Nitric oxide and membrane hyperpolarization are involved in the restoring effect of insulin on angiotensin-(1-7) and bradykinin interaction in type 2 diabetes (2004)
- Authors:
- USP affiliated authors: SCAVONE, CRISTOFORO - ICB ; NIGRO, DOROTHY - ICB ; CARVALHO, MARIA HELENA CATELLI DE - ICB ; PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - ICB ; FORTES, ZULEICA BRUNO - ICB
- Unidade: ICB
- Assunto: FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Rio de Janeiro
- Date published: 2004
- Source:
- Título do periódico: Resumos
- Conference titles: International Symposium Nitric Oxide Cytokines and Inflammation
-
ABNT
AKAMINE, Eliana Hiromi et al. Nitric oxide and membrane hyperpolarization are involved in the restoring effect of insulin on angiotensin-(1-7) and bradykinin interaction in type 2 diabetes. 2004, Anais.. Rio de Janeiro: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2004. . Acesso em: 23 abr. 2024. -
APA
Akamine, E. H., Kawamoto, E. M., Scavone, C., Nigro, D., Carvalho, M. H. C. de, Passaglia, R. de C. A. T., & Fortes, Z. B. (2004). Nitric oxide and membrane hyperpolarization are involved in the restoring effect of insulin on angiotensin-(1-7) and bradykinin interaction in type 2 diabetes. In Resumos. Rio de Janeiro: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Akamine EH, Kawamoto EM, Scavone C, Nigro D, Carvalho MHC de, Passaglia R de CAT, Fortes ZB. Nitric oxide and membrane hyperpolarization are involved in the restoring effect of insulin on angiotensin-(1-7) and bradykinin interaction in type 2 diabetes. Resumos. 2004 ;[citado 2024 abr. 23 ] -
Vancouver
Akamine EH, Kawamoto EM, Scavone C, Nigro D, Carvalho MHC de, Passaglia R de CAT, Fortes ZB. Nitric oxide and membrane hyperpolarization are involved in the restoring effect of insulin on angiotensin-(1-7) and bradykinin interaction in type 2 diabetes. Resumos. 2004 ;[citado 2024 abr. 23 ] - In vivo evidence of oxidative stress in intrauterine programming of hypertensive in wistar rats
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- Angiotensin II induces kinin B1 receptor in aorta of rats
- Insulin corrects the altered vascular reactivity without restoring the reduced nitric oxide synthase activity in diabetic female rats
- Enhanced oxidative stress as a potential mechanism underlying the programming of hypertension in utero
- Metformin treatment restores the altered microvascular reactivity in neonatal streptozotocin-induced diabetic rats increasing NOS activity, but not NOS expression
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