IL-10 and TNF-α differentially modulate the outcome of alveolar bone repair upon homeostatic and infectious conditions (2013)
- Autores:
- Autores USP: CAMPANELLI, ANA PAULA - FOB ; GARLET, GUSTAVO POMPERMAIER - FOB
- Unidade: FOB
- Assuntos: REPARO ÓSSEO; REPARAÇÃO ALVEOLAR; CITOCINAS
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Brazilian Oral Research
- ISSN: 1806-8324
- Volume/Número/Paginação/Ano: v. 27, p. 19, res. HA009, Sept. 2013. Supplement 1
- Nome do evento: Reunião Anual da Sociedade Brasileira de Pesquisa Odontológica - SBPqO
-
ABNT
VIEIRA, A. E. et al. IL-10 and TNF-α differentially modulate the outcome of alveolar bone repair upon homeostatic and infectious conditions. Brazilian Oral Research. São Paulo: Faculdade de Odontologia de Bauru, Universidade de São Paulo. . Acesso em: 19 abr. 2024. , 2013 -
APA
Vieira, A. E., Repeke, C. E. P., Biguetti, C. C., Francisconi, C. F., Silveira, E. M. V., Campanelli, A. P., et al. (2013). IL-10 and TNF-α differentially modulate the outcome of alveolar bone repair upon homeostatic and infectious conditions. Brazilian Oral Research. São Paulo: Faculdade de Odontologia de Bauru, Universidade de São Paulo. -
NLM
Vieira AE, Repeke CEP, Biguetti CC, Francisconi CF, Silveira EMV, Campanelli AP, Trombone APF, Garlet GP. IL-10 and TNF-α differentially modulate the outcome of alveolar bone repair upon homeostatic and infectious conditions. Brazilian Oral Research. 2013 ; 27 19.[citado 2024 abr. 19 ] -
Vancouver
Vieira AE, Repeke CEP, Biguetti CC, Francisconi CF, Silveira EMV, Campanelli AP, Trombone APF, Garlet GP. IL-10 and TNF-α differentially modulate the outcome of alveolar bone repair upon homeostatic and infectious conditions. Brazilian Oral Research. 2013 ; 27 19.[citado 2024 abr. 19 ] - CCR5+ cells modulate RANKL+ cell migration, inflammatory reaction development and experimental periodontitis outcome in mice
- The potential role of suppressors of cytokine signaling in the attenuation of inflammatory reaction and alveolar bone loss associated with apical periodontitis
- Involvement of CCR5 and CXCR3 in RANKL+ cell migration and bone resorption throughout experimental periodontitis in mouse
- Associação diferencial de fatores genéticos, microbianos e inflamatórios com os níveis de MMP-1 no periodonto saudável e com periodontite
- IL-4/CCL22/CCR4 axis controls regulatory T-cell migration that suppresses inflammatory bone loss in murine experimental periodontitis
- Distinct roles for genetic (MMP1-1607 and -519 SNPs) factors and periodontopathogens in the modulation of MMP-1 levels in healthy and diseased periodontal tissues
- The broad effects of the functional IL-10 promoter -592 polymorphism: modulation of IL-10, TIMP-3 and OPG expression and its association with periodontal disease outcome
- Evidence supporting a protective role for Th9 and Th22 cytokines in human and experimental periapical lesions
- Characterization of tumor-infiltrating regulatory T-cell subpopulation in oral squamous cell carcinoma
- Strong and persistent microbial and inflammatory stimuli overcome the genetic predisposition to higher matrix metalloproteinase-1 (MMP-1) expression: a mechanistic explanation for the lack of association of MMP1-1607 single-nucleotide polymorphism genotypes with MMP-1 expression in chronic periodontitis lesions
Como citar
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
