Source: PLOS ONE. Unidade: ICB
Assunto: FISIOLOGIA
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VITZEL, Kaio Fernando et al. Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats. PLOS ONE, v. 8, n. 8, p. 1-10, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0071944. Acesso em: 10 jun. 2024.APA
Vitzel, K. F., Bikopoulos, G., Hung, S., Curi, R., & Ceddia, R. B. (2013). Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats. PLOS ONE, 8( 8), 1-10. doi:10.1371/journal.pone.0071944NLM
Vitzel KF, Bikopoulos G, Hung S, Curi R, Ceddia RB. Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats [Internet]. PLOS ONE. 2013 ; 8( 8): 1-10.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1371/journal.pone.0071944Vancouver
Vitzel KF, Bikopoulos G, Hung S, Curi R, Ceddia RB. Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats [Internet]. PLOS ONE. 2013 ; 8( 8): 1-10.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1371/journal.pone.0071944
