Lack of experimental autoimmune encephalomyelitis (EAE) in mice infected with Trypanosoma cruzi (2000)
- Autores:
- Autores USP: RIZZO, LUIZ VICENTE - ICB ; ABRAHAMSOHN, ISES DE ALMEIDA - ICB
- Unidade: ICB
- Assunto: IMMUNOLOGIA
- Idioma: Inglês
- Imprenta:
- Local: Florianópolis
- Data de publicação: 2000
- Fonte:
- Título do periódico: Abstract
- Nome do evento: Meeting of the Brazilian Society of Immunology
-
ABNT
TADOKORO, C E et al. Lack of experimental autoimmune encephalomyelitis (EAE) in mice infected with Trypanosoma cruzi. 2000, Anais.. Florianópolis: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2000. . Acesso em: 04 maio 2024. -
APA
Tadokoro, C. E., Mosca, T., Rizzo, L. V., & Abrahamsohn, I. de A. (2000). Lack of experimental autoimmune encephalomyelitis (EAE) in mice infected with Trypanosoma cruzi. In Abstract. Florianópolis: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Tadokoro CE, Mosca T, Rizzo LV, Abrahamsohn I de A. Lack of experimental autoimmune encephalomyelitis (EAE) in mice infected with Trypanosoma cruzi. Abstract. 2000 ;[citado 2024 maio 04 ] -
Vancouver
Tadokoro CE, Mosca T, Rizzo LV, Abrahamsohn I de A. Lack of experimental autoimmune encephalomyelitis (EAE) in mice infected with Trypanosoma cruzi. Abstract. 2000 ;[citado 2024 maio 04 ] - Trypanosoma cruzi infection inhibits autoimmune encephalomyelitis
- The effect of t. cruzi infection over eae development
- The development of experimental autoimmune encephalomyelitis could be completed abrogated by immunossuppressive effect of Trypanosoma cruzi infection
- Experimental autoimmune encephalomyelitis can be prevented and cured by infection with Trypanosoma cruzi
- High molecular weight components from Ascaris suum exert suppressive effect on dendritic cells
- Combined immunodeficiency in a chilhood with Cd3 deficiency: case report
- Endogenously produced IL-10 and prostaglandins regulate peripheral blood mononuclear cells proliferation to Trypanosoma cruzi antigens
- Nitric oxide produced by macrophages and regulation of resistance to "Trypanosoma cruzi" at the initial phase of infection
- Egulation of Trypanosoma cruzi infection by type interferons
- Il-12 regulation of ifn-gama synthesis in the acute phase of trypanosoma cruzi infection
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