUE - FM ; ZATZ, ROBERTO - FM ; REMPEL, LISIENNY CAMPOLI TONO - FM ; FAUSTINO, VIVIANE DIAS - FM ; FORESTO NETO, ORESTES - FM ; ARIAS, SIMONE DA COSTA ALARCON - FM ; MOREIRA, GIZELY CRISTINA DA SILVA - FM ; AVILA, VICTOR FERREIRA DE - FM
- Unidades: FM; ICB
- DOI: 10.1152/ajprenal.00367.2018
- Subjects: IMUNOLOGIA; ANÓXIA; INSUFICIÊNCIA RENAL AGUDA; RATOS WISTAR; NEFROPATIAS EM ANIMAL; ESTRESSE OXIDATIVO; IMUNIDADE; IMUNOHISTOQUÍMICA
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: American Journa Physiology: Renal Physiology
- ISSN: 1522-1466
- Volume/Número/Paginação/Ano: v. 317, n. 5, p. F1285-F1292, 2019
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
-
ABNT
REMPEL, Lisienny Campoli Tono et al. Chronic exposure to hypoxia attenuates renal injury and innate immunity activation in the remnant kidney model. American Journa Physiology: Renal Physiology, v. 317, n. 5, p. F1285-F1292, 2019Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00367.2018. Acesso em: 27 abr. 2024. -
APA
Rempel, L. C. T., Faustino, V. D., Foresto Neto, O., Fanelli, C., Arias, S. C. A., Moreira, G. C. da S., et al. (2019). Chronic exposure to hypoxia attenuates renal injury and innate immunity activation in the remnant kidney model. American Journa Physiology: Renal Physiology, 317( 5), F1285-F1292. doi:10.1152/ajprenal.00367.2018 -
NLM
Rempel LCT, Faustino VD, Foresto Neto O, Fanelli C, Arias SCA, Moreira GC da S, Nascimento TF do, Ávila VF de, Malheiros DMAC, Câmara NOS, Fujihara CK, Zatz R. Chronic exposure to hypoxia attenuates renal injury and innate immunity activation in the remnant kidney model [Internet]. American Journa Physiology: Renal Physiology. 2019 ; 317( 5): F1285-F1292.[citado 2024 abr. 27 ] Available from: https://doi.org/10.1152/ajprenal.00367.2018 -
Vancouver
Rempel LCT, Faustino VD, Foresto Neto O, Fanelli C, Arias SCA, Moreira GC da S, Nascimento TF do, Ávila VF de, Malheiros DMAC, Câmara NOS, Fujihara CK, Zatz R. Chronic exposure to hypoxia attenuates renal injury and innate immunity activation in the remnant kidney model [Internet]. American Journa Physiology: Renal Physiology. 2019 ; 317( 5): F1285-F1292.[citado 2024 abr. 27 ] Available from: https://doi.org/10.1152/ajprenal.00367.2018 - Pathogenic role of angiotensin II and the NF-κB system in a model of malignant hypertensive nephrosclerosis
- An association of losartan-hydrochlorothiazide, but not losartan-furosemide, completely arrests progressive injury in the remnant kidney
- NF-κB activation mediates crystal translocation and interstitial inflammation in adenine overload nephropathy
- Innate And Adaptive Immunity are Progressively Activated in Parallel with Renal Injury in the 5/6 Renal Ablation Model
- Losartan (L) and hydrochlorotiazide (H) association (LH) arrests renal inhury in a model of very advanced chronic kidney disease (CKD)
- Losartan (L) and Hydrochlorothiazide (H)Treatment (LH) arrests renal and cardiac injury in advanced chronic kidney disease by mechanisms not entireky dependent on blood pressure
- Renoprotective effect of two dosages of Losartan (L) in a model of crhonic kidney disease (CDK) by nitric oxide inhibition and and salt overload
- Furosemide (F) is less effective than hydroclorothiazide (H) as adjuvant to losartan (L) in arresting the nephropathy associated with the 5/6 renal ablation model (NX)
- TLR2 and TLR4 play opposite role in autophagy associated with cisplatin-induced acute kidney injury
- Associação terapêutica de losartan (L) e hidroclorotiazida (H) previne progressão da lesão renal em modelo de nefropatia crônica resultante da administração de (L) durante a lactação
Informações sobre o DOI: 10.1152/ajprenal.00367.2018 (Fonte: oaDOI API)
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