Further characterization of balb-3t3 transfectants bearing polyoma t antigen genes (1988)
- Authors:
- Autor USP: SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- Subjects: ANTÍGENOS; BIOQUÍMICA
- Language: Português
- Imprenta:
- Source:
- Título do periódico: Arquivos de Biologia e Tecnologia
- Volume/Número/Paginação/Ano: v.31, n.1 , p.34, 1988
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica
-
ABNT
RAMEH, L E e ARMELIN, M C S. Further characterization of balb-3t3 transfectants bearing polyoma t antigen genes. Arquivos de Biologia e Tecnologia. Curitiba: Instituto de Química, Universidade de São Paulo. . Acesso em: 19 abr. 2024. , 1988 -
APA
Rameh, L. E., & Armelin, M. C. S. (1988). Further characterization of balb-3t3 transfectants bearing polyoma t antigen genes. Arquivos de Biologia e Tecnologia. Curitiba: Instituto de Química, Universidade de São Paulo. -
NLM
Rameh LE, Armelin MCS. Further characterization of balb-3t3 transfectants bearing polyoma t antigen genes. Arquivos de Biologia e Tecnologia. 1988 ;31( 1 ): 34.[citado 2024 abr. 19 ] -
Vancouver
Rameh LE, Armelin MCS. Further characterization of balb-3t3 transfectants bearing polyoma t antigen genes. Arquivos de Biologia e Tecnologia. 1988 ;31( 1 ): 34.[citado 2024 abr. 19 ] - Role of polyoma t antigens in balb-c/3t3 cells growth control deregulation
- Controle e subversao da proliferacao
- Glucocorticoid-regulated gene in transformed to normal phenotypic reversion
- T antigens role in polyomavirus transformation: c-myc but not c-fos or c-jun expression is a target for middle t
- Cdna cloning and expression of growth control genes
- Fibroblast growth factor and camp induce the expression of fos and jun proto-oncogenes in pc12 cells
- Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma
- Cloning of differentially expressed genes involved glucocorticoid-induced phenotypic reversion of c6 glioma cells
- Hydrazine derivative - induced proliferation superimposed on citotoxity in cultured moused fibroblasts
- Generation of polyclonal antiserum to the retinoblastoma protein (prb) to study the role played by tumor suppressor genes in c6 / st1 cells' transformed tumor to normal phenotypic reversion
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