Hyperphosphorylated Tau in Mesial Temporal Lobe Epilepsy: a Neuropathological and Cognitive Study (2023)
- Authors:
- USP affiliated authors: GRINBERG, LEA TENENHOLZ - FM ; SUEMOTO, CLAUDIA KIMIE - FM ; NITRINI, RICARDO - FM ; PARADELA, REGINA SILVA - FM
- Unidade: FM
- DOI: 10.1007/s12035-022-03190-x
- Subjects: EPILEPSIA; LOBO TEMPORAL; COGNIÇÃO
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Molecular neurobiology
- ISSN: 0893-7648
- Volume/Número/Paginação/Ano: v. 60, n. 4, p. 2174-2185, 2023
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: green
-
ABNT
TOSCANO, Eliana Cristina de Brito et al. Hyperphosphorylated Tau in Mesial Temporal Lobe Epilepsy: a Neuropathological and Cognitive Study. Molecular neurobiology, v. 60, n. 4, p. 2174-2185, 2023Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/53092. Acesso em: 30 abr. 2024. -
APA
Toscano, E. C. de B., Vieira, É. L. M., Grinberg, L. T., Rocha, N. P., Brant, J. A. S., Paradela, R. S., et al. (2023). Hyperphosphorylated Tau in Mesial Temporal Lobe Epilepsy: a Neuropathological and Cognitive Study. Molecular neurobiology, 60( 4), 2174-2185. doi:10.1007/s12035-022-03190-x -
NLM
Toscano EC de B, Vieira ÉLM, Grinberg LT, Rocha NP, Brant JAS, Paradela RS, Giannetti AV, Suemoto CK, Leite REP, Nitrini R. Hyperphosphorylated Tau in Mesial Temporal Lobe Epilepsy: a Neuropathological and Cognitive Study [Internet]. Molecular neurobiology. 2023 ; 60( 4): 2174-2185.[citado 2024 abr. 30 ] Available from: https://observatorio.fm.usp.br/handle/OPI/53092 -
Vancouver
Toscano EC de B, Vieira ÉLM, Grinberg LT, Rocha NP, Brant JAS, Paradela RS, Giannetti AV, Suemoto CK, Leite REP, Nitrini R. Hyperphosphorylated Tau in Mesial Temporal Lobe Epilepsy: a Neuropathological and Cognitive Study [Internet]. Molecular neurobiology. 2023 ; 60( 4): 2174-2185.[citado 2024 abr. 30 ] Available from: https://observatorio.fm.usp.br/handle/OPI/53092 - Adenovirus-Mediated Transduction of Insulin-Like Growth Factor 1 Protects Hippocampal Neurons from the Toxicity of A beta Oligomers and Prevents Memory Loss in an Alzheimer Mouse Model
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Informações sobre o DOI: 10.1007/s12035-022-03190-x (Fonte: oaDOI API)
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