Mapping of polyomavirus middle T domain that is responsible for AP-1 activation (1998)
- Autores:
- Autor USP: SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- DOI: 10.1038/sj.onc.1201841
- Assunto: BIOQUÍMICA
- Idioma: Inglês
- Imprenta:
- Local: Basingstoke
- Data de publicação: 1998
- Fonte:
- Título do periódico: Oncogene
- Volume/Número/Paginação/Ano: v. 16, n. 23, p. 2975-2982, 1998
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
OLIVEIRA, M. L. S. et al. Mapping of polyomavirus middle T domain that is responsible for AP-1 activation. Oncogene, v. 16, n. 23, p. 2975-2982, 1998Tradução . . Disponível em: https://doi.org/10.1038/sj.onc.1201841. Acesso em: 19 abr. 2024. -
APA
Oliveira, M. L. S., Roberts, T. M., Druker, B. J., & Armelin, M. C. S. (1998). Mapping of polyomavirus middle T domain that is responsible for AP-1 activation. Oncogene, 16( 23), 2975-2982. doi:10.1038/sj.onc.1201841 -
NLM
Oliveira MLS, Roberts TM, Druker BJ, Armelin MCS. Mapping of polyomavirus middle T domain that is responsible for AP-1 activation [Internet]. Oncogene. 1998 ; 16( 23): 2975-2982.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1038/sj.onc.1201841 -
Vancouver
Oliveira MLS, Roberts TM, Druker BJ, Armelin MCS. Mapping of polyomavirus middle T domain that is responsible for AP-1 activation [Internet]. Oncogene. 1998 ; 16( 23): 2975-2982.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1038/sj.onc.1201841 - Role of polyoma t antigens in balb-c/3t3 cells growth control deregulation
- Further characterization of balb-3t3 transfectants bearing polyoma t antigen genes
- Controle e subversao da proliferacao
- Glucocorticoid-regulated gene in transformed to normal phenotypic reversion
- T antigens role in polyomavirus transformation: c-myc but not c-fos or c-jun expression is a target for middle t
- Cdna cloning and expression of growth control genes
- Fibroblast growth factor and camp induce the expression of fos and jun proto-oncogenes in pc12 cells
- Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma
- Cloning of differentially expressed genes involved glucocorticoid-induced phenotypic reversion of c6 glioma cells
- Hydrazine derivative - induced proliferation superimposed on citotoxity in cultured moused fibroblasts
Informações sobre o DOI: 10.1038/sj.onc.1201841 (Fonte: oaDOI API)
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