Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP) (2011)
- Autores:
- Autores USP: VERCESI, JULIANA APARECIDA - FCFRP ; GRANDO, MARCELLA DARUGE - FCFRP ; BENDHACK, LUSIANE MARIA - FCFRP
- Unidade: FCFRP
- DOI: 10.1016/j.vph.2010.12.002
- Assuntos: ÓXIDO NÍTRICO; VASODILATAÇÃO; SEPSE
- Idioma: Inglês
- Imprenta:
- Local: Philadelphia
- Data de publicação: 2011
- Fonte:
- Título do periódico: Vascualr Pharmacology
- ISSN: 1537-1891
- Volume/Número/Paginação/Ano: v. 54, n. 1-2, p. 44-51, 2011
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: publisher-specific-oa
-
ABNT
ARAÚJO, Alice Valença et al. Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP). Vascualr Pharmacology, v. 54, n. 1-2, p. 44-51, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2010.12.002. Acesso em: 19 abr. 2024. -
APA
Araújo, A. V., Ferezin, C. Z., Rodrigues, G. J., Lunardi, C. N., Vercesi, J. A., Grando, M. D., et al. (2011). Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP). Vascualr Pharmacology, 54( 1-2), 44-51. doi:10.1016/j.vph.2010.12.002 -
NLM
Araújo AV, Ferezin CZ, Rodrigues GJ, Lunardi CN, Vercesi JA, Grando MD, Bonaventura D, Bendhack LM. Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP) [Internet]. Vascualr Pharmacology. 2011 ; 54( 1-2): 44-51.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.vph.2010.12.002 -
Vancouver
Araújo AV, Ferezin CZ, Rodrigues GJ, Lunardi CN, Vercesi JA, Grando MD, Bonaventura D, Bendhack LM. Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP) [Internet]. Vascualr Pharmacology. 2011 ; 54( 1-2): 44-51.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.vph.2010.12.002 - Espécies reativas de oxigênio podem modular negativamente o relaxamento induzido pelo nitroprussiato de sódio em aortas de ratos hipertensos
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- Acetylcholine induces relaxation dependent on nitric oxide synthase and cyclooxigenase during sepsis
- Endothelial cyclooxygenase differently modulates the contractile response to phenylephrine but not to thromboxane analog in aorta from normotensive and hypertensive rats
- Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats
- Increased resting tension abolishes the endothelial anti-contractile effect induced by phenylephrine in renal hypertensive (2K-1C) rat aorta
- Hidrogen peroxide decreases the contractile response induced by phenylephrine in renal hypertensive rat aorta
- Augmented nitric oxide production and up-regulation of endothelial nitric oxide synthase during cecal ligation and perforation
- A tolerância induzida pela nitroglicerina pode ter início no endotélio após 5 minutos de exposição in vitro com aumento de EROs
- Relaxation induced by the nitric oxide donor and cyclooxygenase inhibitor NCX2121 in renal hypertensive rat aortas
Informações sobre o DOI: 10.1016/j.vph.2010.12.002 (Fonte: oaDOI API)
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