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  • Source: Experimental and Toxicologic Pathology. Unidades: FM, IF

    Subjects: POLUIÇÃO ATMOSFÉRICA, DOENÇAS RESPIRATÓRIAS, METAIS, PROTEÍNAS QUINASES, COMPOSTOS INORGÂNICOS

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      SERIANI, Robson et al. Enriched inorganic compounds in diesel exhaust particles induce mitogen-activated protein kinase activation, cytoskeleton instability, and cytotoxicity in human bronchial epithelial cells. Experimental and Toxicologic Pathology, v. 67, n. 5-6, p. 323-329, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.etp.2015.02.004. Acesso em: 28 mar. 2024.
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      Seriani, R., Junqueira, M. S., Carvalho-Sousa, C. E., Arruda, A. C. T., Martinez, D., Alencar, A. M., et al. (2015). Enriched inorganic compounds in diesel exhaust particles induce mitogen-activated protein kinase activation, cytoskeleton instability, and cytotoxicity in human bronchial epithelial cells. Experimental and Toxicologic Pathology, 67( 5-6), 323-329. doi:10.1016/j.etp.2015.02.004
    • NLM

      Seriani R, Junqueira MS, Carvalho-Sousa CE, Arruda ACT, Martinez D, Alencar AM, Garippo AL, Brito JM, Martins MA, Saldiva PHN, Negri EM, Mauad T, Macchione M. Enriched inorganic compounds in diesel exhaust particles induce mitogen-activated protein kinase activation, cytoskeleton instability, and cytotoxicity in human bronchial epithelial cells [Internet]. Experimental and Toxicologic Pathology. 2015 ; 67( 5-6): 323-329.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.etp.2015.02.004
    • Vancouver

      Seriani R, Junqueira MS, Carvalho-Sousa CE, Arruda ACT, Martinez D, Alencar AM, Garippo AL, Brito JM, Martins MA, Saldiva PHN, Negri EM, Mauad T, Macchione M. Enriched inorganic compounds in diesel exhaust particles induce mitogen-activated protein kinase activation, cytoskeleton instability, and cytotoxicity in human bronchial epithelial cells [Internet]. Experimental and Toxicologic Pathology. 2015 ; 67( 5-6): 323-329.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.etp.2015.02.004
  • Source: Peptides. Unidades: FM, ICB

    Subjects: PEPTÍDEOS, MELANOMA, APOPTOSE

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      AZEVEDO, Ricardo A. de et al. Mastoparan induces apoptosis in B16F10-Nex2 melanoma cells via the intrinsic mitochondrial pathway and displays antitumor activity in vivo. Peptides, v. 68, p. 113-119, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.peptides.2014.09.024. Acesso em: 28 mar. 2024.
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      Azevedo, R. A. de, Figueiredo, C. R., Ferreira, A. K., Matsuo, A. L., Massaoka, M. H., Girola, N., et al. (2015). Mastoparan induces apoptosis in B16F10-Nex2 melanoma cells via the intrinsic mitochondrial pathway and displays antitumor activity in vivo. Peptides, 68, 113-119. doi:10.1016/j.peptides.2014.09.024
    • NLM

      Azevedo RA de, Figueiredo CR, Ferreira AK, Matsuo AL, Massaoka MH, Girola N, Auada AVV, Farias CF, Pasqualoto KFM, Rodrigues CP, Barbuto JAM, Levy D, Bydlowski SP, Sá-Junior PL de, Travassos LR, Lebrun I. Mastoparan induces apoptosis in B16F10-Nex2 melanoma cells via the intrinsic mitochondrial pathway and displays antitumor activity in vivo [Internet]. Peptides. 2015 ; 68 113-119.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.peptides.2014.09.024
    • Vancouver

      Azevedo RA de, Figueiredo CR, Ferreira AK, Matsuo AL, Massaoka MH, Girola N, Auada AVV, Farias CF, Pasqualoto KFM, Rodrigues CP, Barbuto JAM, Levy D, Bydlowski SP, Sá-Junior PL de, Travassos LR, Lebrun I. Mastoparan induces apoptosis in B16F10-Nex2 melanoma cells via the intrinsic mitochondrial pathway and displays antitumor activity in vivo [Internet]. Peptides. 2015 ; 68 113-119.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.peptides.2014.09.024
  • Source: Experimental and Toxicologic Pathology. Unidade: FM

    Subjects: INFLAMAÇÃO (ETIOLOGIA), POLUIÇÃO AMBIENTAL, PULMÃO (FISIOPATOLOGIA)

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      FERRARO, S. et al. Air particulate matter exacerbates lung response on Sjögren's Syndrome animals. Experimental and Toxicologic Pathology, v. 67, n. 2, p. 125-131, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.etp.2014.10.007. Acesso em: 28 mar. 2024.
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      Ferraro, S., Orona, N., Villalón, L., Saldiva, P. H. N., Tasat, D. R., & Berra, A. (2015). Air particulate matter exacerbates lung response on Sjögren's Syndrome animals. Experimental and Toxicologic Pathology, 67( 2), 125-131. doi:10.1016/j.etp.2014.10.007
    • NLM

      Ferraro S, Orona N, Villalón L, Saldiva PHN, Tasat DR, Berra A. Air particulate matter exacerbates lung response on Sjögren's Syndrome animals [Internet]. Experimental and Toxicologic Pathology. 2015 ; 67( 2): 125-131.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.etp.2014.10.007
    • Vancouver

      Ferraro S, Orona N, Villalón L, Saldiva PHN, Tasat DR, Berra A. Air particulate matter exacerbates lung response on Sjögren's Syndrome animals [Internet]. Experimental and Toxicologic Pathology. 2015 ; 67( 2): 125-131.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.etp.2014.10.007
  • Source: Endocrinology. Unidade: ICB

    Subjects: FISIOLOGIA, SODIO, IONS, HORMÔNIOS TIREOIDIANOS, IODO

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      SERRANO-NASCIMENTO, Caroline et al. The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway. Endocrinology, v. 155, n. 3, p. 1145-1156, 2014Tradução . . Disponível em: https://doi.org/10.1210/en.2013-1665. Acesso em: 28 mar. 2024.
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      Serrano-Nascimento, C., Teixeira, S. da S., Nachbar, R. T., Nicola, J. P., Masini-Repiso, A. M., & Nunes, M. T. (2014). The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway. Endocrinology, 155( 3), 1145-1156. doi:10.1210/en.2013-1665
    • NLM

      Serrano-Nascimento C, Teixeira S da S, Nachbar RT, Nicola JP, Masini-Repiso AM, Nunes MT. The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway [Internet]. Endocrinology. 2014 ; 155( 3): 1145-1156.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/en.2013-1665
    • Vancouver

      Serrano-Nascimento C, Teixeira S da S, Nachbar RT, Nicola JP, Masini-Repiso AM, Nunes MT. The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway [Internet]. Endocrinology. 2014 ; 155( 3): 1145-1156.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/en.2013-1665
  • Source: Peptides. Unidade: FM

    Subjects: SEPSE (COMPLICAÇÕES;TERAPIA), PROSTAGLANDINAS (APLICAÇÕES TERAPÊUTICAS), NEUROPEPTÍDEOS, INFLAMAÇÃO (TERAPIA;ETIOLOGIA)

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      SILVA, Fabiano Pinheiro da e MACHADO, Marcel Cerqueira César e VELASCO, Irineu Tadeu. Neuropeptides in sepsis: From brain pathology to systemic inflammation. Peptides, v. 44, n. 1, p. 135-138, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.peptides.2013.03.029. Acesso em: 28 mar. 2024.
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      Silva, F. P. da, Machado, M. C. C., & Velasco, I. T. (2013). Neuropeptides in sepsis: From brain pathology to systemic inflammation. Peptides, 44( 1), 135-138. doi:10.1016/j.peptides.2013.03.029
    • NLM

      Silva FP da, Machado MCC, Velasco IT. Neuropeptides in sepsis: From brain pathology to systemic inflammation [Internet]. Peptides. 2013 ; 44( 1): 135-138.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.peptides.2013.03.029
    • Vancouver

      Silva FP da, Machado MCC, Velasco IT. Neuropeptides in sepsis: From brain pathology to systemic inflammation [Internet]. Peptides. 2013 ; 44( 1): 135-138.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.peptides.2013.03.029
  • Source: Prostit. Unidade: ICB

    Assunto: PARASITOLOGIA

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      BORGHESAN, Tarcilla Corrente et al. Molecular phylogenetic redefinition of herpetomonas (Kinetoplastea, Trypanosomatidae), a genus of insect parasites associated with flies. Prostit, v. 164, n. 1, p. 129-152, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.protis.2012.06.001. Acesso em: 28 mar. 2024.
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      Borghesan, T. C., Ferreira, R. C., Takata, C. S. de A., Campaner, M., Borda, C. C., Paiva, F., et al. (2013). Molecular phylogenetic redefinition of herpetomonas (Kinetoplastea, Trypanosomatidae), a genus of insect parasites associated with flies. Prostit, 164( 1), 129-152. doi:10.1016/j.protis.2012.06.001
    • NLM

      Borghesan TC, Ferreira RC, Takata CS de A, Campaner M, Borda CC, Paiva F, Milder RV, Teixeira MMG, Camargo EFP de. Molecular phylogenetic redefinition of herpetomonas (Kinetoplastea, Trypanosomatidae), a genus of insect parasites associated with flies [Internet]. Prostit. 2013 ; 164( 1): 129-152.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.protis.2012.06.001
    • Vancouver

      Borghesan TC, Ferreira RC, Takata CS de A, Campaner M, Borda CC, Paiva F, Milder RV, Teixeira MMG, Camargo EFP de. Molecular phylogenetic redefinition of herpetomonas (Kinetoplastea, Trypanosomatidae), a genus of insect parasites associated with flies [Internet]. Prostit. 2013 ; 164( 1): 129-152.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.protis.2012.06.001
  • Source: Veterinary Parasitology. Unidade: ICB

    Assunto: PARASITOLOGIA

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      RODRIGUES, Carla M. F. et al. Follicular degeneration in the ovaries of goats experimentally infected with Trypanosoma vivax from the Brazilian semi-arid region. Veterinary Parasitology, v. 191, n. 1-2, p. 146-153, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.vetpar.2012.08.001. Acesso em: 28 mar. 2024.
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      Rodrigues, C. M. F., Olinda, R. G., Silva, T. M. F., Vale, R. G., Silva, A. E. da, Lima, G. L., et al. (2013). Follicular degeneration in the ovaries of goats experimentally infected with Trypanosoma vivax from the Brazilian semi-arid region. Veterinary Parasitology, 191( 1-2), 146-153. doi:10.1016/j.vetpar.2012.08.001
    • NLM

      Rodrigues CMF, Olinda RG, Silva TMF, Vale RG, Silva AE da, Lima GL, Garcia Perez HA, Teixeira MMG, Batista JS. Follicular degeneration in the ovaries of goats experimentally infected with Trypanosoma vivax from the Brazilian semi-arid region [Internet]. Veterinary Parasitology. 2013 ; 191( 1-2): 146-153.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.vetpar.2012.08.001
    • Vancouver

      Rodrigues CMF, Olinda RG, Silva TMF, Vale RG, Silva AE da, Lima GL, Garcia Perez HA, Teixeira MMG, Batista JS. Follicular degeneration in the ovaries of goats experimentally infected with Trypanosoma vivax from the Brazilian semi-arid region [Internet]. Veterinary Parasitology. 2013 ; 191( 1-2): 146-153.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.vetpar.2012.08.001
  • Source: Regulatory Peptides. Unidade: ICB

    Assunto: FISIOLOGIA

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      ALVES, E. S. et al. Angiotensin II-induced JNK activation is mediated by NAD(P)H oxidase in isolated rat pancreatic islets. Regulatory Peptides, v. 175, n. 1-3, p. 1-6, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.regpep.2012.01.003. Acesso em: 28 mar. 2024.
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      Alves, E. S., Haidar, A. A., Quadros, C. D., Carvalho, D. S., Morgan, D., Rocha, M. S., et al. (2012). Angiotensin II-induced JNK activation is mediated by NAD(P)H oxidase in isolated rat pancreatic islets. Regulatory Peptides, 175( 1-3), 1-6. doi:10.1016/j.regpep.2012.01.003
    • NLM

      Alves ES, Haidar AA, Quadros CD, Carvalho DS, Morgan D, Rocha MS, Curi R, Carpinelli AR, Hirata AE. Angiotensin II-induced JNK activation is mediated by NAD(P)H oxidase in isolated rat pancreatic islets [Internet]. Regulatory Peptides. 2012 ; 175( 1-3): 1-6.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.regpep.2012.01.003
    • Vancouver

      Alves ES, Haidar AA, Quadros CD, Carvalho DS, Morgan D, Rocha MS, Curi R, Carpinelli AR, Hirata AE. Angiotensin II-induced JNK activation is mediated by NAD(P)H oxidase in isolated rat pancreatic islets [Internet]. Regulatory Peptides. 2012 ; 175( 1-3): 1-6.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.regpep.2012.01.003
  • Source: Toxicon. Unidade: ICB

    Assunto: IMUNOLOGIA

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      MOREIRA, Vanessa et al. Local inflammatory events induced by Bothrops atrox snake venom and the release of distinct classes of inflammatory mediators. Toxicon, v. 60, n. 1, p. 12-20, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.toxicon.2012.03.004. Acesso em: 28 mar. 2024.
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      Moreira, V., Dos-Santos, M. C., Nascimento, N. G., Silva, H. B. da, Fernandes, C. M., Lima, M. R. D. 'I., & Teixeira, C. (2012). Local inflammatory events induced by Bothrops atrox snake venom and the release of distinct classes of inflammatory mediators. Toxicon, 60( 1), 12-20. doi:10.1016/j.toxicon.2012.03.004
    • NLM

      Moreira V, Dos-Santos MC, Nascimento NG, Silva HB da, Fernandes CM, Lima MRD'I, Teixeira C. Local inflammatory events induced by Bothrops atrox snake venom and the release of distinct classes of inflammatory mediators [Internet]. Toxicon. 2012 ; 60( 1): 12-20.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.toxicon.2012.03.004
    • Vancouver

      Moreira V, Dos-Santos MC, Nascimento NG, Silva HB da, Fernandes CM, Lima MRD'I, Teixeira C. Local inflammatory events induced by Bothrops atrox snake venom and the release of distinct classes of inflammatory mediators [Internet]. Toxicon. 2012 ; 60( 1): 12-20.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.toxicon.2012.03.004
  • Source: Physiology and Behavior. Unidade: ICB

    Assunto: ANATOMIA

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      SANTOS, Lucélia Mendes dos et al. The role of the ventrolateral caudoputamen in predatory hunting. Physiology and Behavior, v. 105, n. 3, p. 893-898, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.physbeh.2011.10.021. Acesso em: 28 mar. 2024.
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      Santos, L. M. dos, Boschen, S. L., Bortolanza, M., Oliveira, W. F., Furigo, I. C., Mota-Ortiz, S. R., et al. (2012). The role of the ventrolateral caudoputamen in predatory hunting. Physiology and Behavior, 105( 3), 893-898. doi:10.1016/j.physbeh.2011.10.021
    • NLM

      Santos LM dos, Boschen SL, Bortolanza M, Oliveira WF, Furigo IC, Mota-Ortiz SR, Da Cunha C, Canteras NS. The role of the ventrolateral caudoputamen in predatory hunting [Internet]. Physiology and Behavior. 2012 ; 105( 3): 893-898.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.physbeh.2011.10.021
    • Vancouver

      Santos LM dos, Boschen SL, Bortolanza M, Oliveira WF, Furigo IC, Mota-Ortiz SR, Da Cunha C, Canteras NS. The role of the ventrolateral caudoputamen in predatory hunting [Internet]. Physiology and Behavior. 2012 ; 105( 3): 893-898.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.physbeh.2011.10.021
  • Source: Crop Protection. Unidade: ICB

    Assunto: MICROBIOLOGIA

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      ATAYDE, Danielle Diniz et al. Mycobiota and aflatoxins in a peanut variety grown in different regions in the state of São Paulo, Brazil. Crop Protection, v. 33, p. 7-12, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.cropro.2011.11.013. Acesso em: 28 mar. 2024.
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      Atayde, D. D., Reis, T. A. dos, Godoy, I. J., Zorzete, P., Reis, G. M., & Corrêa, B. (2012). Mycobiota and aflatoxins in a peanut variety grown in different regions in the state of São Paulo, Brazil. Crop Protection, 33, 7-12. doi:10.1016/j.cropro.2011.11.013
    • NLM

      Atayde DD, Reis TA dos, Godoy IJ, Zorzete P, Reis GM, Corrêa B. Mycobiota and aflatoxins in a peanut variety grown in different regions in the state of São Paulo, Brazil [Internet]. Crop Protection. 2012 ; 33 7-12.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.cropro.2011.11.013
    • Vancouver

      Atayde DD, Reis TA dos, Godoy IJ, Zorzete P, Reis GM, Corrêa B. Mycobiota and aflatoxins in a peanut variety grown in different regions in the state of São Paulo, Brazil [Internet]. Crop Protection. 2012 ; 33 7-12.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.cropro.2011.11.013
  • Source: Experimental Parasitology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FELIZARDO, Tânia Cristina et al. Lack of signaling by IL-4 or by IL-4/IL-13 has more attenuating effects on Leishmania amazonensis dorsal skin – than on footpad-infected mice. Experimental Parasitology, v. 130, n. 1, p. 48-57, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.exppara.2011.09.015. Acesso em: 28 mar. 2024.
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      Felizardo, T. C., Gaspar-Elsas, M. I. C., Lima, G. M. C. de A., & Abrahamsohn, I. de A. (2012). Lack of signaling by IL-4 or by IL-4/IL-13 has more attenuating effects on Leishmania amazonensis dorsal skin – than on footpad-infected mice. Experimental Parasitology, 130( 1), 48-57. doi:10.1016/j.exppara.2011.09.015
    • NLM

      Felizardo TC, Gaspar-Elsas MIC, Lima GMC de A, Abrahamsohn I de A. Lack of signaling by IL-4 or by IL-4/IL-13 has more attenuating effects on Leishmania amazonensis dorsal skin – than on footpad-infected mice [Internet]. Experimental Parasitology. 2012 ; 130( 1): 48-57.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.exppara.2011.09.015
    • Vancouver

      Felizardo TC, Gaspar-Elsas MIC, Lima GMC de A, Abrahamsohn I de A. Lack of signaling by IL-4 or by IL-4/IL-13 has more attenuating effects on Leishmania amazonensis dorsal skin – than on footpad-infected mice [Internet]. Experimental Parasitology. 2012 ; 130( 1): 48-57.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.exppara.2011.09.015
  • Source: FEMS Immunology and Medical Microbiology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      LOURES, Flávio Vieira et al. Paracoccidioides brasiliensis lipids modulate macrophage activity via Toll-dependent or independent mechanisms. FEMS Immunology and Medical Microbiology, v. 66, n. 1, p. 58-70, 2012Tradução . . Disponível em: https://doi.org/10.1111/j.1574-695X.2012.00986.x. Acesso em: 28 mar. 2024.
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      Loures, F. V., Stroeder, H., Almeida, I., & Calich, V. L. G. (2012). Paracoccidioides brasiliensis lipids modulate macrophage activity via Toll-dependent or independent mechanisms. FEMS Immunology and Medical Microbiology, 66( 1), 58-70. doi:10.1111/j.1574-695X.2012.00986.x
    • NLM

      Loures FV, Stroeder H, Almeida I, Calich VLG. Paracoccidioides brasiliensis lipids modulate macrophage activity via Toll-dependent or independent mechanisms [Internet]. FEMS Immunology and Medical Microbiology. 2012 ; 66( 1): 58-70.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1111/j.1574-695X.2012.00986.x
    • Vancouver

      Loures FV, Stroeder H, Almeida I, Calich VLG. Paracoccidioides brasiliensis lipids modulate macrophage activity via Toll-dependent or independent mechanisms [Internet]. FEMS Immunology and Medical Microbiology. 2012 ; 66( 1): 58-70.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1111/j.1574-695X.2012.00986.x
  • Source: International Journal for Parasitology. Unidade: ICB

    Assunto: PARASITOLOGIA

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      COSTA, Fabio T. M. et al. On the pathogenesis of Plasmodium vivax malaria: perspectives from the Brazilian field. International Journal for Parasitology, v. 42, n. 12, p. 1099-1105, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.ijpara.2012.08.007. Acesso em: 28 mar. 2024.
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      Costa, F. T. M., Lopes, S. C. P., Albrecht, L., Ataíde, R., Siqueira, A. M., Souza, R. M., et al. (2012). On the pathogenesis of Plasmodium vivax malaria: perspectives from the Brazilian field. International Journal for Parasitology, 42( 12), 1099-1105. doi:10.1016/j.ijpara.2012.08.007
    • NLM

      Costa FTM, Lopes SCP, Albrecht L, Ataíde R, Siqueira AM, Souza RM, Russell B, Renia L, Marinho CRF. On the pathogenesis of Plasmodium vivax malaria: perspectives from the Brazilian field [Internet]. International Journal for Parasitology. 2012 ; 42( 12): 1099-1105.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.ijpara.2012.08.007
    • Vancouver

      Costa FTM, Lopes SCP, Albrecht L, Ataíde R, Siqueira AM, Souza RM, Russell B, Renia L, Marinho CRF. On the pathogenesis of Plasmodium vivax malaria: perspectives from the Brazilian field [Internet]. International Journal for Parasitology. 2012 ; 42( 12): 1099-1105.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.ijpara.2012.08.007
  • Source: Physiology and Behavior. Unidade: ICB

    Assunto: FISIOLOGIA

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      SABINO-SILVA, Robinson et al. Baroreceptor-mediated activation of sympathetic nerve activity to salivary glands. Physiology and Behavior, v. 107, n. 3, p. 390-396, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.physbeh.2012.06.019. Acesso em: 28 mar. 2024.
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      Sabino-Silva, R., Ceroni, A., Koganezawa, T., Michelini, L. C., Machado, U. F., & Antunes, V. R. (2012). Baroreceptor-mediated activation of sympathetic nerve activity to salivary glands. Physiology and Behavior, 107( 3), 390-396. doi:10.1016/j.physbeh.2012.06.019
    • NLM

      Sabino-Silva R, Ceroni A, Koganezawa T, Michelini LC, Machado UF, Antunes VR. Baroreceptor-mediated activation of sympathetic nerve activity to salivary glands [Internet]. Physiology and Behavior. 2012 ; 107( 3): 390-396.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.physbeh.2012.06.019
    • Vancouver

      Sabino-Silva R, Ceroni A, Koganezawa T, Michelini LC, Machado UF, Antunes VR. Baroreceptor-mediated activation of sympathetic nerve activity to salivary glands [Internet]. Physiology and Behavior. 2012 ; 107( 3): 390-396.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.physbeh.2012.06.019
  • Source: Pharmacology, Biochemistry and Behavior. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      RIBEIRO, Andrea Frozino et al. A transcriptional study in mice with different ethanol-drinking profiles: possible involvement of the GABAB receptor. Pharmacology, Biochemistry and Behavior, v. 102, n. 2, p. 224-232, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.pbb.2012.04.010. Acesso em: 28 mar. 2024.
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      Ribeiro, A. F., Correia, D., Torres, A. A., Villas Boas, G. R., Rueda, A. V. L., Camarini, R., et al. (2012). A transcriptional study in mice with different ethanol-drinking profiles: possible involvement of the GABAB receptor. Pharmacology, Biochemistry and Behavior, 102( 2), 224-232. doi:10.1016/j.pbb.2012.04.010
    • NLM

      Ribeiro AF, Correia D, Torres AA, Villas Boas GR, Rueda AVL, Camarini R, Chiavegatto S, Boerngen-Lacerda R, Brunialti-Godard AL. A transcriptional study in mice with different ethanol-drinking profiles: possible involvement of the GABAB receptor [Internet]. Pharmacology, Biochemistry and Behavior. 2012 ; 102( 2): 224-232.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.pbb.2012.04.010
    • Vancouver

      Ribeiro AF, Correia D, Torres AA, Villas Boas GR, Rueda AVL, Camarini R, Chiavegatto S, Boerngen-Lacerda R, Brunialti-Godard AL. A transcriptional study in mice with different ethanol-drinking profiles: possible involvement of the GABAB receptor [Internet]. Pharmacology, Biochemistry and Behavior. 2012 ; 102( 2): 224-232.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1016/j.pbb.2012.04.010
  • Source: Archives of Oral Biology. Unidade: ICB

    Assunto: FARMACOLOGIA

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      HERRERA, Bruno S. et al. Platelet-rich plasma stimulates cytokine expression and alkaline phosphatase activity in osteoblast-derived osteosarcoma cells. Archives of Oral Biology, v. 57, n. 9, p. 1282-1289, 2012Tradução . . Disponível em: https://doi.org/10.1590/S0066-782X2012005000016. Acesso em: 28 mar. 2024.
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      Herrera, B. S., Coimbra, L. S., Bastos, A. S., Teixeira, S. A., Steffens, J. P., Muscará, M. N., & Spolidorio, L. C. (2012). Platelet-rich plasma stimulates cytokine expression and alkaline phosphatase activity in osteoblast-derived osteosarcoma cells. Archives of Oral Biology, 57( 9), 1282-1289. doi:10.1590/S0066-782X2012005000016
    • NLM

      Herrera BS, Coimbra LS, Bastos AS, Teixeira SA, Steffens JP, Muscará MN, Spolidorio LC. Platelet-rich plasma stimulates cytokine expression and alkaline phosphatase activity in osteoblast-derived osteosarcoma cells [Internet]. Archives of Oral Biology. 2012 ; 57( 9): 1282-1289.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1590/S0066-782X2012005000016
    • Vancouver

      Herrera BS, Coimbra LS, Bastos AS, Teixeira SA, Steffens JP, Muscará MN, Spolidorio LC. Platelet-rich plasma stimulates cytokine expression and alkaline phosphatase activity in osteoblast-derived osteosarcoma cells [Internet]. Archives of Oral Biology. 2012 ; 57( 9): 1282-1289.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1590/S0066-782X2012005000016
  • Source: AIDS Research and Human Retroviruses. Unidade: ICB

    Assunto: MICROBIOLOGIA

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      MELO, Fernando Lucas de e JAMAL, Leda Fátima e ZANOTTO, Paolo Marinho de Andrade. Characterization of Primary Isolates of HIV Type 1 CRF28_BF, CRF29_BF, and Unique BF Recombinants Circulating in São Paulo, Brazil. AIDS Research and Human Retroviruses, v. 28, n. 9, p. 1082-1088, 2012Tradução . . Disponível em: https://doi.org/10.1089/AID.2011.0123. Acesso em: 28 mar. 2024.
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      Melo, F. L. de, Jamal, L. F., & Zanotto, P. M. de A. (2012). Characterization of Primary Isolates of HIV Type 1 CRF28_BF, CRF29_BF, and Unique BF Recombinants Circulating in São Paulo, Brazil. AIDS Research and Human Retroviruses, 28( 9), 1082-1088. doi:10.1089/AID.2011.0123
    • NLM

      Melo FL de, Jamal LF, Zanotto PM de A. Characterization of Primary Isolates of HIV Type 1 CRF28_BF, CRF29_BF, and Unique BF Recombinants Circulating in São Paulo, Brazil [Internet]. AIDS Research and Human Retroviruses. 2012 ; 28( 9): 1082-1088.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1089/AID.2011.0123
    • Vancouver

      Melo FL de, Jamal LF, Zanotto PM de A. Characterization of Primary Isolates of HIV Type 1 CRF28_BF, CRF29_BF, and Unique BF Recombinants Circulating in São Paulo, Brazil [Internet]. AIDS Research and Human Retroviruses. 2012 ; 28( 9): 1082-1088.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1089/AID.2011.0123
  • Source: Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      LELLIS-SANTOS, Camilo et al. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion. Endocrinology, v. 153, n. 8, p. 3668-3678, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1135. Acesso em: 28 mar. 2024.
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      Lellis-Santos, C., Sakamoto, L. H., Bromati, C. R., Nogueira, T. C. de A., Leite, A. R., Yamanaka, T. S., et al. (2012). The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion. Endocrinology, 153( 8), 3668-3678. doi:10.1210/en.2012-1135
    • NLM

      Lellis-Santos C, Sakamoto LH, Bromati CR, Nogueira TC de A, Leite AR, Yamanaka TS, Kinote A, Anhê GF, Bordin S. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion [Internet]. Endocrinology. 2012 ; 153( 8): 3668-3678.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/en.2012-1135
    • Vancouver

      Lellis-Santos C, Sakamoto LH, Bromati CR, Nogueira TC de A, Leite AR, Yamanaka TS, Kinote A, Anhê GF, Bordin S. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion [Internet]. Endocrinology. 2012 ; 153( 8): 3668-3678.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/en.2012-1135
  • Source: Mediators of Inflammation. Unidade: ICB

    Assunto: IMUNOLOGIA

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      BACHI, André Luis Lacerda et al. Apoptotic cells contribute to melanoma progression and this effect is partially mediated by the platelet-activating factor receptor. Mediators of Inflammation, v. 2012, p. 1-6, 2012Tradução . . Disponível em: https://doi.org/10.1155/2012/610371. Acesso em: 28 mar. 2024.
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      Bachi, A. L. L., Santos, L. C. dos, Nonogaki, S., Jancar, S., & Jasiulionis, M. G. (2012). Apoptotic cells contribute to melanoma progression and this effect is partially mediated by the platelet-activating factor receptor. Mediators of Inflammation, 2012, 1-6. doi:10.1155/2012/610371
    • NLM

      Bachi ALL, Santos LC dos, Nonogaki S, Jancar S, Jasiulionis MG. Apoptotic cells contribute to melanoma progression and this effect is partially mediated by the platelet-activating factor receptor [Internet]. Mediators of Inflammation. 2012 ; 2012 1-6.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1155/2012/610371
    • Vancouver

      Bachi ALL, Santos LC dos, Nonogaki S, Jancar S, Jasiulionis MG. Apoptotic cells contribute to melanoma progression and this effect is partially mediated by the platelet-activating factor receptor [Internet]. Mediators of Inflammation. 2012 ; 2012 1-6.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1155/2012/610371

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