Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma (1994)
- Authors:
- Autor USP: SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- Assunto: BIOLOGIA MOLECULAR
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquimica e Biologia Molecular
- Publisher place: São Paulo
- Date published: 1994
- Source:
- Título do periódico: Programa e Resumos
- Conference titles: Reuniao Anual da Sociedade Brasileira de Bioquimica e Biologia Molecular
-
ABNT
VALENTINI, S R et al. Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma. 1994, Anais.. São Paulo: Sociedade Brasileira de Bioquimica e Biologia Molecular, 1994. . Acesso em: 29 mar. 2024. -
APA
Valentini, S. R., Oliveira, M. L. S., Sasahara, R. M., & Armelin, M. C. S. (1994). Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma. In Programa e Resumos. São Paulo: Sociedade Brasileira de Bioquimica e Biologia Molecular. -
NLM
Valentini SR, Oliveira MLS, Sasahara RM, Armelin MCS. Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma. Programa e Resumos. 1994 ;[citado 2024 mar. 29 ] -
Vancouver
Valentini SR, Oliveira MLS, Sasahara RM, Armelin MCS. Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma. Programa e Resumos. 1994 ;[citado 2024 mar. 29 ] - Role of polyoma t antigens in balb-c/3t3 cells growth control deregulation
- Further characterization of balb-3t3 transfectants bearing polyoma t antigen genes
- Controle e subversao da proliferacao
- Glucocorticoid-regulated gene in transformed to normal phenotypic reversion
- T antigens role in polyomavirus transformation: c-myc but not c-fos or c-jun expression is a target for middle t
- Cdna cloning and expression of growth control genes
- Fibroblast growth factor and camp induce the expression of fos and jun proto-oncogenes in pc12 cells
- Cloning of differentially expressed genes involved glucocorticoid-induced phenotypic reversion of c6 glioma cells
- Hydrazine derivative - induced proliferation superimposed on citotoxity in cultured moused fibroblasts
- Generation of polyclonal antiserum to the retinoblastoma protein (prb) to study the role played by tumor suppressor genes in c6 / st1 cells' transformed tumor to normal phenotypic reversion
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