ReP USP - Resultado da busca

Artigo de periodico
Niacin increases atherogenic proteins in high-density lipoprotein of statin-treated subjects (2021)
Ronsein, Graziella Eliza ; Vaisar, Tomas ; Davidson, W. Sean ; Bornfeldt, Karin E ; Probstfield, Jeffrey L ; O’Brien, Kevin D ; Zhao, Xue-Qiao; Heinecke, Jay W
Source: Arteriosclerosis, Thrombosis, and Vascular Biology. Unidade: IQ
Subjects: ESTATINAS, LIPOPROTEÍNAS HDL, PROTEÔMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RONSEIN, Graziella Eliza et al. Niacin increases atherogenic proteins in high-density lipoprotein of statin-treated subjects. Arteriosclerosis, Thrombosis, and Vascular Biology, v. 41, p. 2330–2341, 2021Tradução . . Disponível em: https://doi.org/10.1161/ATVBAHA.121.316278. Acesso em: 10 jun. 2024.
APA
Ronsein, G. E., Vaisar, T., Davidson, W. S., Bornfeldt, K. E., Probstfield, J. L., O’Brien, K. D., et al. (2021). Niacin increases atherogenic proteins in high-density lipoprotein of statin-treated subjects. Arteriosclerosis, Thrombosis, and Vascular Biology, 41, 2330–2341. doi:10.1161/ATVBAHA.121.316278
NLM
Ronsein GE, Vaisar T, Davidson WS, Bornfeldt KE, Probstfield JL, O’Brien KD, Zhao X-Q, Heinecke JW. Niacin increases atherogenic proteins in high-density lipoprotein of statin-treated subjects [Internet]. Arteriosclerosis, Thrombosis, and Vascular Biology. 2021 ; 41 2330–2341.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1161/ATVBAHA.121.316278
Vancouver
Ronsein GE, Vaisar T, Davidson WS, Bornfeldt KE, Probstfield JL, O’Brien KD, Zhao X-Q, Heinecke JW. Niacin increases atherogenic proteins in high-density lipoprotein of statin-treated subjects [Internet]. Arteriosclerosis, Thrombosis, and Vascular Biology. 2021 ; 41 2330–2341.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1161/ATVBAHA.121.316278
Artigo de periodico
Diabetes impairs cellular cholesterol efflux from ABCA1 to small HDL particles (2020)
He, Yi; Ronsein, Graziella Eliza ; Tang, Chongren; Jarvik, Gail P; Davidson, W. Sean; Kothari, Vishal; Song, Hyun D; Segrest, Jere P; Bornfeldt, Karin E; Heinecke, Jay W
Source: Circulation Research. Unidade: IQ
Subjects: DIABETES MELLITUS, COLESTEROL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HE, Yi et al. Diabetes impairs cellular cholesterol efflux from ABCA1 to small HDL particles. Circulation Research, v. 127, n. 9, p. 1198–1210, 2020Tradução . . Disponível em: https://doi.org/10.1161/CIRCRESAHA.120.317178. Acesso em: 10 jun. 2024.
APA
He, Y., Ronsein, G. E., Tang, C., Jarvik, G. P., Davidson, W. S., Kothari, V., et al. (2020). Diabetes impairs cellular cholesterol efflux from ABCA1 to small HDL particles. Circulation Research, 127( 9), 1198–1210. doi:10.1161/CIRCRESAHA.120.317178
NLM
He Y, Ronsein GE, Tang C, Jarvik GP, Davidson WS, Kothari V, Song HD, Segrest JP, Bornfeldt KE, Heinecke JW. Diabetes impairs cellular cholesterol efflux from ABCA1 to small HDL particles [Internet]. Circulation Research. 2020 ; 127( 9): 1198–1210.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1161/CIRCRESAHA.120.317178
Vancouver
He Y, Ronsein GE, Tang C, Jarvik GP, Davidson WS, Kothari V, Song HD, Segrest JP, Bornfeldt KE, Heinecke JW. Diabetes impairs cellular cholesterol efflux from ABCA1 to small HDL particles [Internet]. Circulation Research. 2020 ; 127( 9): 1198–1210.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1161/CIRCRESAHA.120.317178
Artigo de periodico
Time to ditch HOL-C as a measure of PINION HOL function? (2017)
Ronsein, Graziella Eliza ; Heinecke, Jay W
Source: Current Opinion in Lipidology. Unidade: IQ
Subjects: COLESTEROL, DOENÇAS CARDIOVASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RONSEIN, Graziella Eliza e HEINECKE, Jay W. Time to ditch HOL-C as a measure of PINION HOL function?. Current Opinion in Lipidology, v. 28, n. 5, p. 414-418, 2017Tradução . . Disponível em: https://doi.org/10.1097/MOL.0000000000000446. Acesso em: 10 jun. 2024.
APA
Ronsein, G. E., & Heinecke, J. W. (2017). Time to ditch HOL-C as a measure of PINION HOL function? Current Opinion in Lipidology, 28( 5), 414-418. doi:10.1097/MOL.0000000000000446
NLM
Ronsein GE, Heinecke JW. Time to ditch HOL-C as a measure of PINION HOL function? [Internet]. Current Opinion in Lipidology. 2017 ; 28( 5): 414-418.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1097/MOL.0000000000000446
Vancouver
Ronsein GE, Heinecke JW. Time to ditch HOL-C as a measure of PINION HOL function? [Internet]. Current Opinion in Lipidology. 2017 ; 28( 5): 414-418.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1097/MOL.0000000000000446
Artigo de periodico
Plasminogen promotes cholesterol efflux by the ABCA1 pathway (2017)
Pamir, Nathalie; Hutchins, Patrick M; Ronsein, Graziella Eliza ; Wei, Hao; Tang, Chongren; Das, Riku; Vaisar, Tomas; Plow, Edward; Schuster, Volker; Reardon, Catherine A; Weinberg, Richard; Dichek, David A; Marcovina, Santica; Getz, Godfrey S; Heinecke, Jay W
Source: Jcl insight. Unidade: IQ
Subjects: COLESTEROL, MACRÓFAGOS, APOLIPOPROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PAMIR, Nathalie et al. Plasminogen promotes cholesterol efflux by the ABCA1 pathway. Jcl insight, v. 2, n. 15, p. 1-15 art. 92176, 2017Tradução . . Disponível em: https://doi.org/10.1172/jci.insight.92176. Acesso em: 10 jun. 2024.
APA
Pamir, N., Hutchins, P. M., Ronsein, G. E., Wei, H., Tang, C., Das, R., et al. (2017). Plasminogen promotes cholesterol efflux by the ABCA1 pathway. Jcl insight, 2( 15), 1-15 art. 92176. doi:10.1172/jci.insight.92176
NLM
Pamir N, Hutchins PM, Ronsein GE, Wei H, Tang C, Das R, Vaisar T, Plow E, Schuster V, Reardon CA, Weinberg R, Dichek DA, Marcovina S, Getz GS, Heinecke JW. Plasminogen promotes cholesterol efflux by the ABCA1 pathway [Internet]. Jcl insight. 2017 ; 2( 15): 1-15 art. 92176.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1172/jci.insight.92176
Vancouver
Pamir N, Hutchins PM, Ronsein GE, Wei H, Tang C, Das R, Vaisar T, Plow E, Schuster V, Reardon CA, Weinberg R, Dichek DA, Marcovina S, Getz GS, Heinecke JW. Plasminogen promotes cholesterol efflux by the ABCA1 pathway [Internet]. Jcl insight. 2017 ; 2( 15): 1-15 art. 92176.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1172/jci.insight.92176
Artigo de periodico
Patients with coronary endothelial dysfunction have impaired cholesterol efflux capacity and reduced HDL particle concentration (2016)
Monette, Jeffrey S; Hutchins, Patrick M; Ronsein, Graziella Eliza ; Wimberger, Jake; Irwin, Angela D; Tang, Chongren; Sara, Jaskanwal D; Shao, Baohai; Vaisar, Tomas; Lerman, Amir; Heinecke, Jay W
Source: Circulation Research. Unidade: IQ
Subjects: APOLIPOPROTEÍNAS, ARTERIOSCLEROSE, DOENÇAS CARDIOVASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MONETTE, Jeffrey S et al. Patients with coronary endothelial dysfunction have impaired cholesterol efflux capacity and reduced HDL particle concentration. Circulation Research, v. 119, n. 1, p. 83-90, 2016Tradução . . Disponível em: https://doi.org/10.1161/CIRCRESAHA.116.308357. Acesso em: 10 jun. 2024.
APA
Monette, J. S., Hutchins, P. M., Ronsein, G. E., Wimberger, J., Irwin, A. D., Tang, C., et al. (2016). Patients with coronary endothelial dysfunction have impaired cholesterol efflux capacity and reduced HDL particle concentration. Circulation Research, 119( 1), 83-90. doi:10.1161/CIRCRESAHA.116.308357
NLM
Monette JS, Hutchins PM, Ronsein GE, Wimberger J, Irwin AD, Tang C, Sara JD, Shao B, Vaisar T, Lerman A, Heinecke JW. Patients with coronary endothelial dysfunction have impaired cholesterol efflux capacity and reduced HDL particle concentration [Internet]. Circulation Research. 2016 ; 119( 1): 83-90.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1161/CIRCRESAHA.116.308357
Vancouver
Monette JS, Hutchins PM, Ronsein GE, Wimberger J, Irwin AD, Tang C, Sara JD, Shao B, Vaisar T, Lerman A, Heinecke JW. Patients with coronary endothelial dysfunction have impaired cholesterol efflux capacity and reduced HDL particle concentration [Internet]. Circulation Research. 2016 ; 119( 1): 83-90.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1161/CIRCRESAHA.116.308357
Artigo de periodico
Targeted proteomics identifies Paraoxonase/Arylesterase 1 (PON1) and apolipoprotein Cs as potential risk factors for hypoalphalipoproteinemia in diabetic subjects treated with fenofibrate and rosiglitazone (2016)
Ronsein, Graziella Eliza ; Reyes Soffer, Gissette; He, Yi; Oda, Michael; Ginsberg, Henry; Heinecke, Jay W
Source: Molecular e Cellular Proteomics. Unidade: IQ
Subjects: APOLIPOPROTEÍNAS, LIPOPROTEÍNAS HDL, DIABETES MELLITUS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RONSEIN, Graziella Eliza et al. Targeted proteomics identifies Paraoxonase/Arylesterase 1 (PON1) and apolipoprotein Cs as potential risk factors for hypoalphalipoproteinemia in diabetic subjects treated with fenofibrate and rosiglitazone. Molecular e Cellular Proteomics, v. 15, n. 3, p. 1083-1093, 2016Tradução . . Disponível em: https://doi.org/10.1074/mcp.M115.054528. Acesso em: 10 jun. 2024.
APA
Ronsein, G. E., Reyes Soffer, G., He, Y., Oda, M., Ginsberg, H., & Heinecke, J. W. (2016). Targeted proteomics identifies Paraoxonase/Arylesterase 1 (PON1) and apolipoprotein Cs as potential risk factors for hypoalphalipoproteinemia in diabetic subjects treated with fenofibrate and rosiglitazone. Molecular e Cellular Proteomics, 15( 3), 1083-1093. doi:10.1074/mcp.M115.054528
NLM
Ronsein GE, Reyes Soffer G, He Y, Oda M, Ginsberg H, Heinecke JW. Targeted proteomics identifies Paraoxonase/Arylesterase 1 (PON1) and apolipoprotein Cs as potential risk factors for hypoalphalipoproteinemia in diabetic subjects treated with fenofibrate and rosiglitazone [Internet]. Molecular e Cellular Proteomics. 2016 ; 15( 3): 1083-1093.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1074/mcp.M115.054528
Vancouver
Ronsein GE, Reyes Soffer G, He Y, Oda M, Ginsberg H, Heinecke JW. Targeted proteomics identifies Paraoxonase/Arylesterase 1 (PON1) and apolipoprotein Cs as potential risk factors for hypoalphalipoproteinemia in diabetic subjects treated with fenofibrate and rosiglitazone [Internet]. Molecular e Cellular Proteomics. 2016 ; 15( 3): 1083-1093.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1074/mcp.M115.054528
Artigo de periodico
Niacin therapy increases high-density lipoprotein particles and total cholesterol efflux capacity but not ABCA1-specific cholesterol efflux in statin-treated subjects (2016)
Ronsein, Graziella Eliza ; Hutchins, Patrick M; Isquith, Daniel; Vaisar, Tomas; Zhao, Xue-Qiao; Heinecke, Jay W
Source: Arteriosclerosis Thrombosis and Vascular Biolology. Unidade: IQ
Subjects: ARTERIOSCLEROSE, DOENÇAS CARDIOVASCULARES, MACRÓFAGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RONSEIN, Graziella Eliza et al. Niacin therapy increases high-density lipoprotein particles and total cholesterol efflux capacity but not ABCA1-specific cholesterol efflux in statin-treated subjects. Arteriosclerosis Thrombosis and Vascular Biolology, v. 36, n. 2, p. 404-411, 2016Tradução . . Disponível em: https://doi.org/10.1161/ATVBAHA.115.306268. Acesso em: 10 jun. 2024.
APA
Ronsein, G. E., Hutchins, P. M., Isquith, D., Vaisar, T., Zhao, X. -Q., & Heinecke, J. W. (2016). Niacin therapy increases high-density lipoprotein particles and total cholesterol efflux capacity but not ABCA1-specific cholesterol efflux in statin-treated subjects. Arteriosclerosis Thrombosis and Vascular Biolology, 36( 2), 404-411. doi:10.1161/ATVBAHA.115.306268
NLM
Ronsein GE, Hutchins PM, Isquith D, Vaisar T, Zhao X-Q, Heinecke JW. Niacin therapy increases high-density lipoprotein particles and total cholesterol efflux capacity but not ABCA1-specific cholesterol efflux in statin-treated subjects [Internet]. Arteriosclerosis Thrombosis and Vascular Biolology. 2016 ; 36( 2): 404-411.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1161/ATVBAHA.115.306268
Vancouver
Ronsein GE, Hutchins PM, Isquith D, Vaisar T, Zhao X-Q, Heinecke JW. Niacin therapy increases high-density lipoprotein particles and total cholesterol efflux capacity but not ABCA1-specific cholesterol efflux in statin-treated subjects [Internet]. Arteriosclerosis Thrombosis and Vascular Biolology. 2016 ; 36( 2): 404-411.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1161/ATVBAHA.115.306268
Artigo de periodico
Proteomic analysis of HDL from inbred mouse strains implicates APOE associated with HDL in reduced cholesterol efflux capacity via the ABCA1 pathway (2016)
Pamir, Nathalie; Hutchins, Patrick; Ronsein, Graziella Eliza ; Vaisar, Tomas; Reardon, Catherine A; Getz, Godfrey S; Lusis, Aldons J; Heinecke, Jay W
Source: Journal of Lipid Research. Unidade: IQ
Subjects: ARTERIOSCLEROSE, DOENÇAS CARDIOVASCULARES, APOLIPOPROTEÍNAS, LIPOPROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PAMIR, Nathalie et al. Proteomic analysis of HDL from inbred mouse strains implicates APOE associated with HDL in reduced cholesterol efflux capacity via the ABCA1 pathway. Journal of Lipid Research, v. 57, n. 2, p. 246-257, 2016Tradução . . Disponível em: https://doi.org/10.1194/jlr.M063701. Acesso em: 10 jun. 2024.
APA
Pamir, N., Hutchins, P., Ronsein, G. E., Vaisar, T., Reardon, C. A., Getz, G. S., et al. (2016). Proteomic analysis of HDL from inbred mouse strains implicates APOE associated with HDL in reduced cholesterol efflux capacity via the ABCA1 pathway. Journal of Lipid Research, 57( 2), 246-257. doi:10.1194/jlr.M063701
NLM
Pamir N, Hutchins P, Ronsein GE, Vaisar T, Reardon CA, Getz GS, Lusis AJ, Heinecke JW. Proteomic analysis of HDL from inbred mouse strains implicates APOE associated with HDL in reduced cholesterol efflux capacity via the ABCA1 pathway [Internet]. Journal of Lipid Research. 2016 ; 57( 2): 246-257.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1194/jlr.M063701
Vancouver
Pamir N, Hutchins P, Ronsein GE, Vaisar T, Reardon CA, Getz GS, Lusis AJ, Heinecke JW. Proteomic analysis of HDL from inbred mouse strains implicates APOE associated with HDL in reduced cholesterol efflux capacity via the ABCA1 pathway [Internet]. Journal of Lipid Research. 2016 ; 57( 2): 246-257.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1194/jlr.M063701
Artigo de periodico
Cholesterol efflux capacity and subclasses of HDL particles in healthy women transitioning through menopause (2016)
el Khoudary, Samar R; Hutchins, Patrick M; Matthews, Karen A; Brooks, Maria M; Orchard, Trevor J; Ronsein, Graziella Eliza ; Heinecke, Jay W
Source: Journal of Clinical Endocrinology & Metabolism. Unidade: IQ
Subjects: DOENÇAS CARDIOVASCULARES, LIPOPROTEÍNAS HDL, COLESTEROL, MENOPAUSA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
EL KHOUDARY, Samar R et al. Cholesterol efflux capacity and subclasses of HDL particles in healthy women transitioning through menopause. Journal of Clinical Endocrinology & Metabolism, v. 101, n. 9, p. 3419-3428, 2016Tradução . . Disponível em: https://doi.org/10.1210/jc.2016-2144. Acesso em: 10 jun. 2024.
APA
el Khoudary, S. R., Hutchins, P. M., Matthews, K. A., Brooks, M. M., Orchard, T. J., Ronsein, G. E., & Heinecke, J. W. (2016). Cholesterol efflux capacity and subclasses of HDL particles in healthy women transitioning through menopause. Journal of Clinical Endocrinology & Metabolism, 101( 9), 3419-3428. doi:10.1210/jc.2016-2144
NLM
el Khoudary SR, Hutchins PM, Matthews KA, Brooks MM, Orchard TJ, Ronsein GE, Heinecke JW. Cholesterol efflux capacity and subclasses of HDL particles in healthy women transitioning through menopause [Internet]. Journal of Clinical Endocrinology & Metabolism. 2016 ; 101( 9): 3419-3428.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1210/jc.2016-2144
Vancouver
el Khoudary SR, Hutchins PM, Matthews KA, Brooks MM, Orchard TJ, Ronsein GE, Heinecke JW. Cholesterol efflux capacity and subclasses of HDL particles in healthy women transitioning through menopause [Internet]. Journal of Clinical Endocrinology & Metabolism. 2016 ; 101( 9): 3419-3428.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1210/jc.2016-2144

USP Schools

ReP

Filtros

Authors

Subjects

Source title

Non normalized affiliation