The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction (2013)
- Autores:
- Autores USP: LEOPOLDINO, ANDRÉIA MACHADO - FCFRP ; CURTI, CARLOS - FCFRP
- Unidade: FCFRP
- Assuntos: NEOPLASIAS DE CABEÇA E PESCOÇO; CARCINOMA DE CÉLULAS ESCAMOSAS; ESTRESSE OXIDATIVO
- Idioma: Inglês
- Imprenta:
- Editora: SBBq
- Local: Foz do Iguaçu
- Data de publicação: 2013
- Fonte:
- Título do periódico: Abstracts
- Nome do evento: Annual Meeting of SBBq
-
ABNT
PESTANA, C. R. e LEOPOLDINO, Andréia Machado e CURTI, Carlos. The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction. 2013, Anais.. Foz do Iguaçu: SBBq, 2013. . Acesso em: 19 abr. 2024. -
APA
Pestana, C. R., Leopoldino, A. M., & Curti, C. (2013). The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction. In Abstracts. Foz do Iguaçu: SBBq. -
NLM
Pestana CR, Leopoldino AM, Curti C. The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction. Abstracts. 2013 ;[citado 2024 abr. 19 ] -
Vancouver
Pestana CR, Leopoldino AM, Curti C. The accumulation of SET protein in head and neck squamous cell carcinoma (HNSCCs) increases basal autophagy and prevents mitochondrial dysfunction. Abstracts. 2013 ;[citado 2024 abr. 19 ] - SET/I2PP2A is an essential protein for cell migration in head and neck squamous cell carcinoma
- SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant
- Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation
- SET protects mitochondrial function and induces autophagy in response to redox unbalance
- SET protein regulates expression of glutathione S-transferase pi 1 (GSTP-1) in oral carcinoma
- Different responses of two HNSCC cell lineages to oxidative stress
- Knockdown of SET protein sensitizes HNSCC-HN12 cells to UCN-01
- ATP production in HnRNP K-overexpressing HEK293 cells
- Maternal protein restriction increases respiratory and sympathetic activities and sensitizes peripheral chemoreflex in male rat offspring
- SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis
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