Role of PPAR-gamma in the modulation of CD36 and FcgammaRII induced by LDL with low and high degrees of oxidation during the differentiation of the monocytic THP-1 cell line (2008)
- Authors:
- USP affiliated authors: NEGRO, SONIA JANCAR - ICB ; GIDLUND, MAGNUS AKE - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Cellular Physiology and Biochemistry
- ISSN: 1015-8987
- Volume/Número/Paginação/Ano: v. 22, n. 1/4, p. 279-286, 2008
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ABNT
RIOS, Francisco José de Oliveira et al. Role of PPAR-gamma in the modulation of CD36 and FcgammaRII induced by LDL with low and high degrees of oxidation during the differentiation of the monocytic THP-1 cell line. Cellular Physiology and Biochemistry, v. 22, n. 1/4, p. 279-286, 2008Tradução . . Acesso em: 26 abr. 2024. -
APA
Rios, F. J. de O., Jancar, S., Melo, I. B., Ketelhuth, D. F. J., & Gidlund, M. A. (2008). Role of PPAR-gamma in the modulation of CD36 and FcgammaRII induced by LDL with low and high degrees of oxidation during the differentiation of the monocytic THP-1 cell line. Cellular Physiology and Biochemistry, 22( 1/4), 279-286. -
NLM
Rios FJ de O, Jancar S, Melo IB, Ketelhuth DFJ, Gidlund MA. Role of PPAR-gamma in the modulation of CD36 and FcgammaRII induced by LDL with low and high degrees of oxidation during the differentiation of the monocytic THP-1 cell line. Cellular Physiology and Biochemistry. 2008 ; 22( 1/4): 279-286.[citado 2024 abr. 26 ] -
Vancouver
Rios FJ de O, Jancar S, Melo IB, Ketelhuth DFJ, Gidlund MA. Role of PPAR-gamma in the modulation of CD36 and FcgammaRII induced by LDL with low and high degrees of oxidation during the differentiation of the monocytic THP-1 cell line. Cellular Physiology and Biochemistry. 2008 ; 22( 1/4): 279-286.[citado 2024 abr. 26 ] - Oxidized low density lipoprotein inhibits LPS/IFN-induced nitric oxide production in RAW264.7 cells - effects of a PAF-antagonist
- Vascular permeability is increased by oxidized low density lipoprotein (oxLDL), size-inclusion subfractions and slelcted immunogenic apolipoprotein B (apoB) derived synthetic peptides
- Myeloperoxidase, hydrogen peroxide and tumor necrosis factor 'alfa' production as methods to access platelet aggregating factor - like activity of oxidazed low density lipoprotein (OXLDL)
- Oxidized LDL induces alternative macrophage phenotype through activation of CD36 and PAFR
- OxLDL inibe a produção de óxido nítrico induzida por LPS e IFN-gamma em células RAW264.7 via receptor de PAF
- Oxidized low density lipoprotein (LDL) can increase the expression of scavenger receptor (CD36) and FCgRII by interaction with platelet-activating factor (PAF) receptor
- Oxidized low density lipoprotein (oxLDL) and purified oxLDL products inhibits nitric oxide releasem iNOS and COX-2 expression in macrophages
- Oxidized low density lipoprotein inhibits LPS/IFN-induced nitric oxideproduction in RAW264.7 cells - effect of a PAF-antagonist
- Degree of modification of low density lipoprotein (LDL) displays different properties in size and eletric charge and induction of CD11B and CD36 in THP-1 cells
- Vascular permeability is increased by oxidized low density lopoprotein (oxLDL) and selected apolipoprotein B derived synthetic polypeptides
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