'NA'-k atpase inhibitors (atpase-i) urinary sodium (unav) and dopamine (udav) in experimental coarctation of the aorta (coa) (1994)
- Authors:
- USP affiliated authors: SEQUEIRA, DEOLINDA MARIA CARDOSO DE - FM ; HEIMANN, JOEL CLAUDIO - FM ; FURUKAWA, LUZIA NAOKO SHINOHARA - HU
- Unidades: FM; HU
- Assunto: NEFROLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Hypertension
- Volume/Número/Paginação/Ano: v.12, n.suppl.3, p.100, mar. 1994
- Conference titles: Scientific Meeting of the International Society of Hypertension
-
ABNT
SEQUEIRA, D M C et al. 'NA'-k atpase inhibitors (atpase-i) urinary sodium (unav) and dopamine (udav) in experimental coarctation of the aorta (coa). Journal of Hypertension. London: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 26 abr. 2024. , 1994 -
APA
Sequeira, D. M. C., Shinohara, L. N., Strunz, C. M. C., & Heimann, J. C. (1994). 'NA'-k atpase inhibitors (atpase-i) urinary sodium (unav) and dopamine (udav) in experimental coarctation of the aorta (coa). Journal of Hypertension. London: Faculdade de Medicina, Universidade de São Paulo. -
NLM
Sequeira DMC, Shinohara LN, Strunz CMC, Heimann JC. 'NA'-k atpase inhibitors (atpase-i) urinary sodium (unav) and dopamine (udav) in experimental coarctation of the aorta (coa). Journal of Hypertension. 1994 ;12( suppl.3): 100.[citado 2024 abr. 26 ] -
Vancouver
Sequeira DMC, Shinohara LN, Strunz CMC, Heimann JC. 'NA'-k atpase inhibitors (atpase-i) urinary sodium (unav) and dopamine (udav) in experimental coarctation of the aorta (coa). Journal of Hypertension. 1994 ;12( suppl.3): 100.[citado 2024 abr. 26 ] - 'NA'-k atpase plasma inhibitors (atp-i), urinary sodium (unav) and depamina (udav) excretion in experimental coarctation of the aorta (coa)
- Inibidores da 'NA'-k atpase (atpase-i), excrecao urinaria de sodio (unav) e dopamina (udav) em coartacao da aorta experimental (coa)
- Efeito da restrição salina crônica sobre a sensibilidade à insulina: o papel do sistema nervoso simpático
- Obesity induced by chronic salt restriction
- Metabolic and hemodynamic characterization of wistar rats from the institutional animal facility of the University of São Paulo School of Medicine
- Sympathetic and Renin-Angiotensin Systems Contribute to Increased Blood Pressure in Sucrose-Fed Rats
- Perinatal salt restriction: A new pathway to programming adiposity indices in adult female Wistar rats
- Salt overload during pregnancy alters uterine blood flow, placenta renin angiotensin system (RAS) and placenta weight
- High-salt intake during pregnancy modifies placenta phenotype at least intil the third generation
- Myocardial hypertrophy induced by high salt consumption is prevented by angiotensin II AT2 receptor agonist
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