Mycophenolate mofetil reduces renal injury and angiotensin II expression in the chronic NO synthase inhibition model (1998)
- Autores:
- Autores USP: NUCCI, GILBERTO DE - ICB ; ZATZ, ROBERTO - FM
- Unidades: ICB; FM
- Assunto: FARMACOLOGIA
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Journal of American Society of Nephrology
- Volume/Número/Paginação/Ano: v. 9, p. 609, 1998
- Nome do evento: Annual Meeting of the American Society of Nephrology
-
ABNT
DE NUCCI, Gilberto et al. Mycophenolate mofetil reduces renal injury and angiotensin II expression in the chronic NO synthase inhibition model. Journal of American Society of Nephrology. Baltimore: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 23 abr. 2024. , 1998 -
APA
De Nucci, G., Zatz, R., Oliveira, I. B. O., Fujihara, C. K., Sena, C. R., Malheiros, D. M. A. C., et al. (1998). Mycophenolate mofetil reduces renal injury and angiotensin II expression in the chronic NO synthase inhibition model. Journal of American Society of Nephrology. Baltimore: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
De Nucci G, Zatz R, Oliveira IBO, Fujihara CK, Sena CR, Malheiros DMAC, Antunes GR, Noronha IL. Mycophenolate mofetil reduces renal injury and angiotensin II expression in the chronic NO synthase inhibition model. Journal of American Society of Nephrology. 1998 ; 9 609.[citado 2024 abr. 23 ] -
Vancouver
De Nucci G, Zatz R, Oliveira IBO, Fujihara CK, Sena CR, Malheiros DMAC, Antunes GR, Noronha IL. Mycophenolate mofetil reduces renal injury and angiotensin II expression in the chronic NO synthase inhibition model. Journal of American Society of Nephrology. 1998 ; 9 609.[citado 2024 abr. 23 ] - Hypertension induce by chronic no synthesis blockade is not related to the induction of tissular ace activity but is prevented by enalapril
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- Effects of diltiazem on hypertension and cardiac hypertrophy induced by chronic inhibition of nitric oxide (no) synthesis
- In vivo inhibition of nitric oxide synthesis does not depend on renin-angiotensin system activation
- Nitric oxide synthase isoforms and renal macrophage infiltration in early and late experimental diabetes
- Renal effects of acute and chronic nitric oxide inhibition in experimental diabetes
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