A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells (2021)
- Authors:
- USP affiliated authors: ORTIS, FERNANDA - ICB ; CARPINELLI, ANGELO RAFAEL - ICB ; BÔAS, ELOÍSA APARECIDA VILAS - IQ
- Unidades: ICB; IQ
- DOI: 10.1016/j.freeradbiomed.2021.08.173
- Subjects: BIOLOGIA CELULAR; FISIOLOGIA; ILHOTAS DE LANGERHANS; ATIVAÇÃO ENZIMÁTICA; OXIDAÇÃO; INFLAMAÇÃO; ENZIMAS OXIRREDUTORAS; RADICAIS LIVRES
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2021
- Source:
- Título do periódico: Free Radical Biology & Medicine
- Volume/Número/Paginação/Ano: v. 177, Supl. 1, p. S112, 2021
- Conference titles: Annual Meeting Abstracts "Redox Biology in the 21. Century"
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
BOAS, Eloisa Aparecida Vilas et al. A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells. Free Radical Biology & Medicine. Philadelphia: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.freeradbiomed.2021.08.173. Acesso em: 01 maio 2024. , 2021 -
APA
Boas, E. A. V., Ortis, F., Roma, L. P., & Carpinelli, A. R. (2021). A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells. Free Radical Biology & Medicine. Philadelphia: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1016/j.freeradbiomed.2021.08.173 -
NLM
Boas EAV, Ortis F, Roma LP, Carpinelli AR. A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells [Internet]. Free Radical Biology & Medicine. 2021 ; 177 S112.[citado 2024 maio 01 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2021.08.173 -
Vancouver
Boas EAV, Ortis F, Roma LP, Carpinelli AR. A role for NADPH oxidase in mediating lipotoxicity and inflammation in β-cells [Internet]. Free Radical Biology & Medicine. 2021 ; 177 S112.[citado 2024 maio 01 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2021.08.173 - Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets
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- Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells
- Chronic activation of GPR40 does not negatively impact upon BRIN-BD11 pancreatic β-cell physiology and function
- Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival
- Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling
- Metabolic memory of ß-cells controls insulin secretion and is mediated by CaMKII
- The non-canonical NF-κB pathway is induced by cytokines in pancreatic beta cells and contributes to cell death and proinflammatory responses in vitro
- Papel de BRG1 e Brm, reguladores globais de transcrição, na reversão fenotípica de células ST1 pela ação de glicocorticóides
Informações sobre o DOI: 10.1016/j.freeradbiomed.2021.08.173 (Fonte: oaDOI API)
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