Interferon-gamma modulates apoptosis by inducing nitric oxide production and fas expression during the acute phase of experimental Trypanosoma cruzi infection (1998)
- Authors:
- USP affiliated authors: CUNHA, FERNANDO DE QUEIROZ - FMRP ; SILVA, JOAO SANTANA DA - FMRP
- Unidade: FMRP
- Subjects: DOENÇAS PARASITÁRIAS; DOENÇA DE CHAGAS
- Language: Inglês
- Imprenta:
- Publisher place: Rio de Janeiro
- Date published: 1998
- Source:
- Título do periódico: Memórias do Instituto Oswaldo Cruz
- Volume/Número/Paginação/Ano: v. 93, supl. 2, p. 257-258, res. IM-101, 1998
- Conference titles: Annual Meeting on Basic Research in Chagas Disease
-
ABNT
MARTINS, G A et al. Interferon-gamma modulates apoptosis by inducing nitric oxide production and fas expression during the acute phase of experimental Trypanosoma cruzi infection. Memórias do Instituto Oswaldo Cruz. Rio de Janeiro: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 19 abr. 2024. , 1998 -
APA
Martins, G. A., Vieira, L. Q., Cunha, F. de Q., & Silva, J. S. da. (1998). Interferon-gamma modulates apoptosis by inducing nitric oxide production and fas expression during the acute phase of experimental Trypanosoma cruzi infection. Memórias do Instituto Oswaldo Cruz. Rio de Janeiro: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Martins GA, Vieira LQ, Cunha F de Q, Silva JS da. Interferon-gamma modulates apoptosis by inducing nitric oxide production and fas expression during the acute phase of experimental Trypanosoma cruzi infection. Memórias do Instituto Oswaldo Cruz. 1998 ; 93 257-258.[citado 2024 abr. 19 ] -
Vancouver
Martins GA, Vieira LQ, Cunha F de Q, Silva JS da. Interferon-gamma modulates apoptosis by inducing nitric oxide production and fas expression during the acute phase of experimental Trypanosoma cruzi infection. Memórias do Instituto Oswaldo Cruz. 1998 ; 93 257-258.[citado 2024 abr. 19 ] - Trypanocidal activity and chemokines MRNA expression by cultured mice myocardial cells infected with Trypanosoma cruzi
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- Neutrophil recruitment in immunized mice depends on MIP-2 inducing the sequential release of MIP-1'alfa', TNF-'alfa' and LT'B IND.4'
- Nitric oxide is involved in control of trypanosoma cruzi - induced parasitemia and directely kills the parasite in vitro
- Tnf-gamma and tnf-alpha mediated resistance to trypanosoma cruzi through no production
- Trypanosoma cruzi-infected cardiomyocytes produce chemokines and cytokines that trigger potent no-dependent trypanocidal
- Detrimental role of endogenous nitric oxide in host defence against Sporothrix schenckii
- The role of cytokines and chemokines in a new model of inflammatory bowel disease
- MIP-1'alfa'/CCL3 acting on the CCR1 receptor mediates neutrophil migration in immune inflammation via sequential release of TNF'alfa' and LTB4
- Inducible cyclooxygenase release prostaglandin mediates immunosupression in acute phase of experimental trypanossoma cruzi infection
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