Expression of checkpoint kinase 2 in breast carcinomas: correlation with key regulators of tumor cell proliferation, angiogenesis, and survival (2006)
- Authors:
- USP affiliated authors: SILVA, ALFREDO RIBEIRO DA - FMRP ; ZUCOLOTO, SÉRGIO - FMRP
- Unidade: FMRP
- Subjects: CARCINOMA; IMUNOHISTOQUÍMICA; NEOPLASIAS MAMÁRIAS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Histology and Histopathology Celular and Molecular Biology
- ISSN: 0213-3911
- Volume/Número/Paginação/Ano: v. 21, n. 4, p. 373-382, 2006
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ABNT
RIBEIRO-SILVA, Alfredo et al. Expression of checkpoint kinase 2 in breast carcinomas: correlation with key regulators of tumor cell proliferation, angiogenesis, and survival. Histology and Histopathology Celular and Molecular Biology, v. 21, n. 4, p. 373-382, 2006Tradução . . Acesso em: 20 abr. 2024. -
APA
Ribeiro-Silva, A., Moutinho, M. A. K., Moura, H. B. de, Vale, F. R. do, & Zucoloto, S. (2006). Expression of checkpoint kinase 2 in breast carcinomas: correlation with key regulators of tumor cell proliferation, angiogenesis, and survival. Histology and Histopathology Celular and Molecular Biology, 21( 4), 373-382. -
NLM
Ribeiro-Silva A, Moutinho MAK, Moura HB de, Vale FR do, Zucoloto S. Expression of checkpoint kinase 2 in breast carcinomas: correlation with key regulators of tumor cell proliferation, angiogenesis, and survival. Histology and Histopathology Celular and Molecular Biology. 2006 ; 21( 4): 373-382.[citado 2024 abr. 20 ] -
Vancouver
Ribeiro-Silva A, Moutinho MAK, Moura HB de, Vale FR do, Zucoloto S. Expression of checkpoint kinase 2 in breast carcinomas: correlation with key regulators of tumor cell proliferation, angiogenesis, and survival. Histology and Histopathology Celular and Molecular Biology. 2006 ; 21( 4): 373-382.[citado 2024 abr. 20 ] - Prognostic impact of p53 and p63 immunoexpression in oral squamous cell carcinoma
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- Checkpoint kinase 2 in breast cancer: a clinicopathologic and immuno-histochemical study
- The differencial regulation of human telomerase reverse transcriptase and vascular endothelial growth factor may contribute to the clinically more aggressive behavior of p63-positive breast carcinomas
- The differential regulation of p16 and human telomerase reverse transcriptase may contribute to the clinically more aggressive behavior of breast carcinomas with the basal-like phenotype
- Expression of apoptosis-related protein Bcl-2 correlates with breast carcinomas of luminal of basal-like subtypes
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