Filtros : "ICB" "Calich, Vera Lúcia Garcia" Limpar

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  • Source: Journal of Infectious Diseases. Unidades: ICB, IB

    Subjects: PARACOCCIDIOIDOMICOSE, IMUNOLOGIA, MUTAÇÃO GENÉTICA, PREDISPOSIÇÃO GENÉTICA PARA DOENÇA, INFECÇÕES BACTERIANAS E MICOSES

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      SCHIMKE, Lena F et al. Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity. Journal of Infectious Diseases, v. 216, n. 12, p. 1623-1634, 2017Tradução . . Disponível em: https://doi.org/10.1093/infdis/jix522. Acesso em: 28 mar. 2024.
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      Schimke, L. F., Hibbard, J., Martinez-Barricarte, R., Khan, T. A., Cavalcante, R. de S., Oliveira-Junior, E. B. de, et al. (2017). Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity. Journal of Infectious Diseases, 216( 12), 1623-1634. doi:10.1093/infdis/jix522
    • NLM

      Schimke LF, Hibbard J, Martinez-Barricarte R, Khan TA, Cavalcante R de S, Oliveira-Junior EB de, França TT, Iqbal A, Yamamoto G, Arslanian C, Feriotti C, Costa TA, Bustamante J, Boisson-Dupuis S, Casanova J-L, Barbuto JAM, Zatz M, Mendes RP, Calich VLG, Ochs HD, Torgerson TR, Cabral-Marques O, Condino Neto A. Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity [Internet]. Journal of Infectious Diseases. 2017 ; 216( 12): 1623-1634.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1093/infdis/jix522
    • Vancouver

      Schimke LF, Hibbard J, Martinez-Barricarte R, Khan TA, Cavalcante R de S, Oliveira-Junior EB de, França TT, Iqbal A, Yamamoto G, Arslanian C, Feriotti C, Costa TA, Bustamante J, Boisson-Dupuis S, Casanova J-L, Barbuto JAM, Zatz M, Mendes RP, Calich VLG, Ochs HD, Torgerson TR, Cabral-Marques O, Condino Neto A. Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity [Internet]. Journal of Infectious Diseases. 2017 ; 216( 12): 1623-1634.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1093/infdis/jix522
  • Source: The Journal of Immunology. Unidades: ICB, FMRP

    Subjects: LEISHMANIA, FAGOCITOSE, ESPÉCIES REATIVAS DE OXIGÊNIO

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      LIMA JÚNIOR, Djalma de Souza et al. Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication. The Journal of Immunology, v. 199, n. 6, p. 2055-2068, 2017Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1700258. Acesso em: 28 mar. 2024.
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      Lima Júnior, D. de S., Mineo, T. W. P., Calich, V. L. G., & Zamboni, D. S. (2017). Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication. The Journal of Immunology, 199( 6), 2055-2068. doi:10.4049/jimmunol.1700258
    • NLM

      Lima Júnior D de S, Mineo TWP, Calich VLG, Zamboni DS. Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication [Internet]. The Journal of Immunology. 2017 ; 199( 6): 2055-2068.[citado 2024 mar. 28 ] Available from: https://doi.org/10.4049/jimmunol.1700258
    • Vancouver

      Lima Júnior D de S, Mineo TWP, Calich VLG, Zamboni DS. Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication [Internet]. The Journal of Immunology. 2017 ; 199( 6): 2055-2068.[citado 2024 mar. 28 ] Available from: https://doi.org/10.4049/jimmunol.1700258
  • Source: PLoS Pathogens. Unidade: ICB

    Assunto: IMUNOLOGIA

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      ARAÚJO, Eliseu Frank de et al. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner. PLoS Pathogens, v. 12, n. 12, p. 1-29, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1006115. Acesso em: 28 mar. 2024.
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      Araújo, E. F. de, Medeiros, D. H., Galdino, N. A. de L., Condino-Neto, A., Calich, V. L. G., & Loures, F. V. (2016). Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner. PLoS Pathogens, 12( 12), 1-29. doi:10.1371/journal.ppat.1006115
    • NLM

      Araújo EF de, Medeiros DH, Galdino NA de L, Condino-Neto A, Calich VLG, Loures FV. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner [Internet]. PLoS Pathogens. 2016 ; 12( 12): 1-29.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1371/journal.ppat.1006115
    • Vancouver

      Araújo EF de, Medeiros DH, Galdino NA de L, Condino-Neto A, Calich VLG, Loures FV. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner [Internet]. PLoS Pathogens. 2016 ; 12( 12): 1-29.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1371/journal.ppat.1006115
  • Unidade: ICB

    Subjects: CAMUNDONGOS, CITOCINAS, IMUNIDADE, PARACOCCIDIOIDOMICOSE, MICOSES (IMUNOLOGIA)

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      COSTA, Tânia Alves da. A função da IL-10 na paracoccidioidomicose pulmonar murina. 2010. Tese (Doutorado) – Universidade de São Paulo, São Paulo, 2010. Disponível em: https://teses.usp.br/teses/disponiveis/42/42133/tde-16122010-113911/. Acesso em: 28 mar. 2024.
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      Costa, T. A. da. (2010). A função da IL-10 na paracoccidioidomicose pulmonar murina (Tese (Doutorado). Universidade de São Paulo, São Paulo. Recuperado de https://teses.usp.br/teses/disponiveis/42/42133/tde-16122010-113911/
    • NLM

      Costa TA da. A função da IL-10 na paracoccidioidomicose pulmonar murina [Internet]. 2010 ;[citado 2024 mar. 28 ] Available from: https://teses.usp.br/teses/disponiveis/42/42133/tde-16122010-113911/
    • Vancouver

      Costa TA da. A função da IL-10 na paracoccidioidomicose pulmonar murina [Internet]. 2010 ;[citado 2024 mar. 28 ] Available from: https://teses.usp.br/teses/disponiveis/42/42133/tde-16122010-113911/
  • Source: Infection and Immunity. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FELONATO, Maíra et al. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis. Infection and Immunity, v. 78, n. 11, p. 4922-4935, 2010Tradução . . Disponível em: https://doi.org/10.1128/IAI.00297-10. Acesso em: 28 mar. 2024.
    • APA

      Felonato, M., Pina, A., Bernardino, S., Loures, F. V., Araújo, E. F. de, & Calich, V. L. G. (2010). CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis. Infection and Immunity, 78( 11), 4922-4935. doi:10.1128/IAI.00297-10
    • NLM

      Felonato M, Pina A, Bernardino S, Loures FV, Araújo EF de, Calich VLG. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis [Internet]. Infection and Immunity. 2010 ; 78( 11): 4922-4935.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1128/IAI.00297-10
    • Vancouver

      Felonato M, Pina A, Bernardino S, Loures FV, Araújo EF de, Calich VLG. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis [Internet]. Infection and Immunity. 2010 ; 78( 11): 4922-4935.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1128/IAI.00297-10
  • Source: Infection and Immunity. Unidade: ICB

    Assunto: IMUNOLOGIA

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      LOURES, Flávio Vieira et al. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity, v. 78, n. 3, p. 1078-1088, 2010Tradução . . Acesso em: 28 mar. 2024.
    • APA

      Loures, F. V., Pina, A., Felonato, M., Araújo, E. F. de, Leite, K. R. M., & Calich, V. L. G. (2010). Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity, 78( 3), 1078-1088.
    • NLM

      Loures FV, Pina A, Felonato M, Araújo EF de, Leite KRM, Calich VLG. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity. 2010 ; 78( 3): 1078-1088.[citado 2024 mar. 28 ]
    • Vancouver

      Loures FV, Pina A, Felonato M, Araújo EF de, Leite KRM, Calich VLG. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity. 2010 ; 78( 3): 1078-1088.[citado 2024 mar. 28 ]
  • Source: Molecular Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      TOLEDO, Renan G. et al. Mannose-binding lectin complement pathway plays a key role in complemente activation by Paracoccidioides brasiliensis. Molecular Immunology, v. 48, n. 1-3, p. 26-36, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.molimm.2010.09.015. Acesso em: 28 mar. 2024.
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      Toledo, R. G., Silva, W. D. da, Calich, V. L. G., & Kipnis, T. L. (2010). Mannose-binding lectin complement pathway plays a key role in complemente activation by Paracoccidioides brasiliensis. Molecular Immunology, 48( 1-3), 26-36. doi:10.1016/j.molimm.2010.09.015
    • NLM

      Toledo RG, Silva WD da, Calich VLG, Kipnis TL. Mannose-binding lectin complement pathway plays a key role in complemente activation by Paracoccidioides brasiliensis [Internet]. Molecular Immunology. 2010 ; 48( 1-3): 26-36.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.molimm.2010.09.015
    • Vancouver

      Toledo RG, Silva WD da, Calich VLG, Kipnis TL. Mannose-binding lectin complement pathway plays a key role in complemente activation by Paracoccidioides brasiliensis [Internet]. Molecular Immunology. 2010 ; 48( 1-3): 26-36.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.molimm.2010.09.015
  • Source: Abstracts. Conference titles: Latin American Congress Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      LOURES, Flavio Vieira e CALICH, Vera Lúcia Garcia. TLR4 is used by a fungal pathogen to infect macrophages but also to induce protective immunity. 2009, Anais.. Viña de Mar: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2009. . Acesso em: 28 mar. 2024.
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      Loures, F. V., & Calich, V. L. G. (2009). TLR4 is used by a fungal pathogen to infect macrophages but also to induce protective immunity. In Abstracts. Viña de Mar: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Loures FV, Calich VLG. TLR4 is used by a fungal pathogen to infect macrophages but also to induce protective immunity. Abstracts. 2009 ;[citado 2024 mar. 28 ]
    • Vancouver

      Loures FV, Calich VLG. TLR4 is used by a fungal pathogen to infect macrophages but also to induce protective immunity. Abstracts. 2009 ;[citado 2024 mar. 28 ]
  • Source: Journal of Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      LOURES, Flávio Vieira et al. TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection. Journal of Immunology, v. 183, n. 2, p. 1279-90, 2009Tradução . . Acesso em: 28 mar. 2024.
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      Loures, F. V., Pina, A., Felonato, M., & Calich, V. L. G. (2009). TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection. Journal of Immunology, 183( 2), 1279-90.
    • NLM

      Loures FV, Pina A, Felonato M, Calich VLG. TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection. Journal of Immunology. 2009 ; 183( 2): 1279-90.[citado 2024 mar. 28 ]
    • Vancouver

      Loures FV, Pina A, Felonato M, Calich VLG. TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection. Journal of Immunology. 2009 ; 183( 2): 1279-90.[citado 2024 mar. 28 ]
  • Source: Resumos. Conference titles: Congress of the Brazilian Society for Immunology - SBI. Unidade: ICB

    Assunto: IMUNOLOGIA

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      TUNES, C. F. e CALICH, Vera Lúcia Garcia. Mannose receptors play a distinct role in the interaction of P. brasiliensis cells with murine macrophages of resistant and susceptible mice. 2008, Anais.. Ribeirão Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2008. . Acesso em: 28 mar. 2024.
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      Tunes, C. F., & Calich, V. L. G. (2008). Mannose receptors play a distinct role in the interaction of P. brasiliensis cells with murine macrophages of resistant and susceptible mice. In Resumos. Ribeirão Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Tunes CF, Calich VLG. Mannose receptors play a distinct role in the interaction of P. brasiliensis cells with murine macrophages of resistant and susceptible mice. Resumos. 2008 ;[citado 2024 mar. 28 ]
    • Vancouver

      Tunes CF, Calich VLG. Mannose receptors play a distinct role in the interaction of P. brasiliensis cells with murine macrophages of resistant and susceptible mice. Resumos. 2008 ;[citado 2024 mar. 28 ]
  • Source: Resumos. Conference titles: Congress of the Brazilian Society for Immunology - SBI. Unidade: ICB

    Assunto: IMUNOLOGIA

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      BERNARDINO, Simone e CALICH, Vera Lúcia Garcia. iNOS KO mice develop a protective T cell immunity against pulmonary paracoccidioidomycosis infection which can be abolished by TNF'alfa' neutralization. 2008, Anais.. Ribeirão Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2008. . Acesso em: 28 mar. 2024.
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      Bernardino, S., & Calich, V. L. G. (2008). iNOS KO mice develop a protective T cell immunity against pulmonary paracoccidioidomycosis infection which can be abolished by TNF'alfa' neutralization. In Resumos. Ribeirão Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Bernardino S, Calich VLG. iNOS KO mice develop a protective T cell immunity against pulmonary paracoccidioidomycosis infection which can be abolished by TNF'alfa' neutralization. Resumos. 2008 ;[citado 2024 mar. 28 ]
    • Vancouver

      Bernardino S, Calich VLG. iNOS KO mice develop a protective T cell immunity against pulmonary paracoccidioidomycosis infection which can be abolished by TNF'alfa' neutralization. Resumos. 2008 ;[citado 2024 mar. 28 ]
  • Source: Journal of Leukocyte Biology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      PINA, Adriana e BERNARDINO, Simone e CALICH, Vera Lúcia Garcia. Alveolar macrophages from susceptible mice are more competent than those of resistant mice to control initial Paracoccidioides brasiliensis infection. Journal of Leukocyte Biology, v. 83, n. 5, p. 1088-1099, 2008Tradução . . Disponível em: https://doi.org/10.1189/jlb.1107738. Acesso em: 28 mar. 2024.
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      Pina, A., Bernardino, S., & Calich, V. L. G. (2008). Alveolar macrophages from susceptible mice are more competent than those of resistant mice to control initial Paracoccidioides brasiliensis infection. Journal of Leukocyte Biology, 83( 5), 1088-1099. doi:10.1189/jlb.1107738
    • NLM

      Pina A, Bernardino S, Calich VLG. Alveolar macrophages from susceptible mice are more competent than those of resistant mice to control initial Paracoccidioides brasiliensis infection [Internet]. Journal of Leukocyte Biology. 2008 ; 83( 5): 1088-1099.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1189/jlb.1107738
    • Vancouver

      Pina A, Bernardino S, Calich VLG. Alveolar macrophages from susceptible mice are more competent than those of resistant mice to control initial Paracoccidioides brasiliensis infection [Internet]. Journal of Leukocyte Biology. 2008 ; 83( 5): 1088-1099.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1189/jlb.1107738
  • Source: Biomédica. Conference titles: Proceedings of the International Congress on Paracoccidioidomycosis. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FELONATO, M. e CALICH, Vera Lúcia Garcia. CD28 costimulatory molecule deficiency results in more severe Paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 28 mar. 2024. , 2008
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      Felonato, M., & Calich, V. L. G. (2008). CD28 costimulatory molecule deficiency results in more severe Paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Felonato M, Calich VLG. CD28 costimulatory molecule deficiency results in more severe Paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology. Biomédica. 2008 ; 28[citado 2024 mar. 28 ]
    • Vancouver

      Felonato M, Calich VLG. CD28 costimulatory molecule deficiency results in more severe Paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology. Biomédica. 2008 ; 28[citado 2024 mar. 28 ]
  • Source: Resumos. Conference titles: Congress of the Brazilian Society for Immunology - SBI. Unidade: ICB

    Assunto: IMUNOLOGIA

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      ARAUJO, Eliseu Frank de e CALICH, Vera Lúcia Garcia. Susceptible and resistant mice to P. Brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth. 2008, Anais.. Ribeirão Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2008. . Acesso em: 28 mar. 2024.
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      Araujo, E. F. de, & Calich, V. L. G. (2008). Susceptible and resistant mice to P. Brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth. In Resumos. Ribeirão Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Araujo EF de, Calich VLG. Susceptible and resistant mice to P. Brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth. Resumos. 2008 ;[citado 2024 mar. 28 ]
    • Vancouver

      Araujo EF de, Calich VLG. Susceptible and resistant mice to P. Brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth. Resumos. 2008 ;[citado 2024 mar. 28 ]
  • Source: FEMS Immunology and Medical Microbiology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      CALICH, Vera Lúcia Garcia et al. Toll-like receptors and fungal infections: the role of TLR2, TLR4 and MyD88 in paracoccidioidomycosis. FEMS Immunology and Medical Microbiology, v. 53, n. 1, p. 1-7, 2008Tradução . . Disponível em: https://doi.org/10.1111/j.1574-695x.2008.00378.x. Acesso em: 28 mar. 2024.
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      Calich, V. L. G., Pina, A., Felonato, M., Bernardino, S., Costa, T. A., & Loures, F. V. (2008). Toll-like receptors and fungal infections: the role of TLR2, TLR4 and MyD88 in paracoccidioidomycosis. FEMS Immunology and Medical Microbiology, 53( 1), 1-7. doi:10.1111/j.1574-695x.2008.00378.x
    • NLM

      Calich VLG, Pina A, Felonato M, Bernardino S, Costa TA, Loures FV. Toll-like receptors and fungal infections: the role of TLR2, TLR4 and MyD88 in paracoccidioidomycosis [Internet]. FEMS Immunology and Medical Microbiology. 2008 ; 53( 1): 1-7.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1111/j.1574-695x.2008.00378.x
    • Vancouver

      Calich VLG, Pina A, Felonato M, Bernardino S, Costa TA, Loures FV. Toll-like receptors and fungal infections: the role of TLR2, TLR4 and MyD88 in paracoccidioidomycosis [Internet]. FEMS Immunology and Medical Microbiology. 2008 ; 53( 1): 1-7.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1111/j.1574-695x.2008.00378.x
  • Source: Resumos. Conference titles: Congress of the Brazilian Society for Immunology - SBI. Unidade: ICB

    Assunto: IMUNOLOGIA

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      PINA, A. e CALICH, Vera Lúcia Garcia. Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice. 2008, Anais.. Ribeirão Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2008. . Acesso em: 28 mar. 2024.
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      Pina, A., & Calich, V. L. G. (2008). Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice. In Resumos. Ribeirão Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Pina A, Calich VLG. Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice. Resumos. 2008 ;[citado 2024 mar. 28 ]
    • Vancouver

      Pina A, Calich VLG. Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice. Resumos. 2008 ;[citado 2024 mar. 28 ]
  • Source: Biomédica. Conference titles: Proceedings of the International Congress on Paracoccidioidomycosis. Unidade: ICB

    Assunto: IMUNOLOGIA

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      LOURES, Flávio V. e CALICH, Vera Lúcia Garcia. Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 28 mar. 2024. , 2008
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      Loures, F. V., & Calich, V. L. G. (2008). Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Loures FV, Calich VLG. Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells. Biomédica. 2008 ; 28[citado 2024 mar. 28 ]
    • Vancouver

      Loures FV, Calich VLG. Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells. Biomédica. 2008 ; 28[citado 2024 mar. 28 ]
  • Source: Biomédica. Conference titles: Proceedings of the International Congress on Paracoccidioidomycosis. Unidade: ICB

    Assunto: IMUNOLOGIA

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      CALICH, Vera Lúcia Garcia e LOURES, Flávio V. Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 28 mar. 2024. , 2008
    • APA

      Calich, V. L. G., & Loures, F. V. (2008). Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Calich VLG, Loures FV. Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis. Biomédica. 2008 ; 28[citado 2024 mar. 28 ]
    • Vancouver

      Calich VLG, Loures FV. Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis. Biomédica. 2008 ; 28[citado 2024 mar. 28 ]
  • Source: Mycopathologia. Unidade: ICB

    Assunto: IMUNOLOGIA

    Acesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      CALICH, Vera Lúcia Garcia et al. Innate immunity to Paracoccidioides brasiliensis infection. Mycopathologia, v. 165, n. 4-5, p. 223-236, 2008Tradução . . Disponível em: https://doi.org/10.1007/s11046-007-9048-1. Acesso em: 28 mar. 2024.
    • APA

      Calich, V. L. G., Costa, T. A. da, Felonato, M., Arruda, C., Bernardino, S., Loures, F. V., et al. (2008). Innate immunity to Paracoccidioides brasiliensis infection. Mycopathologia, 165( 4-5), 223-236. doi:10.1007/s11046-007-9048-1
    • NLM

      Calich VLG, Costa TA da, Felonato M, Arruda C, Bernardino S, Loures FV, Ribeiro LRR, Valente-Ferreira R de C, Pina A. Innate immunity to Paracoccidioides brasiliensis infection [Internet]. Mycopathologia. 2008 ; 165( 4-5): 223-236.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1007/s11046-007-9048-1
    • Vancouver

      Calich VLG, Costa TA da, Felonato M, Arruda C, Bernardino S, Loures FV, Ribeiro LRR, Valente-Ferreira R de C, Pina A. Innate immunity to Paracoccidioides brasiliensis infection [Internet]. Mycopathologia. 2008 ; 165( 4-5): 223-236.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1007/s11046-007-9048-1
  • Source: Biomédica. Conference titles: Proceedings of the International Congress on Paracoccidioidomycosis. Unidade: ICB

    Assunto: IMUNOLOGIA

    How to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      PINA, A. e CALICH, Vera Lúcia Garcia. Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmocytoid subpopulation which induces a more severe pulmonary infection when transferred to resistant mice. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 28 mar. 2024. , 2008
    • APA

      Pina, A., & Calich, V. L. G. (2008). Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmocytoid subpopulation which induces a more severe pulmonary infection when transferred to resistant mice. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Pina A, Calich VLG. Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmocytoid subpopulation which induces a more severe pulmonary infection when transferred to resistant mice. Biomédica. 2008 ; 28[citado 2024 mar. 28 ]
    • Vancouver

      Pina A, Calich VLG. Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmocytoid subpopulation which induces a more severe pulmonary infection when transferred to resistant mice. Biomédica. 2008 ; 28[citado 2024 mar. 28 ]

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