CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development (2018)
- Autores:
- Autores USP: CRUZ, MARIO COSTA - ICB ; HIYANE, MEIRE IOSHIE - ICB ; SILVA, JOÃO SANTANA DA - FMRP ; CUCCOVIA, IOLANDA MIDEA - IQ ; CÂMARA, NIELS OLSEN SARAIVA - ICB
- Unidades: ICB; FMRP; IQ
- DOI: 10.1007/s10787-017-0317-4
- Assuntos: MONÓCITOS; QUIMIOCINAS; INFLAMAÇÃO; TÉCNICAS DE DIAGNÓSTICO UROLÓGICO
- Agências de fomento:
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Inflammopharmacology
- ISSN: 0925-4692
- Volume/Número/Paginação/Ano: v. 26, p. 403-411, 2018
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
BRAGA, Tárcio Teodoro et al. CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development. Inflammopharmacology, v. 26, p. 403-411, 2018Tradução . . Disponível em: https://doi.org/10.1007/s10787-017-0317-4. Acesso em: 28 mar. 2024. -
APA
Braga, T. T., Costa, M. C., Silva, R. C., Cruz, M. C., Hiyane, M. I., Silva, J. S. da, et al. (2018). CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development. Inflammopharmacology, 26, 403-411. doi:10.1007/s10787-017-0317-4 -
NLM
Braga TT, Costa MC, Silva RC, Cruz MC, Hiyane MI, Silva JS da, Perez KR, Cuccovia IM, Câmara NOS. CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development [Internet]. Inflammopharmacology. 2018 ; 26 403-411.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1007/s10787-017-0317-4 -
Vancouver
Braga TT, Costa MC, Silva RC, Cruz MC, Hiyane MI, Silva JS da, Perez KR, Cuccovia IM, Câmara NOS. CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development [Internet]. Inflammopharmacology. 2018 ; 26 403-411.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1007/s10787-017-0317-4 - Macrophage trafficking as key mediator of adenine-induced kidney injury
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Informações sobre o DOI: 10.1007/s10787-017-0317-4 (Fonte: oaDOI API)
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