Mesenchymal stem cells as active prohealing and immunosuppressive agents in periapical environment: evidence from human and experimental periapical lesions (2014)
- Autores:
- Autores USP: CAMPANELLI, ANA PAULA - FOB ; GARLET, GUSTAVO POMPERMAIER - FOB
- Unidade: FOB
- DOI: 10.1016/j.joen.2014.02.012
- Assuntos: PERIODONTITE PERIAPICAL; EXPRESSÃO GÊNICA; CÉLULAS-TRONCO
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Journal of Endodontics
- ISSN: 0099-2399
- Volume/Número/Paginação/Ano: v. 40, n. 10, p. 1560-1565, Oct. 2014
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
ARAUJO-PIRES, Ana Claudia et al. Mesenchymal stem cells as active prohealing and immunosuppressive agents in periapical environment: evidence from human and experimental periapical lesions. Journal of Endodontics, v. 40, n. 10, p. 1560-1565, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.joen.2014.02.012. Acesso em: 25 abr. 2024. -
APA
Araujo-Pires, A. C., Biguetti, C. C., Repeke, C. E., Rodini, C. de O., Campanelli, A. P., Trombone, A. P. F., et al. (2014). Mesenchymal stem cells as active prohealing and immunosuppressive agents in periapical environment: evidence from human and experimental periapical lesions. Journal of Endodontics, 40( 10), 1560-1565. doi:10.1016/j.joen.2014.02.012 -
NLM
Araujo-Pires AC, Biguetti CC, Repeke CE, Rodini C de O, Campanelli AP, Trombone APF, Letra A, Silva RM, Garlet GP. Mesenchymal stem cells as active prohealing and immunosuppressive agents in periapical environment: evidence from human and experimental periapical lesions [Internet]. Journal of Endodontics. 2014 ; 40( 10): 1560-1565.[citado 2024 abr. 25 ] Available from: https://doi.org/10.1016/j.joen.2014.02.012 -
Vancouver
Araujo-Pires AC, Biguetti CC, Repeke CE, Rodini C de O, Campanelli AP, Trombone APF, Letra A, Silva RM, Garlet GP. Mesenchymal stem cells as active prohealing and immunosuppressive agents in periapical environment: evidence from human and experimental periapical lesions [Internet]. Journal of Endodontics. 2014 ; 40( 10): 1560-1565.[citado 2024 abr. 25 ] Available from: https://doi.org/10.1016/j.joen.2014.02.012 - CCR5+ cells modulate RANKL+ cell migration, inflammatory reaction development and experimental periodontitis outcome in mice
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- Involvement of CCR5 and CXCR3 in RANKL+ cell migration and bone resorption throughout experimental periodontitis in mouse
- Associação diferencial de fatores genéticos, microbianos e inflamatórios com os níveis de MMP-1 no periodonto saudável e com periodontite
- IL-4/CCL22/CCR4 axis controls regulatory T-cell migration that suppresses inflammatory bone loss in murine experimental periodontitis
- Distinct roles for genetic (MMP1-1607 and -519 SNPs) factors and periodontopathogens in the modulation of MMP-1 levels in healthy and diseased periodontal tissues
- The broad effects of the functional IL-10 promoter -592 polymorphism: modulation of IL-10, TIMP-3 and OPG expression and its association with periodontal disease outcome
- Evidence supporting a protective role for Th9 and Th22 cytokines in human and experimental periapical lesions
- IL-10 and TNF-α differentially modulate the outcome of alveolar bone repair upon homeostatic and infectious conditions
- Characterization of tumor-infiltrating regulatory T-cell subpopulation in oral squamous cell carcinoma
Informações sobre o DOI: 10.1016/j.joen.2014.02.012 (Fonte: oaDOI API)
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