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Artigo de periodico
Breastfeeding lifespan control of growth, maintenance, and metabolism of small intestinal epithelium (2023)
Costa, Aline Vasques da ; Rattes, Isadora Campos ; Goes, Carolina Purcell ; Lobo, Larissa Honda Greco; Barreto, Laylla Barreto e ; Gama, Patrícia
Fonte: Journal of Cellular Physiology. Unidades: ICB, ESALQ
Assuntos: BIOLOGIA CELULAR, ALEITAMENTO MATERNO, INTESTINO DELGADO, CÉLULAS EPITELIAIS, RATOS WISTAR, JEJUNO DE ANIMAL, MODELOS ANIMAIS
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COSTA, Aline Vasques da et al. Breastfeeding lifespan control of growth, maintenance, and metabolism of small intestinal epithelium. Journal of Cellular Physiology, v. 238, n. 10, p. 2304-2315, 2023Tradução . . Disponível em: https://doi.org/10.1002/jcp.31089. Acesso em: 19 abr. 2024.
APA
Costa, A. V. da, Rattes, I. C., Goes, C. P., Lobo, L. H. G., Barreto, L. B. e, & Gama, P. (2023). Breastfeeding lifespan control of growth, maintenance, and metabolism of small intestinal epithelium. Journal of Cellular Physiology, 238( 10), 2304-2315. doi:10.1002/jcp.31089
NLM
Costa AV da, Rattes IC, Goes CP, Lobo LHG, Barreto LB e, Gama P. Breastfeeding lifespan control of growth, maintenance, and metabolism of small intestinal epithelium [Internet]. Journal of Cellular Physiology. 2023 ; 238( 10): 2304-2315.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.31089
Vancouver
Costa AV da, Rattes IC, Goes CP, Lobo LHG, Barreto LB e, Gama P. Breastfeeding lifespan control of growth, maintenance, and metabolism of small intestinal epithelium [Internet]. Journal of Cellular Physiology. 2023 ; 238( 10): 2304-2315.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.31089
Artigo de periodico
FOXO1 is downregulated in obese mice subjected to short‐term strength training (2022)
Pereira, Rodrigo Martins; Rodrigues, Kellen Cristina da Cruz; Sant'Ana, Marcella R.; Rocha, Alisson Luiz da ; Morelli, Ana Paula; Veras, Allice S. C.; Gaspar, Rodrigo Stellzer; Fernandes, Célio Junior da Costa; Teixeira, Giovana Rampazzo; Simabuco, Fernando Moreira; Silva, Adelino Sanchez Ramos da ; Cintra, Dennys Esper; Ropelle, Eduardo Rochete; Pauli, José Rodrigo; Moura, Leandro Pereira de
Fonte: Journal of Cellular Physiology. Unidades: EEFERP, FMRP
Assuntos: DIABETES MELLITUS, INSULINA, RESISTÊNCIA À INSULINA, NUTRIÇÃO, OBESIDADE
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PEREIRA, Rodrigo Martins et al. FOXO1 is downregulated in obese mice subjected to short‐term strength training. Journal of Cellular Physiology, v. 237, n. 11, p. 4262-4274, 2022Tradução . . Disponível em: https://doi.org/10.1002/jcp.30882. Acesso em: 19 abr. 2024.
APA
Pereira, R. M., Rodrigues, K. C. da C., Sant'Ana, M. R., Rocha, A. L. da, Morelli, A. P., Veras, A. S. C., et al. (2022). FOXO1 is downregulated in obese mice subjected to short‐term strength training. Journal of Cellular Physiology, 237( 11), 4262-4274. doi:10.1002/jcp.30882
NLM
Pereira RM, Rodrigues KC da C, Sant'Ana MR, Rocha AL da, Morelli AP, Veras ASC, Gaspar RS, Fernandes CJ da C, Teixeira GR, Simabuco FM, Silva ASR da, Cintra DE, Ropelle ER, Pauli JR, Moura LP de. FOXO1 is downregulated in obese mice subjected to short‐term strength training [Internet]. Journal of Cellular Physiology. 2022 ; 237( 11): 4262-4274.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.30882
Vancouver
Pereira RM, Rodrigues KC da C, Sant'Ana MR, Rocha AL da, Morelli AP, Veras ASC, Gaspar RS, Fernandes CJ da C, Teixeira GR, Simabuco FM, Silva ASR da, Cintra DE, Ropelle ER, Pauli JR, Moura LP de. FOXO1 is downregulated in obese mice subjected to short‐term strength training [Internet]. Journal of Cellular Physiology. 2022 ; 237( 11): 4262-4274.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.30882
Artigo de periodico
Reverted effect of mesenchymal stem cells in glioblastoma treated with agathisflavone and its selective antitumoral effect on cell viability, migration, and differentiation via STAT3 (2021)
Nascimento, Ravena P; Santos, Balbino Lino dos ; Silva, Karina C. da; Silva, Victor D. Amaral da; Costa, Maria de Fátima; David, Jorge M; David, Juceni P; Moura-Neto, Vivaldo; Oliveira, Mona das Neves; Ulrich, Henning ; Lopes, Giselle P. de Faria; Costa, Silvia Lima
Fonte: Journal of Cellular Physiology. Unidade: IQ
Assuntos: CÉLULAS-TRONCO, FLAVONOIDES, NEOPLASIAS DO SISTEMA NERVOSO
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NASCIMENTO, Ravena P et al. Reverted effect of mesenchymal stem cells in glioblastoma treated with agathisflavone and its selective antitumoral effect on cell viability, migration, and differentiation via STAT3. Journal of Cellular Physiology, v. 236, n. 7, p. 5022-5035, 2021Tradução . . Disponível em: https://doi.org/10.1002/jcp.30209. Acesso em: 19 abr. 2024.
APA
Nascimento, R. P., Santos, B. L. dos, Silva, K. C. da, Silva, V. D. A. da, Costa, M. de F., David, J. M., et al. (2021). Reverted effect of mesenchymal stem cells in glioblastoma treated with agathisflavone and its selective antitumoral effect on cell viability, migration, and differentiation via STAT3. Journal of Cellular Physiology, 236( 7), 5022-5035. doi:10.1002/jcp.30209
NLM
Nascimento RP, Santos BL dos, Silva KC da, Silva VDA da, Costa M de F, David JM, David JP, Moura-Neto V, Oliveira M das N, Ulrich H, Lopes GP de F, Costa SL. Reverted effect of mesenchymal stem cells in glioblastoma treated with agathisflavone and its selective antitumoral effect on cell viability, migration, and differentiation via STAT3 [Internet]. Journal of Cellular Physiology. 2021 ; 236( 7): 5022-5035.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.30209
Vancouver
Nascimento RP, Santos BL dos, Silva KC da, Silva VDA da, Costa M de F, David JM, David JP, Moura-Neto V, Oliveira M das N, Ulrich H, Lopes GP de F, Costa SL. Reverted effect of mesenchymal stem cells in glioblastoma treated with agathisflavone and its selective antitumoral effect on cell viability, migration, and differentiation via STAT3 [Internet]. Journal of Cellular Physiology. 2021 ; 236( 7): 5022-5035.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.30209
Artigo de periodico
Temporomandibular inflammation regulates the matrix metalloproteinases MMP‐2 and MMP‐9 in limbic structures (2021)
Nascimento, Glauce Crivelaro do; Paula, Bruna Balbino de; Gerlach, Raquel Fernanda; Leite-Panissi, Christie Ramos Andrade
Fonte: Journal of Cellular Physiology. Unidades: FORP, FFCLRP
Assuntos: MATRIZ EXTRACELULAR, INFLAMAÇÃO, SISTEMA LÍMBICO, METALOPROTEINASES, DOR FACIAL
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NASCIMENTO, Glauce Crivelaro do et al. Temporomandibular inflammation regulates the matrix metalloproteinases MMP‐2 and MMP‐9 in limbic structures. Journal of Cellular Physiology, v. 236, n. 9, p. 6571-6580, 2021Tradução . . Disponível em: https://doi.org/10.1002/jcp.30341. Acesso em: 19 abr. 2024.
APA
Nascimento, G. C. do, Paula, B. B. de, Gerlach, R. F., & Leite-Panissi, C. R. A. (2021). Temporomandibular inflammation regulates the matrix metalloproteinases MMP‐2 and MMP‐9 in limbic structures. Journal of Cellular Physiology, 236( 9), 6571-6580. doi:10.1002/jcp.30341
NLM
Nascimento GC do, Paula BB de, Gerlach RF, Leite-Panissi CRA. Temporomandibular inflammation regulates the matrix metalloproteinases MMP‐2 and MMP‐9 in limbic structures [Internet]. Journal of Cellular Physiology. 2021 ; 236( 9): 6571-6580.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.30341
Vancouver
Nascimento GC do, Paula BB de, Gerlach RF, Leite-Panissi CRA. Temporomandibular inflammation regulates the matrix metalloproteinases MMP‐2 and MMP‐9 in limbic structures [Internet]. Journal of Cellular Physiology. 2021 ; 236( 9): 6571-6580.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.30341
Artigo de periodico
Osteoporosis and osteoblasts cocultured with adipocytes inhibit osteoblast differentiation by downregulating histone acetylation (2021)
Abuna, Rodrigo Paolo Flores ; Almeida, Luciana Oliveira de; Souza, Alann Thaffarell Portilho de ; Fernandes, Roger Rodrigo ; Sverzut, Thales Fabro Vanzela; Rosa, Adalberto Luiz ; Beloti, Márcio Mateus
Fonte: Journal of Cellular Physiology. Unidades: FORP, FMRP
Assuntos: OSTEOBLASTO, OSTEOPOROSE, CÉLULAS ESTROMAIS, TECIDO ADIPOSO, PROTEÍNAS, CITOFISIOLOGIA
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ABUNA, Rodrigo Paolo Flores et al. Osteoporosis and osteoblasts cocultured with adipocytes inhibit osteoblast differentiation by downregulating histone acetylation. Journal of Cellular Physiology, v. 236, n. 5, p. 3906-3917, 2021Tradução . . Disponível em: https://doi.org/10.1002/jcp.30131. Acesso em: 19 abr. 2024.
APA
Abuna, R. P. F., Almeida, L. O. de, Souza, A. T. P. de, Fernandes, R. R., Sverzut, T. F. V., Rosa, A. L., & Beloti, M. M. (2021). Osteoporosis and osteoblasts cocultured with adipocytes inhibit osteoblast differentiation by downregulating histone acetylation. Journal of Cellular Physiology, 236( 5), 3906-3917. doi:10.1002/jcp.30131
NLM
Abuna RPF, Almeida LO de, Souza ATP de, Fernandes RR, Sverzut TFV, Rosa AL, Beloti MM. Osteoporosis and osteoblasts cocultured with adipocytes inhibit osteoblast differentiation by downregulating histone acetylation [Internet]. Journal of Cellular Physiology. 2021 ; 236( 5): 3906-3917.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.30131
Vancouver
Abuna RPF, Almeida LO de, Souza ATP de, Fernandes RR, Sverzut TFV, Rosa AL, Beloti MM. Osteoporosis and osteoblasts cocultured with adipocytes inhibit osteoblast differentiation by downregulating histone acetylation [Internet]. Journal of Cellular Physiology. 2021 ; 236( 5): 3906-3917.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.30131
Artigo de periodico
Muscle regeneration in adiponectin knockout mice showed early activation of anti‐inflammatory response with perturbations in myogenesis (2020)
Mosele, Francielle Caroline ; Barros, Rafael Bissi Ricci de ; Teixeira, Phablo Sávio Abreu ; Rosa Neto, José Cesar
Fonte: Journal of Cellular Physiology. Unidades: ICB, IQ
Assuntos: BIOLOGIA CELULAR, REGENERAÇÃO ÓSSEA, INFLAMAÇÃO, PROLIFERAÇÃO CELULAR, ANTI-INFLAMATÓRIOS, MIOBLASTOS, ADIPONECTINA, RNA MENSAGEIRO, CAMUNDONGOS
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MOSELE, Francielle Caroline et al. Muscle regeneration in adiponectin knockout mice showed early activation of anti‐inflammatory response with perturbations in myogenesis. Journal of Cellular Physiology, p. 1-11, 2020Tradução . . Disponível em: https://doi.org/10.1002/jcp.29547. Acesso em: 19 abr. 2024.
APA
Mosele, F. C., Barros, R. B. R. de, Teixeira, P. S. A., & Rosa Neto, J. C. (2020). Muscle regeneration in adiponectin knockout mice showed early activation of anti‐inflammatory response with perturbations in myogenesis. Journal of Cellular Physiology, 1-11. doi:10.1002/jcp.29547
NLM
Mosele FC, Barros RBR de, Teixeira PSA, Rosa Neto JC. Muscle regeneration in adiponectin knockout mice showed early activation of anti‐inflammatory response with perturbations in myogenesis [Internet]. Journal of Cellular Physiology. 2020 ; 1-11.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.29547
Vancouver
Mosele FC, Barros RBR de, Teixeira PSA, Rosa Neto JC. Muscle regeneration in adiponectin knockout mice showed early activation of anti‐inflammatory response with perturbations in myogenesis [Internet]. Journal of Cellular Physiology. 2020 ; 1-11.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.29547
Artigo de periodico
Endoplasmic reticulum distribution during bovine oocyte activation is regulated by protein kinase C via actin filaments (2020)
Feitosa, Weber Beringui ; Lopes, Everton; Visintin, Jose Antonio ; Assumpção, Mayra Elena Ortiz D'Ávila
Fonte: Journal of Cellular Physiology. Unidade: FMVZ
Assuntos: BOVINOS, OÓCITOS, PROTEÍNAS QUINASES, RETÍCULO ENDOPLASMÁTICO, CÁLCIO
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FEITOSA, Weber Beringui et al. Endoplasmic reticulum distribution during bovine oocyte activation is regulated by protein kinase C via actin filaments. Journal of Cellular Physiology, v. 235, n. 7/8, p. 5823-5834, 2020Tradução . . Disponível em: https://doi.org/10.1002/jcp.29516. Acesso em: 19 abr. 2024.
APA
Feitosa, W. B., Lopes, E., Visintin, J. A., & Assumpção, M. E. O. D. 'Á. (2020). Endoplasmic reticulum distribution during bovine oocyte activation is regulated by protein kinase C via actin filaments. Journal of Cellular Physiology, 235( 7/8), 5823-5834. doi:10.1002/jcp.29516
NLM
Feitosa WB, Lopes E, Visintin JA, Assumpção MEOD'Á. Endoplasmic reticulum distribution during bovine oocyte activation is regulated by protein kinase C via actin filaments [Internet]. Journal of Cellular Physiology. 2020 ; 235( 7/8): 5823-5834.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.29516
Vancouver
Feitosa WB, Lopes E, Visintin JA, Assumpção MEOD'Á. Endoplasmic reticulum distribution during bovine oocyte activation is regulated by protein kinase C via actin filaments [Internet]. Journal of Cellular Physiology. 2020 ; 235( 7/8): 5823-5834.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.29516
Artigo de periodico
Frizzled 6 disruption suppresses osteoblast differentiation induced by nanotopography through the canonical Wnt signaling pathway (2020)
Abuna, Rodrigo Paolo Flores ; Oliveira, Fabiola Singaretti de ; Adolpho, Letícia Faustino ; Fernandes, Roger Rodrigo ; Rosa, Adalberto Luiz ; Beloti, Márcio Mateus
Fonte: Journal of Cellular Physiology. Unidades: FORP, FMRP
Assuntos: CITOFISIOLOGIA, OSTEOBLASTO, TITÂNIO, DIFERENCIAÇÃO CELULAR
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ABUNA, Rodrigo Paolo Flores et al. Frizzled 6 disruption suppresses osteoblast differentiation induced by nanotopography through the canonical Wnt signaling pathway. Journal of Cellular Physiology, v. 235, n. 11, p. 8293-8303, 2020Tradução . . Disponível em: https://doi.org/10.1002/jcp.29674. Acesso em: 19 abr. 2024.
APA
Abuna, R. P. F., Oliveira, F. S. de, Adolpho, L. F., Fernandes, R. R., Rosa, A. L., & Beloti, M. M. (2020). Frizzled 6 disruption suppresses osteoblast differentiation induced by nanotopography through the canonical Wnt signaling pathway. Journal of Cellular Physiology, 235( 11), 8293-8303. doi:10.1002/jcp.29674
NLM
Abuna RPF, Oliveira FS de, Adolpho LF, Fernandes RR, Rosa AL, Beloti MM. Frizzled 6 disruption suppresses osteoblast differentiation induced by nanotopography through the canonical Wnt signaling pathway [Internet]. Journal of Cellular Physiology. 2020 ; 235( 11): 8293-8303.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.29674
Vancouver
Abuna RPF, Oliveira FS de, Adolpho LF, Fernandes RR, Rosa AL, Beloti MM. Frizzled 6 disruption suppresses osteoblast differentiation induced by nanotopography through the canonical Wnt signaling pathway [Internet]. Journal of Cellular Physiology. 2020 ; 235( 11): 8293-8303.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.29674
Artigo de periodico
Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R (2020)
Lino, Caroline Antunes ; Teixeira, Larissa de Bortoli ; Simões, Sarah Capelupe; Silva, Tábatha de Oliveira ; Diniz, Gabriela Placoná ; Costa Neto, Claudio Miguel da ; Barreto-Chaves, Maria Luiza Morais
Fonte: Journal of Cellular Physiology. Unidades: FMRP, ICB
Assuntos: ANATOMIA, HIPERTROFIA VENTRICULAR ESQUERDA, HORMÔNIOS TIREOIDIANOS, ANGIOTENSINA II, SISTEMA CARDIOVASCULAR, IMUNOHISTOQUÍMICA, PROTEÍNAS QUINASES
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LINO, Caroline Antunes et al. Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R. Journal of Cellular Physiology, v. 236, n. 6, p. 4640-4654, 2020Tradução . . Disponível em: https://doi.org/10.1002/jcp.30187. Acesso em: 19 abr. 2024.
APA
Lino, C. A., Teixeira, L. de B., Simões, S. C., Silva, T. de O., Diniz, G. P., Costa Neto, C. M. da, & Barreto-Chaves, M. L. M. (2020). Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R. Journal of Cellular Physiology, 236( 6), 4640-4654. doi:10.1002/jcp.30187
NLM
Lino CA, Teixeira L de B, Simões SC, Silva T de O, Diniz GP, Costa Neto CM da, Barreto-Chaves MLM. Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R [Internet]. Journal of Cellular Physiology. 2020 ; 236( 6): 4640-4654.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.30187
Vancouver
Lino CA, Teixeira L de B, Simões SC, Silva T de O, Diniz GP, Costa Neto CM da, Barreto-Chaves MLM. Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R [Internet]. Journal of Cellular Physiology. 2020 ; 236( 6): 4640-4654.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.30187
Artigo de periodico
Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB (2020)
Senger, Nathalia ; Parletta, Aline C. ; Marques, Bruno Vinicius Duarte ; Campagnole-Santos, Maria Jose ; Santos, Robson A. S. ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná ; Akamine, Eliana Hiromi
Fonte: Journal of Cellular Physiology. Unidades: ICB, FMRP
Assuntos: FARMACOLOGIA, ANATOMIA, DOENÇAS CARDIOVASCULARES, GLÂNDULA TIREOIDE, ANGIOTENSINAS, TRIIODOTIRONINA, INSUFICIÊNCIA CARDÍACA, ANTIOXIDANTES, CATALASE, ATIVAÇÃO ENZIMÁTICA, HORMÔNIOS TIREOIDIANOS
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SENGER, Nathalia et al. Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB. Journal of Cellular Physiology, p. 14 , 2020Tradução . . Disponível em: https://doi.org/10.1002/jcp.30069. Acesso em: 19 abr. 2024.
APA
Senger, N., Parletta, A. C., Marques, B. V. D., Campagnole-Santos, M. J., Santos, R. A. S., Barreto-Chaves, M. L. M., et al. (2020). Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB. Journal of Cellular Physiology, 14 . doi:10.1002/jcp.30069
NLM
Senger N, Parletta AC, Marques BVD, Campagnole-Santos MJ, Santos RAS, Barreto-Chaves MLM, Diniz GP, Akamine EH. Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB [Internet]. Journal of Cellular Physiology. 2020 ;14 .[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.30069
Vancouver
Senger N, Parletta AC, Marques BVD, Campagnole-Santos MJ, Santos RAS, Barreto-Chaves MLM, Diniz GP, Akamine EH. Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB [Internet]. Journal of Cellular Physiology. 2020 ;14 .[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.30069
Artigo de periodico
Laminin-derived peptide C16 regulates Tks expression and reactive oxygen species generation in human prostate cancer cells. (2020)
Santos, Livia Caire do; Silva, Suély Vieira da; Smuczek, Basilio; Siqueira, Adriane Souza de ; Augusto, Taize Machado ; Carvalho, Hernandes Fautino de ; Jaeger, Ruy Gastaldoni ; Freitas, Vanessa Morais ; Barbuto, José Alexandre Marzagão
Fonte: Journal of Cellular Physiology. Unidade: ICB
Assuntos: BIOLOGIA CELULAR, IMUNOLOGIA, CÉLULAS CULTIVADAS DE TUMOR, REGULAÇÃO GÊNICA, CITOMETRIA DE FLUXO, PRÓSTATA, FLUORESCÊNCIA, PEPTÍDEOS, NEOPLASIAS PROSTÁTICAS
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SANTOS, Livia Caire do et al. Laminin-derived peptide C16 regulates Tks expression and reactive oxygen species generation in human prostate cancer cells. Journal of Cellular Physiology, v. 235, p. 587-598, 2020Tradução . . Disponível em: https://doi.org/10.1002/jcp.28997. Acesso em: 19 abr. 2024.
APA
Santos, L. C. do, Silva, S. V. da, Smuczek, B., Siqueira, A. S. de, Augusto, T. M., Carvalho, H. F. de, et al. (2020). Laminin-derived peptide C16 regulates Tks expression and reactive oxygen species generation in human prostate cancer cells. Journal of Cellular Physiology, 235, 587-598. doi:10.1002/jcp.28997
NLM
Santos LC do, Silva SV da, Smuczek B, Siqueira AS de, Augusto TM, Carvalho HF de, Jaeger RG, Freitas VM, Barbuto JAM. Laminin-derived peptide C16 regulates Tks expression and reactive oxygen species generation in human prostate cancer cells. [Internet]. Journal of Cellular Physiology. 2020 ; 235 587-598.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.28997
Vancouver
Santos LC do, Silva SV da, Smuczek B, Siqueira AS de, Augusto TM, Carvalho HF de, Jaeger RG, Freitas VM, Barbuto JAM. Laminin-derived peptide C16 regulates Tks expression and reactive oxygen species generation in human prostate cancer cells. [Internet]. Journal of Cellular Physiology. 2020 ; 235 587-598.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.28997
Artigo de periodico
Selection of reference genes for quantitative real-time polymerase chain reaction studies in rat osteoblasts (2019)
Abuna, Rodrigo Paolo Flores ; Oliveira, Fabiola Singaretti de ; Ramos, Jaqueline Isadora Reis ; Lopes, Helena Bacha ; Freitas, Gileade Pereira ; Souza, Alann Thaffarell Portilho de ; Beloti, Márcio Mateus ; Rosa, Adalberto Luiz
Fonte: Journal of Cellular Physiology. Unidade: FORP
Assuntos: OSSO E OSSOS, RNA MENSAGEIRO, OSTEOBLASTO
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ABUNA, Rodrigo Paolo Flores et al. Selection of reference genes for quantitative real-time polymerase chain reaction studies in rat osteoblasts. Journal of Cellular Physiology, v. 234, n. 1, p. 749-756, 2019Tradução . . Disponível em: https://doi.org/10.1002/jcp.26886. Acesso em: 19 abr. 2024.
APA
Abuna, R. P. F., Oliveira, F. S. de, Ramos, J. I. R., Lopes, H. B., Freitas, G. P., Souza, A. T. P. de, et al. (2019). Selection of reference genes for quantitative real-time polymerase chain reaction studies in rat osteoblasts. Journal of Cellular Physiology, 234( 1), 749-756. doi:10.1002/jcp.26886
NLM
Abuna RPF, Oliveira FS de, Ramos JIR, Lopes HB, Freitas GP, Souza ATP de, Beloti MM, Rosa AL. Selection of reference genes for quantitative real-time polymerase chain reaction studies in rat osteoblasts [Internet]. Journal of Cellular Physiology. 2019 ; 234( 1): 749-756.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.26886
Vancouver
Abuna RPF, Oliveira FS de, Ramos JIR, Lopes HB, Freitas GP, Souza ATP de, Beloti MM, Rosa AL. Selection of reference genes for quantitative real-time polymerase chain reaction studies in rat osteoblasts [Internet]. Journal of Cellular Physiology. 2019 ; 234( 1): 749-756.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.26886
Artigo de periodico
Castration‐induced prostate epithelial cell apoptosis results from targeted oxidative stress attack of M1142‐macrophages (2019)
Barbosa, Guilherme O; Silva, Juliete A. f; Berti, Aline Siqueira ; Nishan, Umar; Ribeiro, Rafaela da Rosa; Oliveira, Silvia B. P; Baratti, Mariana O; Ferrucci, Danilo ; Santana, Julio C. O; Souza, Danilo Marchete Damas de; Bruni-Cardoso, Alexandre ; Augusto, Taize M ; Silva, Felipe Correa da; Vieira, Pedro M. Moraes; Machado, Dagmar Ruth Stach; Felisbino, Sergio L; Menezes, Gustavo B; Cesar, Carlos L; Carvalho, Hernandes F
Fonte: Journal of Cellular Physiology. Unidade: IQ
Assuntos: APOPTOSE, CÉLULAS EPITELIAIS, MACRÓFAGOS
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ABNT
BARBOSA, Guilherme O et al. Castration‐induced prostate epithelial cell apoptosis results from targeted oxidative stress attack of M1142‐macrophages. Journal of Cellular Physiology, v. 234, n. 10, p. 19048-19058, 2019Tradução . . Disponível em: https://doi.org/10.1002/jcp.28544. Acesso em: 19 abr. 2024.
APA
Barbosa, G. O., Silva, J. A. f, Berti, A. S., Nishan, U., Ribeiro, R. da R., Oliveira, S. B. P., et al. (2019). Castration‐induced prostate epithelial cell apoptosis results from targeted oxidative stress attack of M1142‐macrophages. Journal of Cellular Physiology, 234( 10), 19048-19058. doi:10.1002/jcp.28544
NLM
Barbosa GO, Silva JA f, Berti AS, Nishan U, Ribeiro R da R, Oliveira SBP, Baratti MO, Ferrucci D, Santana JCO, Souza DMD de, Bruni-Cardoso A, Augusto TM, Silva FC da, Vieira PMM, Machado DRS, Felisbino SL, Menezes GB, Cesar CL, Carvalho HF. Castration‐induced prostate epithelial cell apoptosis results from targeted oxidative stress attack of M1142‐macrophages [Internet]. Journal of Cellular Physiology. 2019 ; 234( 10): 19048-19058.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.28544
Vancouver
Barbosa GO, Silva JA f, Berti AS, Nishan U, Ribeiro R da R, Oliveira SBP, Baratti MO, Ferrucci D, Santana JCO, Souza DMD de, Bruni-Cardoso A, Augusto TM, Silva FC da, Vieira PMM, Machado DRS, Felisbino SL, Menezes GB, Cesar CL, Carvalho HF. Castration‐induced prostate epithelial cell apoptosis results from targeted oxidative stress attack of M1142‐macrophages [Internet]. Journal of Cellular Physiology. 2019 ; 234( 10): 19048-19058.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.28544
Artigo de periodico
Regulation of autophagy as a therapy for immunosenescence‐driven cancer and neurodegenerative diseases: the role of exercise (2019)
Batatinha, Helena Angelica Pereira ; Diniz, Tiego Aparecido ; Teixeira, Alexandre Abilio de Souza ; Krüger, Karsten; Rosa Neto, José Cesar
Fonte: Journal of Cellular Physiology. Unidade: ICB
Assuntos: BIOLOGIA CELULAR, DOENÇAS NEURODEGENERATIVAS, EXERCÍCIO FÍSICO, NEOPLASIAS, ENVELHECIMENTO, FATORES DE RISCO, INFLAMAÇÃO, HOMEOSTASE, PROTEÍNAS QUINASES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BATATINHA, Helena Angelica Pereira et al. Regulation of autophagy as a therapy for immunosenescence‐driven cancer and neurodegenerative diseases: the role of exercise. Journal of Cellular Physiology, v. 234, n. 9, p. 14883-14895, 2019Tradução . . Disponível em: https://doi.org/10.1002/jcp.28318. Acesso em: 19 abr. 2024.
APA
Batatinha, H. A. P., Diniz, T. A., Teixeira, A. A. de S., Krüger, K., & Rosa Neto, J. C. (2019). Regulation of autophagy as a therapy for immunosenescence‐driven cancer and neurodegenerative diseases: the role of exercise. Journal of Cellular Physiology, 234( 9), 14883-14895. doi:10.1002/jcp.28318
NLM
Batatinha HAP, Diniz TA, Teixeira AA de S, Krüger K, Rosa Neto JC. Regulation of autophagy as a therapy for immunosenescence‐driven cancer and neurodegenerative diseases: the role of exercise [Internet]. Journal of Cellular Physiology. 2019 ; 234( 9): 14883-14895.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.28318
Vancouver
Batatinha HAP, Diniz TA, Teixeira AA de S, Krüger K, Rosa Neto JC. Regulation of autophagy as a therapy for immunosenescence‐driven cancer and neurodegenerative diseases: the role of exercise [Internet]. Journal of Cellular Physiology. 2019 ; 234( 9): 14883-14895.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.28318
Artigo de periodico
The role of SDF1 in prostate epithelial morphogenesis (2019)
Barbosa, Guilherme Oliveira; Augusto, Taize Machado; Bruni-Cardoso, Alexandre ; Carvalho, Hernandes F
Fonte: Journal of Cellular Physiology. Unidade: IQ
Assuntos: PRÓSTATA, MORFOGÊNESE, EPITÉLIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BARBOSA, Guilherme Oliveira et al. The role of SDF1 in prostate epithelial morphogenesis. Journal of Cellular Physiology, v. 234, p. 6886-6807, 2019Tradução . . Disponível em: https://doi.org/10.1002/jcp.27447. Acesso em: 19 abr. 2024.
APA
Barbosa, G. O., Augusto, T. M., Bruni-Cardoso, A., & Carvalho, H. F. (2019). The role of SDF1 in prostate epithelial morphogenesis. Journal of Cellular Physiology, 234, 6886-6807. doi:10.1002/jcp.27447
NLM
Barbosa GO, Augusto TM, Bruni-Cardoso A, Carvalho HF. The role of SDF1 in prostate epithelial morphogenesis [Internet]. Journal of Cellular Physiology. 2019 ; 234 6886-6807.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.27447
Vancouver
Barbosa GO, Augusto TM, Bruni-Cardoso A, Carvalho HF. The role of SDF1 in prostate epithelial morphogenesis [Internet]. Journal of Cellular Physiology. 2019 ; 234 6886-6807.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.27447
Artigo de periodico
Interleukin‐10 responses from acute exercise in healthy subjects: a systematic review (2019)
Santos, Carolina Cabral ; Lima Junior, Edson Alves de ; Fernandes, Isabela Maia da Cruz; Pinto, Rafael Zambelli ; Rosa Neto, José Cesar ; Bishop, Nicolette Charllote ; Lira, Fábio Santos
Fonte: Journal of Cellular Physiology. Unidade: ICB
Assuntos: BIOLOGIA CELULAR, INTERLEUCINA 10, EXERCÍCIO FÍSICO, REVISÃO SISTEMÁTICA, INFLAMAÇÃO, ANTI-INFLAMATÓRIOS, CITOCINAS, ENSAIO CLÍNICO CONTROLADO RANDOMIZADO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Carolina Cabral et al. Interleukin‐10 responses from acute exercise in healthy subjects: a systematic review. Journal of Cellular Physiology, v. 234, n. 7, p. 9956-9965, 2019Tradução . . Disponível em: https://doi.org/10.1002/jcp.27920. Acesso em: 19 abr. 2024.
APA
Santos, C. C., Lima Junior, E. A. de, Fernandes, I. M. da C., Pinto, R. Z., Rosa Neto, J. C., Bishop, N. C., & Lira, F. S. (2019). Interleukin‐10 responses from acute exercise in healthy subjects: a systematic review. Journal of Cellular Physiology, 234( 7), 9956-9965. doi:10.1002/jcp.27920
NLM
Santos CC, Lima Junior EA de, Fernandes IM da C, Pinto RZ, Rosa Neto JC, Bishop NC, Lira FS. Interleukin‐10 responses from acute exercise in healthy subjects: a systematic review [Internet]. Journal of Cellular Physiology. 2019 ; 234( 7): 9956-9965.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.27920
Vancouver
Santos CC, Lima Junior EA de, Fernandes IM da C, Pinto RZ, Rosa Neto JC, Bishop NC, Lira FS. Interleukin‐10 responses from acute exercise in healthy subjects: a systematic review [Internet]. Journal of Cellular Physiology. 2019 ; 234( 7): 9956-9965.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.27920
Artigo de periodico
Exercise rescues the immune response fine‐tuned impaired by peroxisome proliferator‐activated receptors γ deletion in macrophages (2019)
Silveira, Loreana Sanches ; Batatinha, Helena Angélica Pereira; Castoldi, Angela; Câmara, Niels Olsen Saraiva ; Festuccia, William Tadeu Lara ; Souza, Camila Oliveira de ; Rosa Neto, José Cesar ; Lira, Fábio Santos
Fonte: Journal of Cellular Physiology. Unidade: ICB
Assuntos: MACRÓFAGOS, INFLAMAÇÃO, TREINAMENTO FÍSICO, CAMUNDONGOS, SISTEMA IMUNE, GLICOSE, HOMEOSTASE, PEROXISSOMOS
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ABNT
SILVEIRA, Loreana Sanches et al. Exercise rescues the immune response fine‐tuned impaired by peroxisome proliferator‐activated receptors γ deletion in macrophages. Journal of Cellular Physiology, v. 234, n. 4, p. 5241-5251, 2019Tradução . . Disponível em: https://doi.org/10.1002/jcp.27333. Acesso em: 19 abr. 2024.
APA
Silveira, L. S., Batatinha, H. A. P., Castoldi, A., Câmara, N. O. S., Festuccia, W. T. L., Souza, C. O. de, et al. (2019). Exercise rescues the immune response fine‐tuned impaired by peroxisome proliferator‐activated receptors γ deletion in macrophages. Journal of Cellular Physiology, 234( 4), 5241-5251. doi:10.1002/jcp.27333
NLM
Silveira LS, Batatinha HAP, Castoldi A, Câmara NOS, Festuccia WTL, Souza CO de, Rosa Neto JC, Lira FS. Exercise rescues the immune response fine‐tuned impaired by peroxisome proliferator‐activated receptors γ deletion in macrophages [Internet]. Journal of Cellular Physiology. 2019 ; 234( 4): 5241-5251.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.27333
Vancouver
Silveira LS, Batatinha HAP, Castoldi A, Câmara NOS, Festuccia WTL, Souza CO de, Rosa Neto JC, Lira FS. Exercise rescues the immune response fine‐tuned impaired by peroxisome proliferator‐activated receptors γ deletion in macrophages [Internet]. Journal of Cellular Physiology. 2019 ; 234( 4): 5241-5251.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.27333
Artigo de periodico
High fat diet reduces the expression of miRNA‐29b in heart and increases susceptibility of myocardium to ischemia/reperfusion injury (2019)
Guedes, Elaine Castilho; Silva, Ivson Bezerra da; Lima, Vanessa Morais; Miranda, Juliane Braga ; Albuquerque, Rudá Prestes e ; Ferreira, Julio Cesar Batista ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Fonte: Journal of Cellular Physiology. Unidade: ICB
Assuntos: ANATOMIA, OBESIDADE, CORAÇÃO, ANÓXIA, MIOCÁRDIO, ISQUEMIA DO MIOCÁRDIO ANIMAL, CAMUNDONGOS, CARDIOLOGIA VETERINÁRIA, DIETA ANIMAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUEDES, Elaine Castilho et al. High fat diet reduces the expression of miRNA‐29b in heart and increases susceptibility of myocardium to ischemia/reperfusion injury. Journal of Cellular Physiology, v. 234, n. 6, p. 9399-9407, 2019Tradução . . Disponível em: https://doi.org/10.1002/jcp.27624. Acesso em: 19 abr. 2024.
APA
Guedes, E. C., Silva, I. B. da, Lima, V. M., Miranda, J. B., Albuquerque, R. P. e, Ferreira, J. C. B., et al. (2019). High fat diet reduces the expression of miRNA‐29b in heart and increases susceptibility of myocardium to ischemia/reperfusion injury. Journal of Cellular Physiology, 234( 6), 9399-9407. doi:10.1002/jcp.27624
NLM
Guedes EC, Silva IB da, Lima VM, Miranda JB, Albuquerque RP e, Ferreira JCB, Barreto-Chaves MLM, Diniz GP. High fat diet reduces the expression of miRNA‐29b in heart and increases susceptibility of myocardium to ischemia/reperfusion injury [Internet]. Journal of Cellular Physiology. 2019 ; 234( 6): 9399-9407.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.27624
Vancouver
Guedes EC, Silva IB da, Lima VM, Miranda JB, Albuquerque RP e, Ferreira JCB, Barreto-Chaves MLM, Diniz GP. High fat diet reduces the expression of miRNA‐29b in heart and increases susceptibility of myocardium to ischemia/reperfusion injury [Internet]. Journal of Cellular Physiology. 2019 ; 234( 6): 9399-9407.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.27624
Artigo de periodico
Exercise increases Rho-kinase activity and insulin signaling in skeletal muscle (2018)
Muñoz, Vitor Rosetto ; Gaspar, Rafael Calais; Kuga, Gabriel Keine ; Rocha, Alisson Luiz da ; Crisol, Barbara Moreira ; Botezelli, José Diego; Baptista, Igor Luchini ; Mekary, Rania A.; Silva, Adelino Sanchez Ramos da ; Cintra, Dennys Esper ; Moura, Leandro Pereira de; Ropelle, Eduardo Rochete ; Pauli, José Rodrigo
Fonte: Journal of Cellular Physiology. Unidades: EEFERP, FMRP
Assuntos: HOMEOSTASE, GLICOSE, RESISTÊNCIA À INSULINA, EXERCÍCIO FÍSICO, MÚSCULO ESQUELÉTICO, TRANSDUÇÃO DE SINAL CELULAR, NATAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MUÑOZ, Vitor Rosetto et al. Exercise increases Rho-kinase activity and insulin signaling in skeletal muscle. Journal of Cellular Physiology, v. 233, n. 6, p. 4791-4800, 2018Tradução . . Disponível em: https://doi.org/10.1002/jcp.26278. Acesso em: 19 abr. 2024.
APA
Muñoz, V. R., Gaspar, R. C., Kuga, G. K., Rocha, A. L. da, Crisol, B. M., Botezelli, J. D., et al. (2018). Exercise increases Rho-kinase activity and insulin signaling in skeletal muscle. Journal of Cellular Physiology, 233( 6), 4791-4800. doi:10.1002/jcp.26278
NLM
Muñoz VR, Gaspar RC, Kuga GK, Rocha AL da, Crisol BM, Botezelli JD, Baptista IL, Mekary RA, Silva ASR da, Cintra DE, Moura LP de, Ropelle ER, Pauli JR. Exercise increases Rho-kinase activity and insulin signaling in skeletal muscle [Internet]. Journal of Cellular Physiology. 2018 ; 233( 6): 4791-4800.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.26278
Vancouver
Muñoz VR, Gaspar RC, Kuga GK, Rocha AL da, Crisol BM, Botezelli JD, Baptista IL, Mekary RA, Silva ASR da, Cintra DE, Moura LP de, Ropelle ER, Pauli JR. Exercise increases Rho-kinase activity and insulin signaling in skeletal muscle [Internet]. Journal of Cellular Physiology. 2018 ; 233( 6): 4791-4800.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.26278
Artigo de periodico
Integrated molecular, biochemical, and physiological assessment unravels key extraction method mediated influences on rat neonatal cardiomyocytes (2018)
Jensen, Leonardo S; Neri, Elida Adalgisa; Bassaneze, Vinicius; Oliveira, Nathalia C de Almeida; Dariolli, Rafael; Turaça, Lauro Thiago; Levy, Débora; Veronez, Douglas; Ferraz, Mariana Sacrini Ayres; Alencar, Adriano Mesquita; Bydlowski, Sérgio Paulo; Cestari, Idágene Aparecida; Krieger, José Eduardo
Fonte: Journal of Cellular Physiology. Unidades: FM, IF
Assuntos: CARDIOPATIAS, FISIOLOGIA MOLECULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
JENSEN, Leonardo S et al. Integrated molecular, biochemical, and physiological assessment unravels key extraction method mediated influences on rat neonatal cardiomyocytes. Journal of Cellular Physiology, v. 233, p. 5420–5430, 2018Tradução . . Disponível em: https://doi.org/10.1002/jcp.26380. Acesso em: 19 abr. 2024.
APA
Jensen, L. S., Neri, E. A., Bassaneze, V., Oliveira, N. C. de A., Dariolli, R., Turaça, L. T., et al. (2018). Integrated molecular, biochemical, and physiological assessment unravels key extraction method mediated influences on rat neonatal cardiomyocytes. Journal of Cellular Physiology, 233, 5420–5430. doi:10.1002/jcp.26380
NLM
Jensen LS, Neri EA, Bassaneze V, Oliveira NC de A, Dariolli R, Turaça LT, Levy D, Veronez D, Ferraz MSA, Alencar AM, Bydlowski SP, Cestari IA, Krieger JE. Integrated molecular, biochemical, and physiological assessment unravels key extraction method mediated influences on rat neonatal cardiomyocytes [Internet]. Journal of Cellular Physiology. 2018 ; 233 5420–5430.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.26380
Vancouver
Jensen LS, Neri EA, Bassaneze V, Oliveira NC de A, Dariolli R, Turaça LT, Levy D, Veronez D, Ferraz MSA, Alencar AM, Bydlowski SP, Cestari IA, Krieger JE. Integrated molecular, biochemical, and physiological assessment unravels key extraction method mediated influences on rat neonatal cardiomyocytes [Internet]. Journal of Cellular Physiology. 2018 ; 233 5420–5430.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1002/jcp.26380

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