Mutated ej-c-ha-ras-l human oncogene causes malignant transformation without abolishing growth requirement for fgf and egf (1987)
- Authors:
- USP affiliated authors: ARMELIN, HUGO AGUIRRE - IQ ; SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- Subjects: ONCOGENES; BIOQUÍMICA
- Language: Português
- Source:
- Título do periódico: Arquivos de Biologia e Tecnologia
- Volume/Número/Paginação/Ano: v.30, n.1 , p.67, 1987
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica
-
ABNT
KOVARY, K e ARMELIN, M C S e ARMELIN, Hugo Aguirre. Mutated ej-c-ha-ras-l human oncogene causes malignant transformation without abolishing growth requirement for fgf and egf. Arquivos de Biologia e Tecnologia. [S.l.]: Instituto de Química, Universidade de São Paulo. . Acesso em: 19 abr. 2024. , 1987 -
APA
Kovary, K., Armelin, M. C. S., & Armelin, H. A. (1987). Mutated ej-c-ha-ras-l human oncogene causes malignant transformation without abolishing growth requirement for fgf and egf. Arquivos de Biologia e Tecnologia. Instituto de Química, Universidade de São Paulo. -
NLM
Kovary K, Armelin MCS, Armelin HA. Mutated ej-c-ha-ras-l human oncogene causes malignant transformation without abolishing growth requirement for fgf and egf. Arquivos de Biologia e Tecnologia. 1987 ;30( 1 ): 67.[citado 2024 abr. 19 ] -
Vancouver
Kovary K, Armelin MCS, Armelin HA. Mutated ej-c-ha-ras-l human oncogene causes malignant transformation without abolishing growth requirement for fgf and egf. Arquivos de Biologia e Tecnologia. 1987 ;30( 1 ): 67.[citado 2024 abr. 19 ] - Generation of myc / fos transfectant bolb - 3t3 cell line
- Ha-ras-1 oncogene dosage differentially affects balb / 3t3 cells , growth factor requirement and tumorigenicity
- Fudr selection of revertants from ej-ras-transformed 3t3 cells
- Mode of v-fos oncogene integration in 3t3 cells genome
- Isolation and characterization of balb/c double transfectant cell lines contitutively expressing c-myc and v-fos oncogenes
- Mechanisms of action of growth factors and oncogene expression
- Glucocorticoid dexamethasone reversibly complements 'E IND.J'-ras oncogene to transform mouse embryo balb - 3t3 cells
- Role played by oncogene products in peptide growth factor's action
- Interactions of peptide growth factors and oncogenes
- Interaction between myc and fos oncogenes in the control of cell proliferation
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