Chemokine profile during allogeneic heart transplant rejection (1999)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; ROSSI, MARCOS ANTONIO - FMRP ; FERRAZ, AGENOR SPALLINI - FMRP
- Unidade: FMRP
- DOI: 10.1016/s0041-1345(99)00633-8
- Assunto: TRANSPLANTES
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Transplantation Proceedings
- ISSN: 0041-1345
- Volume/Número/Paginação/Ano: v. 31, p. 2978-2981, 1999
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
SABER, L. T. S. et al. Chemokine profile during allogeneic heart transplant rejection. Transplantation Proceedings, v. 31, p. 2978-2981, 1999Tradução . . Disponível em: https://doi.org/10.1016/s0041-1345(99)00633-8. Acesso em: 24 abr. 2024. -
APA
Saber, L. T. S., Aliberti, J. C. S., Silva, J. S. da, Rossi, M. A., & Ferraz, A. S. (1999). Chemokine profile during allogeneic heart transplant rejection. Transplantation Proceedings, 31, 2978-2981. doi:10.1016/s0041-1345(99)00633-8 -
NLM
Saber LTS, Aliberti JCS, Silva JS da, Rossi MA, Ferraz AS. Chemokine profile during allogeneic heart transplant rejection [Internet]. Transplantation Proceedings. 1999 ; 31 2978-2981.[citado 2024 abr. 24 ] Available from: https://doi.org/10.1016/s0041-1345(99)00633-8 -
Vancouver
Saber LTS, Aliberti JCS, Silva JS da, Rossi MA, Ferraz AS. Chemokine profile during allogeneic heart transplant rejection [Internet]. Transplantation Proceedings. 1999 ; 31 2978-2981.[citado 2024 abr. 24 ] Available from: https://doi.org/10.1016/s0041-1345(99)00633-8 - Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy
- Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy
- nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes
- Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi
- Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi
- Role of interleukin (IL)-18 in experimental paracoccidioidomycosis
- B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection
- Intensification of acute t. Cruzi myocarditis in balb / c nice pretreated
- Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice
- The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice
Informações sobre o DOI: 10.1016/s0041-1345(99)00633-8 (Fonte: oaDOI API)
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